Andreas Puschmann

5.9k total citations
57 papers, 1.6k citations indexed

About

Andreas Puschmann is a scholar working on Neurology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Andreas Puschmann has authored 57 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Neurology, 25 papers in Molecular Biology and 25 papers in Cellular and Molecular Neuroscience. Recurrent topics in Andreas Puschmann's work include Parkinson's Disease Mechanisms and Treatments (16 papers), Genetic Neurodegenerative Diseases (16 papers) and Neurological disorders and treatments (11 papers). Andreas Puschmann is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (16 papers), Genetic Neurodegenerative Diseases (16 papers) and Neurological disorders and treatments (11 papers). Andreas Puschmann collaborates with scholars based in Sweden, United States and Poland. Andreas Puschmann's co-authors include Zbigniew K. Wszołek, Owen A. Ross, Dennis W. Dickson, Takuya Konno, Elisabet Englund, Jianfeng Xiao, Mark S. LeDoux, Oskar Hansson, Ruben Smith and John C. van Swieten and has published in prestigious journals such as Gastroenterology, Brain and Neurology.

In The Last Decade

Andreas Puschmann

54 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andreas Puschmann Sweden 24 1.0k 530 408 379 282 57 1.6k
Miryam Carecchio Italy 24 749 0.7× 342 0.6× 499 1.2× 365 1.0× 324 1.1× 78 1.6k
Morten Blaabjerg Denmark 25 602 0.6× 543 1.0× 446 1.1× 168 0.4× 265 0.9× 65 1.6k
Junling Wang China 22 595 0.6× 665 1.3× 803 2.0× 188 0.5× 214 0.8× 140 1.6k
Christophe Verny France 20 701 0.7× 578 1.1× 853 2.1× 421 1.1× 285 1.0× 51 1.9k
Yasuo Miki Japan 27 1.4k 1.4× 649 1.2× 577 1.4× 491 1.3× 419 1.5× 101 2.1k
Valerie Joers United States 18 811 0.8× 590 1.1× 657 1.6× 322 0.8× 571 2.0× 27 1.9k
Masashi Takanashi Japan 24 919 0.9× 468 0.9× 575 1.4× 503 1.3× 397 1.4× 67 1.7k
Roberto Di Maio United States 19 752 0.8× 641 1.2× 796 2.0× 365 1.0× 266 0.9× 35 1.8k
Giovanna Zorzi Italy 30 1.1k 1.1× 891 1.7× 898 2.2× 206 0.5× 706 2.5× 101 2.5k
Hiroyuki Tomiyama Japan 23 1.3k 1.3× 614 1.2× 513 1.3× 430 1.1× 549 1.9× 43 2.0k

Countries citing papers authored by Andreas Puschmann

Since Specialization
Citations

This map shows the geographic impact of Andreas Puschmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andreas Puschmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andreas Puschmann more than expected).

Fields of papers citing papers by Andreas Puschmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andreas Puschmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andreas Puschmann. The network helps show where Andreas Puschmann may publish in the future.

