Mustafa Batbayli

628 total citations
10 papers, 411 citations indexed

About

Mustafa Batbayli is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Mustafa Batbayli has authored 10 papers receiving a total of 411 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 3 papers in Cellular and Molecular Neuroscience and 3 papers in Physiology. Recurrent topics in Mustafa Batbayli's work include Mitochondrial Function and Pathology (4 papers), Alzheimer's disease research and treatments (3 papers) and Retinal Development and Disorders (2 papers). Mustafa Batbayli is often cited by papers focused on Mitochondrial Function and Pathology (4 papers), Alzheimer's disease research and treatments (3 papers) and Retinal Development and Disorders (2 papers). Mustafa Batbayli collaborates with scholars based in Denmark, Faroe Islands and Sweden. Mustafa Batbayli's co-authors include Morten Dunø, Elsebet Østergaard, Thomas Rosenberg, Jette Stokholm, M. Schwartz, Gunhild Waldemar, Jørgen E. Nielsen, Suzanne Granhøj Lindquist, Ida E. Holm and Ian Law and has published in prestigious journals such as Journal of the Neurological Sciences, Journal of Medical Genetics and Acta Paediatrica.

In The Last Decade

Mustafa Batbayli

10 papers receiving 406 citations

Peers

Countries citing papers authored by Mustafa Batbayli

Since Specialization

Citations

This map shows the geographic impact of Mustafa Batbayli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mustafa Batbayli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mustafa Batbayli more than expected).

Fields of papers citing papers by Mustafa Batbayli

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mustafa Batbayli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mustafa Batbayli. The network helps show where Mustafa Batbayli may publish in the future.

Co-authorship network of co-authors of Mustafa Batbayli

This figure shows the co-authorship network connecting the top 25 collaborators of Mustafa Batbayli. A scholar is included among the top collaborators of Mustafa Batbayli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mustafa Batbayli. Mustafa Batbayli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

#	Work	Indexed citations
1	A novel RNASEH2B splice site mutation responsible for Aicardi–Goutieres syndrome in the Faroe Islands 2012 ·Acta Paediatrica ·Elsebet Østergaard, (unknown), Karin Sundberg, Morten Dunø, Flemming Juul Hansen, Mustafa Batbayli, Nicolina Sørensen, Alfred Peter Born	6
2	Corticobasal and ataxia syndromes widen the spectrum of C9ORF72 hexanucleotide expansion disease 2012 ·Clinical Genetics ·(unknown), Morten Dunø, Mustafa Batbayli, Andreas Puschmann, Hans Brændgaard, (unknown), Kirsten Svenstrup, Lars H. Pinborg, K. Vestergaard, L. E. Hjermind, Jette Stokholm, (unknown), Peter Johannsen, (unknown)	104
3	A novel MERTK deletion is a common founder mutation in the Faroe Islands and is responsible for a high proportion of retinitis pigmentosa cases. 2011 ·PubMed ·Elsebet Østergaard, Morten Dunø, Mustafa Batbayli, (unknown), Thomas Rosenberg	50
4	Respiratory chain complex I deficiency due to NDUFA12 mutations as a new cause of Leigh syndrome 2011 ·Journal of Medical Genetics ·Elsebet Østergaard, Richard J. Rodenburg, Michiel van den Brand, L. L. Thomsen, Morten Dunø, Mustafa Batbayli, Flemming Wibrand, Leo Nijtmans	49
5	Mutations in PCDH21 cause autosomal recessive cone-rod dystrophy 2010 ·Journal of Medical Genetics ·Elsebet Østergaard, Mustafa Batbayli, Morten Dunø, (unknown), Thomas Rosenberg	48
6	Genetic testing in familial AD and FTD: Mutation and phenotype spectrum in a Danish cohort 2009 ·Clinical Genetics ·(unknown), M. Schwartz, Mustafa Batbayli, Gunhild Waldemar, (unknown)	20
7	A novel missense mutation in SUCLG1 associated with mitochondrial DNA depletion, encephalomyopathic form, with methylmalonic aciduria 2009 ·European Journal of Pediatrics ·Elsebet Østergaard, Marianne Schwartz, Mustafa Batbayli, Ernst Christensen, Ola Hjalmarson, Gittan Kollberg, Elisabeth Holme	39
8	Alzheimer disease‐like clinical phenotype in a family with FTDP‐17 caused by a MAPT R406W mutation 2008 ·European Journal of Neurology ·Suzanne Granhøj Lindquist, Ida E. Holm, M. Schwartz, Ian Law, Jette Stokholm, Mustafa Batbayli, Gunhild Waldemar, Jørgen E. Nielsen	62
9	A novel presenilin 2 mutation (V393M) in early‐onset dementia with profound language impairment 2008 ·European Journal of Neurology ·Suzanne Granhøj Lindquist, Lis Hasholt, Justyna M.C. Bahl, (unknown), (unknown), Anne Nørremølle, Jette Stokholm, M. Schwartz, Mustafa Batbayli, Henning Laursen, Raphaëlle Pardossi‐Piquard, F. Chen, Peter St George‐Hyslop, Gunhild Waldemar, Jørgen E. Nielsen	16
10	Atypical early-onset Alzheimer's disease caused by the Iranian APP mutation 2008 ·Journal of the Neurological Sciences ·Suzanne Granhøj Lindquist, Jørgen E. Nielsen, Jette Stokholm, Marianne Schwartz, Mustafa Batbayli, Martin Ballegaard, Jesper Erdal, Katja Krabbe, Gunhild Waldemar	17

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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