Nadine Hövelmeyer
- Immunology top 5%
- Molecular Biology
- Neurology top 2%
- Oncology top 10%
- Cancer Research top 5%
- Co-authors
- Ari WaismanF. Thomas WunderlichClaudia M. WunderlichBurkhard BecherMarco PrinzJeppe FalsigGennadij RaivichJosef Priller
- Topics
- Immune Cell Function and Interaction (13 papers)Immune Response and Inflammation (13 papers)NF-κB Signaling Pathways (13 papers)
- Partner nations
- GermanyUnited StatesSweden
In The Last Decade
Nadine Hövelmeyer
38 papers receiving 1.8k citations
Hit Papers
Peers
Comparison fields: 5 of 89
- Immunology 912
- Molecular Biology 601
- Neurology 462
- Oncology 323
- Cancer Research 322
Countries citing papers authored by Nadine Hövelmeyer
This map shows the geographic impact of Nadine Hövelmeyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadine Hövelmeyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadine Hövelmeyer more than expected).
Fields of papers citing papers by Nadine Hövelmeyer
This network shows the impact of papers produced by Nadine Hövelmeyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadine Hövelmeyer. The network helps show where Nadine Hövelmeyer may publish in the future.
Co-authorship network of co-authors of Nadine Hövelmeyer
This figure shows the co-authorship network connecting the top 25 collaborators of Nadine Hövelmeyer. A scholar is included among the top collaborators of Nadine Hövelmeyer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadine Hövelmeyer. Nadine Hövelmeyer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 3 | |
| 4 | 7 | |
| 5 | 25 | |
| 6 | 14 | |
| 7 | 121 | |
| 8 | 23 | |
| 9 | 26 | |
| 10 | 40 | |
| 11 | 19 | |
| 12 | 7 | |
| 13 | 26 | |
| 14 | 143 | |
| 15 | 82 | |
| 16 | 9 | |
| 17 | Experimental autoimmune encephalomyelitis repressed by microglial paralysis (vol 11, pg 146, 2005) | 0 |
| 18 | 137 | |
| 19 | Experimental autoimmune encephalomyelitis repressed by microglial paralysisbreakdown → | 597 |
| 20 | 33 |
About Nadine Hövelmeyer
Nadine Hövelmeyer is a scholar working on Immunology, Cancer Research and Biological Psychiatry, having authored 40 papers that have together received 1.8k indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (13 papers), Immune Response and Inflammation (13 papers) and NF-κB Signaling Pathways (13 papers). The work is most often cited by research in Neurology (462 citations), Immunology (912 citations) and Developmental Neuroscience (128 citations). Nadine Hövelmeyer has collaborated with scholars based in Germany, United States and Sweden. Frequent co-authors include Ari Waisman, F. Thomas Wunderlich, Claudia M. Wunderlich, Burkhard Becher, Marco Prinz, Jeppe Falsig, Gennadij Raivich, Josef Priller, Frank L. Heppner and Adriano Aguzzi. Their work appears in journals such as Nature Medicine, Nature Communications and The Journal of Experimental Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.