Co-authorship network of co-authors of Andreas Puschmann

This figure shows the co-authorship network connecting the top 25 collaborators of Andreas Puschmann. A scholar is included among the top collaborators of Andreas Puschmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andreas Puschmann. Andreas Puschmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Siuda, Joanna, Jarosław Sławek, Andreas Puschmann, et al.. (2024). Structural and Functional Characterization of the Most Frequent Pathogenic PRKN Substitution p.R275W. Cells. 13(18). 1540–1540. 2 indexed citations
2.
Ilinca, Andreea, et al.. (2024). Diagnosing Monogenic Stroke at Younger Age. Stroke. 55(12). 2846–2855. 1 indexed citations
3.
Brolin, Kajsa, David Bäckström, Ziv Gan‐Or, et al.. (2024). GBA1 T369M and Parkinson's disease - Further evidence of a lack of association in the Swedish population. Parkinsonism & Related Disorders. 130. 107191–107191.
4.
Anderson, Glenn, et al.. (2024). Maculopathy and adult‐onset ataxia in patients with biallelic MFSD8 variants. Molecular Genetics & Genomic Medicine. 12(8). e2505–e2505. 2 indexed citations
5.
Wassélius, Johan, et al.. (2023). Small vessel disease in primary familial brain calcification with novel truncating PDGFB variants. Neurologia i Neurochirurgia Polska. 58(1). 94–105. 2 indexed citations
6.
Ilinca, Andreea, Andreas Puschmann, Jukka Putaala, et al.. (2022). Updated Stroke Gene Panels: Rapid evolution of knowledge on monogenic causes of stroke. European Journal of Human Genetics. 31(2). 239–242. 5 indexed citations
7.
Gorcenco, Sorina, Christin Karremo, & Andreas Puschmann. (2022). Patients’ Perspective in Hereditary Ataxia. The Cerebellum. 23(1). 82–91. 7 indexed citations
8.
Mulroy, Eoin, et al.. (2021). Throat‐Clearing Vocalizations in Primary Brain Calcification Syndromes. Movement Disorders Clinical Practice. 8(4). 627–630. 1 indexed citations
9.
Puschmann, Andreas. (2017). New Genes Causing Hereditary Parkinson’s Disease or Parkinsonism. Current Neurology and Neuroscience Reports. 17(9). 66–66. 77 indexed citations
10.
Smith, Ruben, Andreas Puschmann, Michael Schöll, et al.. (2016). 18 F-AV-1451 tau PET imaging correlates strongly with tau neuropathology in MAPT mutation carriers. Brain. 139(9). 2372–2379. 130 indexed citations
11.
Dulski, Jarosław, Witold Sołtan, Michał Schinwelski, et al.. (2016). Clinical variability of neuroacanthocytosis syndromes—a series of six patients with long follow-up. Clinical Neurology and Neurosurgery. 147. 78–83. 18 indexed citations
12.
Konno, Takuya, Owen A. Ross, Andreas Puschmann, Dennis W. Dickson, & Zbigniew K. Wszołek. (2015). Autosomal dominant Parkinson's disease caused by SNCA duplications. Parkinsonism & Related Disorders. 22. S1–S6. 143 indexed citations
13.
Taroni, Franco, et al.. (2014). Atypical Friedreich ataxia in patients with FXN p.R165P point mutation or comorbid hemochromatosis. Parkinsonism & Related Disorders. 20(8). 919–923. 9 indexed citations
14.
LeDoux, Mark S., Jianfeng Xiao, Monika Rudzińska, et al.. (2012). Genotype–phenotype correlations in THAP1 dystonia: Molecular foundations and description of new cases. Parkinsonism & Related Disorders. 18(5). 414–425. 55 indexed citations
15.
Hedera, Peter, et al.. (2012). Novel PRRT2 mutation in an African-American family with paroxysmal kinesigenic dyskinesia. BMC Neurology. 12(1). 93–93. 21 indexed citations
16.
Puschmann, Andreas, Christophe Verbeeck, Michael G. Heckman, et al.. (2011). Human leukocyte antigen variation and Parkinson’s disease. Parkinsonism & Related Disorders. 17(5). 376–378. 30 indexed citations
17.
Puschmann, Andreas, Elisabet Englund, Owen A. Ross, et al.. (2011). First neuropathological description of a patient with Parkinson’s disease and LRRK2 p.N1437H mutation. Parkinsonism & Related Disorders. 18(4). 332–338. 39 indexed citations
18.
Puschmann, Andreas, Owen A. Ross, Carles Vilariño‐Güell, et al.. (2009). A Swedish family with de novo α-synuclein A53T mutation: Evidence for early cortical dysfunction. Parkinsonism & Related Disorders. 15(9). 627–632. 77 indexed citations
19.
Neu, Bruno, Andreas Puschmann, Artur Mayerhofer, et al.. (2003). TNF-α induces apoptosis of parietal cells. Biochemical Pharmacology. 65(10). 1755–1760. 30 indexed citations
20.
Puschmann, Andreas, et al.. (1994). Functional evidence for a glibenclamide-sensitive K+ channel in rat ileal smooth muscle. European Journal of Pharmacology. 271(2-3). 379–386. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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