Geert Bultynck

29.2k total citations · 2 hit papers
238 papers, 12.7k citations indexed

About

Geert Bultynck is a scholar working on Molecular Biology, Cell Biology and Physiology. According to data from OpenAlex, Geert Bultynck has authored 238 papers receiving a total of 12.7k indexed citations (citations by other indexed papers that have themselves been cited), including 186 papers in Molecular Biology, 55 papers in Cell Biology and 38 papers in Physiology. Recurrent topics in Geert Bultynck's work include Cell death mechanisms and regulation (48 papers), Ion channel regulation and function (47 papers) and Connexins and lens biology (46 papers). Geert Bultynck is often cited by papers focused on Cell death mechanisms and regulation (48 papers), Ion channel regulation and function (47 papers) and Connexins and lens biology (46 papers). Geert Bultynck collaborates with scholars based in Belgium, United States and United Kingdom. Geert Bultynck's co-authors include Jan B. Parys, Humbert De Smedt, Ludwig Missiaen, Luc Leybaert, Jean-Paul Decuypere, Tim Vervliet, Martin D. Bootman, Elke Decrock, Marijke De Bock and Giovanni Monaco and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Geert Bultynck

235 papers receiving 12.6k citations

Hit Papers

PERK is required at the ER-mitochondrial contact sites to... 2012 2026 2016 2021 2012 2025 200 400 600

Peers

Geert Bultynck
H. Llewelyn Roderick United Kingdom
Geert Bultynck
Citations per year, relative to Geert Bultynck Geert Bultynck (= 1×) peers H. Llewelyn Roderick

Countries citing papers authored by Geert Bultynck

Since Specialization
Citations

This map shows the geographic impact of Geert Bultynck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Geert Bultynck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Geert Bultynck more than expected).

Fields of papers citing papers by Geert Bultynck

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Geert Bultynck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Geert Bultynck. The network helps show where Geert Bultynck may publish in the future.

Co-authorship network of co-authors of Geert Bultynck

This figure shows the co-authorship network connecting the top 25 collaborators of Geert Bultynck. A scholar is included among the top collaborators of Geert Bultynck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Geert Bultynck. Geert Bultynck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lemos, Fernanda O., Larry E. Wagner, Martin D. Bootman, et al.. (2024). Tetrameric, active PKM2 inhibits IP3 receptors, potentially requiring GRP75 as an additional interaction partner. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1871(7). 119796–119796. 2 indexed citations
2.
Kale, Justin, Larry E. Wagner, Allen Kaasik, et al.. (2024). CISD2 counteracts the inhibition of ER-mitochondrial calcium transfer by anti-apoptotic BCL-2. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1872(1). 119857–119857. 5 indexed citations
3.
Bultynck, Geert, et al.. (2024). Recent advances in canonical versus non-canonical Ca2+-signaling-related anti-apoptotic Bcl-2 functions and prospects for cancer treatment. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1871(5). 119713–119713. 5 indexed citations
4.
Hulpiau, Paco, Larry E. Wagner, Katja Witschas, et al.. (2024). IP3RPEP6, a novel peptide inhibitor of IP3 receptor channels that does not affect connexin‐43 hemichannels. Acta Physiologica. 240(3). e14086–e14086. 4 indexed citations
5.
Gadet, Rudy, Olivier Lohez, Ivan Mikaélian, et al.. (2024). The endoplasmic reticulum pool of Bcl-xL prevents cell death through IP3R-dependent calcium release. Cell Death Discovery. 10(1). 346–346. 5 indexed citations
6.
Verhelst, Steven H. L., et al.. (2023). Cellular effects of BAPTA: Are they only about Ca2+ chelation?. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1871(2). 119589–119589. 6 indexed citations
7.
Kerkhofs, Martijn, Kirsten Welkenhuyzen, Rita La Rovere, et al.. (2023). Intracellular BAPTA directly inhibits PFKFB3, thereby impeding mTORC1-driven Mcl-1 translation and killing MCL-1-addicted cancer cells. Cell Death and Disease. 14(9). 600–600. 3 indexed citations
8.
Leybaert, Luc, Maarten De Smet, H. Llewelyn Roderick, et al.. (2023). Connexin hemichannels as candidate targets for cardioprotective and anti-arrhythmic treatments. Journal of Clinical Investigation. 133(6). 33 indexed citations
9.
Kania, Elżbieta, Jaclyn S. Long, David G. McEwan, et al.. (2023). LRRK2 phosphorylation status and kinase activity regulate (macro)autophagy in a Rab8a/Rab10-dependent manner. Cell Death and Disease. 14(7). 436–436. 10 indexed citations
10.
Shapovalov, George, Hristina Ivanova, Spyridoula Karamanou, et al.. (2022). Allosteric cross-talk between the hydrophobic cleft and the BH4 domain of Bcl-2 in control of inositol 1,4,5-trisphosphate receptor activity. SHILAP Revista de lepidopterología. 3(3). 375–391. 2 indexed citations
11.
Smet, Maarten De, Nan Wang, Eef Dries, et al.. (2021). Cx43 hemichannel microdomain signaling at the intercalated disc enhances cardiac excitability. Journal of Clinical Investigation. 131(7). 60 indexed citations
13.
Vervloessem, Tamara, Binu K. Sasi, Spyridoula Karamanou, et al.. (2020). BDA-366, a putative Bcl-2 BH4 domain antagonist, induces apoptosis independently of Bcl-2 in a variety of cancer cell models. Cell Death and Disease. 11(9). 769–769. 20 indexed citations
14.
Xu, Ming, Yanqi Chang, Tomas Luyten, et al.. (2019). Ion and pH Sensitivity of a TMBIM Ca2+ Channel. Structure. 27(6). 1013–1021.e3. 15 indexed citations
15.
Ivanova, Hristina, Larry E. Wagner, Akihiko Tanimura, et al.. (2019). Bcl-2 and IP3 compete for the ligand-binding domain of IP3Rs modulating Ca2+ signaling output. Cellular and Molecular Life Sciences. 76(19). 3843–3859. 35 indexed citations
16.
Hulpiau, Paco, Tânia Martins‐Marques, Nan Wang, et al.. (2019). RyR2 regulates Cx43 hemichannel intracellular Ca2+-dependent activation in cardiomyocytes. Cardiovascular Research. 117(1). 123–136. 32 indexed citations
17.
Decuypere, Jean-Paul, Jan B. Parys, & Geert Bultynck. (2015). ITPRs/inositol 1,4,5-trisphosphate receptors in autophagy: From enemy to ally. Autophagy. 11(10). 1944–1948. 26 indexed citations
18.
Parys, Jan B., Martin D. Bootman, David I. Yule, & Geert Bultynck. (2014). Calcium techniques : a laboratory manual. 5 indexed citations
19.
Verfaillie, Tom, Noemí Rubio, Abhishek D. Garg, et al.. (2012). PERK IS REQUIRED AT THE ER-TO-MITOCHONDRIA CONTACT SITES TO CONVEY APOPTOSIS FOLLOWING ROS-MEDIATED ER STRESS. Open Repository and Bibliography (University of Liège). 1 indexed citations
20.
Keddie, Neil S., Tomas Luyten, Davide Bello, et al.. (2011). Development of inositol-based antagonists for the D-myo-inositol 1,4,5-trisphosphate receptor. Oxford University Research Archive (ORA) (University of Oxford). 241. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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