L. E. Hjermind

506 total citations
8 papers, 226 citations indexed

About

L. E. Hjermind is a scholar working on Neurology, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, L. E. Hjermind has authored 8 papers receiving a total of 226 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Neurology, 5 papers in Molecular Biology and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in L. E. Hjermind's work include Neurological disorders and treatments (4 papers), Genetic Neurodegenerative Diseases (4 papers) and Mitochondrial Function and Pathology (3 papers). L. E. Hjermind is often cited by papers focused on Neurological disorders and treatments (4 papers), Genetic Neurodegenerative Diseases (4 papers) and Mitochondrial Function and Pathology (3 papers). L. E. Hjermind collaborates with scholars based in Denmark, Sweden and Germany. L. E. Hjermind's co-authors include Jette Stokholm, Kirsten Svenstrup, Morten Dunø, Hans Brændgaard, K. Vestergaard, Peter Johannsen, E. Dupont, Mustafa Batbayli, Andreas Puschmann and Lars H. Pinborg and has published in prestigious journals such as Brain, Neurology and Journal of Neurology Neurosurgery & Psychiatry.

In The Last Decade

L. E. Hjermind

8 papers receiving 224 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
L. E. Hjermind Denmark 8 160 83 62 44 41 8 226
Chiara Reale Italy 11 157 1.0× 113 1.4× 121 2.0× 40 0.9× 87 2.1× 19 320
Valerija Dobričić Serbia 11 165 1.0× 163 2.0× 124 2.0× 33 0.8× 22 0.5× 21 296
Diana A. Olszewska Ireland 8 148 0.9× 60 0.7× 71 1.1× 76 1.7× 40 1.0× 32 240
Viorica Chelban United Kingdom 13 185 1.2× 162 2.0× 128 2.1× 41 0.9× 85 2.1× 27 381
Daniela Corongiu Italy 8 113 0.7× 61 0.7× 96 1.5× 39 0.9× 47 1.1× 12 239
Elly F. Ippel Netherlands 9 49 0.3× 96 1.2× 139 2.2× 51 1.2× 26 0.6× 10 254
R. Ou China 11 211 1.3× 63 0.8× 68 1.1× 29 0.7× 54 1.3× 21 301
Cid Czarina E. Diesta Philippines 11 214 1.3× 146 1.8× 34 0.5× 11 0.3× 32 0.8× 25 278
Fabrizia Stregapede Italy 9 43 0.3× 90 1.1× 83 1.3× 21 0.5× 40 1.0× 17 205
D. G. Hernandez United Kingdom 3 126 0.8× 45 0.5× 67 1.1× 15 0.3× 55 1.3× 3 216

Countries citing papers authored by L. E. Hjermind

Since Specialization
Citations

This map shows the geographic impact of L. E. Hjermind's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by L. E. Hjermind with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites L. E. Hjermind more than expected).

Fields of papers citing papers by L. E. Hjermind

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by L. E. Hjermind. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by L. E. Hjermind. The network helps show where L. E. Hjermind may publish in the future.

Co-authorship network of co-authors of L. E. Hjermind

This figure shows the co-authorship network connecting the top 25 collaborators of L. E. Hjermind. A scholar is included among the top collaborators of L. E. Hjermind based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with L. E. Hjermind. L. E. Hjermind is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Vinther‐Jensen, Tua, Anja Hviid Simonsen, Esben Budtz–Jørgensen, L. E. Hjermind, & Jørgen E. Nielsen. (2015). Ubiquitin: a potential cerebrospinal fluid progression marker in Huntington's disease. European Journal of Neurology. 22(10). 1378–1384. 16 indexed citations
2.
Vinther‐Jensen, Tua, Troels T. Nielsen, Esben Budtz–Jørgensen, et al.. (2015). Psychiatric and cognitive symptoms in Huntington's disease are modified by polymorphisms in catecholamine regulating enzyme genes. Clinical Genetics. 89(3). 320–327. 10 indexed citations
3.
Dunø, Morten, Mustafa Batbayli, Andreas Puschmann, et al.. (2012). Corticobasal and ataxia syndromes widen the spectrum of C9ORF72 hexanucleotide expansion disease. Clinical Genetics. 83(3). 279–283. 104 indexed citations
4.
Schultz, Kirk R., Karin Nilsson, Suzanne Granhøj Lindquist, et al.. (2009). Transthyretin as a potential CSF biomarker for Alzheimer’s disease and dementia with Lewy bodies: effects of treatment with cholinesterase inhibitors. European Journal of Neurology. 17(3). 456–460. 22 indexed citations
5.
Svenstrup, Kirsten, G Giraud, Odile Boespflug‐Tanguy, et al.. (2009). Hereditary spastic paraplegia caused by the PLP1 'rumpshaker mutation'. Journal of Neurology Neurosurgery & Psychiatry. 81(6). 666–672. 11 indexed citations
6.
Hjermind, L. E., John Vissing, Friedrich Asmus, et al.. (2008). No muscle involvement in myoclonus‐dystonia caused by ɛ‐sarcoglycan gene mutations. European Journal of Neurology. 15(5). 525–529. 12 indexed citations
7.
Asmus, Friedrich, L. E. Hjermind, E. Dupont, et al.. (2007). Genomic deletion size at the epsilon-sarcoglycan locus determines the clinical phenotype. Brain. 130(10). 2736–2745. 34 indexed citations
8.
Hjermind, L. E., Lene Werdelin, Hans Eiberg, et al.. (2003). A novel mutation in the ε-sarcoglycan gene causing myoclonus–dystonia syndrome. Neurology. 60(9). 1536–1539. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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