Susan C. Hasmall

701 total citations
17 papers, 559 citations indexed

About

Susan C. Hasmall is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Susan C. Hasmall has authored 17 papers receiving a total of 559 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Susan C. Hasmall's work include Peroxisome Proliferator-Activated Receptors (13 papers), Drug Transport and Resistance Mechanisms (7 papers) and Carcinogens and Genotoxicity Assessment (4 papers). Susan C. Hasmall is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (13 papers), Drug Transport and Resistance Mechanisms (7 papers) and Carcinogens and Genotoxicity Assessment (4 papers). Susan C. Hasmall collaborates with scholars based in United Kingdom, Switzerland and France. Susan C. Hasmall's co-authors include Ruth Roberts, Neil H. James, Neil Macdonald, Anthony R. Soames, Sabina Cosulich, Stéphan Chevalier, Clifford R. Elcombe, Douglas A. West, N.J Woodyatt and Peter R. Holden and has published in prestigious journals such as Environmental Health Perspectives, Annals of the New York Academy of Sciences and Pharmacology & Therapeutics.

In The Last Decade

Susan C. Hasmall

17 papers receiving 540 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Susan C. Hasmall United Kingdom 15 374 156 149 102 76 17 559
Ronny Fransson-Steen Sweden 15 259 0.7× 198 1.3× 110 0.7× 121 1.2× 109 1.4× 23 605
F. Bieri Switzerland 13 346 0.9× 92 0.6× 119 0.8× 44 0.4× 144 1.9× 27 581
Barbara A. Elswick United States 10 164 0.4× 109 0.7× 123 0.8× 108 1.1× 202 2.7× 14 647
Doris Oesterle Germany 14 194 0.5× 250 1.6× 80 0.5× 140 1.4× 73 1.0× 42 556
Nancy Thompson Hayner United States 8 222 0.6× 163 1.0× 135 0.9× 67 0.7× 155 2.0× 9 690
Engin M. Gözükara United States 14 322 0.9× 146 0.9× 91 0.6× 64 0.6× 74 1.0× 21 585
Gong Xiao United States 12 228 0.6× 98 0.6× 97 0.7× 42 0.4× 98 1.3× 20 450
Chunja Lee Canada 14 191 0.5× 65 0.4× 170 1.1× 80 0.8× 175 2.3× 22 494
Yanying Zhou China 12 176 0.5× 72 0.5× 60 0.4× 89 0.9× 96 1.3× 35 479
Alexandra Rogue France 10 280 0.7× 64 0.4× 73 0.5× 33 0.3× 103 1.4× 15 557

Countries citing papers authored by Susan C. Hasmall

Since Specialization
Citations

This map shows the geographic impact of Susan C. Hasmall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Susan C. Hasmall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Susan C. Hasmall more than expected).

Fields of papers citing papers by Susan C. Hasmall

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Susan C. Hasmall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Susan C. Hasmall. The network helps show where Susan C. Hasmall may publish in the future.

Co-authorship network of co-authors of Susan C. Hasmall

This figure shows the co-authorship network connecting the top 25 collaborators of Susan C. Hasmall. A scholar is included among the top collaborators of Susan C. Hasmall based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Susan C. Hasmall. Susan C. Hasmall is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Elcombe, Clifford R., J. Odum, John R. Foster, et al.. (2002). Prediction of rodent nongenotoxic carcinogenesis: evaluation of biochemical and tissue changes in rodents following exposure to nine nongenotoxic NTP carcinogens.. Environmental Health Perspectives. 110(4). 363–375. 59 indexed citations
2.
Hasmall, Susan C.. (2002). Downregulation of Lactoferrin by PPARalpha Ligands: Role in Perturbation of Hepatocyte Proliferation and Apoptosis. Toxicological Sciences. 68(2). 304–313. 14 indexed citations
3.
Roberts, Ruth, Stéphan Chevalier, Susan C. Hasmall, et al.. (2002). PPARα and the regulation of cell division and apoptosis. Toxicology. 181-182. 167–170. 64 indexed citations
4.
Roberts, Ruth, Neil H. James, Sabina Cosulich, Susan C. Hasmall, & George M. Orphanides. (2001). Role of cytokines in non-genotoxic hepatocarcinogenesis: cause or effect?. Toxicology Letters. 120(1-3). 301–306. 16 indexed citations
5.
6.
Hasmall, Susan C., Neil H. James, Neil Macdonald, Anthony R. Soames, & Ruth Roberts. (2000). Species differences in response to diethylhexylphthalate: suppression of apoptosis, induction of DNA synthesis and peroxisome proliferator activated receptor alpha-mediated gene expression. Archives of Toxicology. 74(2). 85–91. 62 indexed citations
7.
Roberts, Ruth, Neil H. James, Susan C. Hasmall, et al.. (2000). Apoptosis and proliferation in nongenotoxic carcinogenesis: species differences and role of PPARα. Toxicology Letters. 112-113. 49–57. 56 indexed citations
8.
Hasmall, Susan C., Neil H. James, Neil Macdonald, et al.. (2000). Suppression of mouse hepatocyte apoptosis by peroxisome proliferators: role of PPARα and TNFα. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 448(2). 193–200. 45 indexed citations
9.
Hasmall, Susan C.. (2000). Role of hepatic non-parenchymal cells in the response of rat hepatocytes to the peroxisome proliferator nafenopin in vitro. Carcinogenesis. 21(12). 2159–2165. 34 indexed citations
10.
Hasmall, Susan C. & Ruth Roberts. (2000). The Nongenotoxic Hepatocarcinogens Diethylhexylphthalate and Methylclofenapate Induce DNA Synthesis Preferentially in Octoploid Rat Hepatocytes. Toxicologic Pathology. 28(4). 503–509. 6 indexed citations
11.
Hasmall, Susan C. & Ruth Roberts. (1999). The Perturbation of Apoptosis and Mitosis by Drugs and Xenobiotics. Pharmacology & Therapeutics. 82(1). 63–70. 38 indexed citations
12.
13.
James, Neil H., Jason H. Gill, Richard Brindle, et al.. (1998). Peroxisome proliferator-activated receptor (PPAR) alpha-regulated growth responses and their importance to hepatocarcinogenesis. Toxicology Letters. 102-103. 91–96. 23 indexed citations
14.
Hasmall, Susan C., et al.. (1998). The peroxisome proliferator nafenopin does not suppress hepatocyte apoptosis in guinea-pig liver in vivo nor in human hepatocytes in vitro. Archives of Toxicology. 72(12). 777–783. 22 indexed citations
15.
Hasmall, Susan C.. (1997). Expression of the Immediate–Early Genes, c-fos,c-jun,and c-myc:A Comparison in Rats of Nongenotoxic Hepatocarcinogens with Noncarcinogenic Liver Mitogens. Fundamental and Applied Toxicology. 40(1). 129–137. 18 indexed citations
16.
Hasmall, Susan C. & Ruth Roberts. (1997). Hepatic Ploidy, Nuclearity, and Distribution of DNA Synthesis: A Comparison of Nongenotoxic Hepatocarcinogens with Noncarcinogenic Liver Mitogens. Toxicology and Applied Pharmacology. 144(2). 287–293. 19 indexed citations
17.
Elcombe, Clifford R., David Bell, Elwyn Elias, Susan C. Hasmall, & Nick Plant. (1996). Peroxisome Proliferators: Species Differences in Response of Primary Hepatocyte Cultures. Annals of the New York Academy of Sciences. 804(1). 628–635. 32 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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