Melanie Meyer‐Luehmann

7.3k total citations · 3 hit papers
45 papers, 5.6k citations indexed

About

Melanie Meyer‐Luehmann is a scholar working on Physiology, Neurology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Melanie Meyer‐Luehmann has authored 45 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Physiology, 25 papers in Neurology and 17 papers in Cellular and Molecular Neuroscience. Recurrent topics in Melanie Meyer‐Luehmann's work include Alzheimer's disease research and treatments (33 papers), Neuroinflammation and Neurodegeneration Mechanisms (22 papers) and Neuroscience and Neuropharmacology Research (13 papers). Melanie Meyer‐Luehmann is often cited by papers focused on Alzheimer's disease research and treatments (33 papers), Neuroinflammation and Neurodegeneration Mechanisms (22 papers) and Neuroscience and Neuropharmacology Research (13 papers). Melanie Meyer‐Luehmann collaborates with scholars based in Germany, United States and Switzerland. Melanie Meyer‐Luehmann's co-authors include Bradley T. Hyman, Tara L. Spires‐Jones, Brian J. Bacskai, Mónica García‐Alloza, Jessica Koenigsknecht-Talboo, David M. Holtzman, Marco Prinz, Alix de Calignon, Mathias Jucker and Paolo d’Errico and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Melanie Meyer‐Luehmann

45 papers receiving 5.5k citations

Hit Papers

Rapid appearance and local toxicity of amyloid-β plaques ... 2006 2026 2012 2019 2008 2006 2009 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease
    2008 814 citations Melanie Meyer‐Luehmann, Tara L. Spires‐Jones et al. Nature profile →
  2. Exogenous Induction of Cerebral ß-Amyloidogenesis Is Governed by Agent and Host
    2006 744 citations Melanie Meyer‐Luehmann, Matthias Staufenbiel et al. profile →
  3. Oligomeric amyloid β associates with postsynaptic densities and correlates with excitatory synapse loss near senile plaques
    2009 672 citations Melanie Meyer‐Luehmann, Mónica García‐Alloza et al. profile →

Peers

Melanie Meyer‐Luehmann
Joanna L. Jankowsky United States
Jill Richardson United Kingdom
Christopher Janus United States
Gabriela Peña Argentina
Tristan Bolmont Switzerland
Yong Shen China
Jeffy P. Jimenez United States
Soyon Hong United States
Maj-Linda B. Selenica United States
Milene L. Brownlow United States
Joanna L. Jankowsky United States
Melanie Meyer‐Luehmann
Citations per year, relative to Melanie Meyer‐Luehmann Melanie Meyer‐Luehmann (= 1×) peers Joanna L. Jankowsky

Countries citing papers authored by Melanie Meyer‐Luehmann

Since Specialization
Citations

This map shows the geographic impact of Melanie Meyer‐Luehmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Melanie Meyer‐Luehmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Melanie Meyer‐Luehmann more than expected).

Fields of papers citing papers by Melanie Meyer‐Luehmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Melanie Meyer‐Luehmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Melanie Meyer‐Luehmann. The network helps show where Melanie Meyer‐Luehmann may publish in the future.

Co-authorship network of co-authors of Melanie Meyer‐Luehmann

This figure shows the co-authorship network connecting the top 25 collaborators of Melanie Meyer‐Luehmann. A scholar is included among the top collaborators of Melanie Meyer‐Luehmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Melanie Meyer‐Luehmann. Melanie Meyer‐Luehmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
d’Errico, Paolo, et al.. (2025). Neuroprotective and plasticity promoting effects of repetitive transcranial magnetic stimulation (rTMS): A role for microglia. Brain stimulation. 18(3). 810–821. 2 indexed citations
2.
Meyer‐Luehmann, Melanie, et al.. (2024). The intricate interplay between microglia and adult neurogenesis in Alzheimer’s disease. Frontiers in Cellular Neuroscience. 18. 1456253–1456253. 5 indexed citations
3.
Ziegler‐Waldkirch, Stephanie, Stefan Tholen, Marius Schwabenland, et al.. (2023). Tpr Misregulation in Hippocampal Neural Stem Cells in Mouse Models of Alzheimer’s Disease. Cells. 12(23). 2757–2757. 5 indexed citations
4.
Ziegler‐Waldkirch, Stephanie, Desirée Loreth, Jonas‐Frederic Sauer, et al.. (2022). Seed-induced Aβ deposition alters neuronal function and impairs olfaction in a mouse model of Alzheimer’s disease. Molecular Psychiatry. 27(10). 4274–4284. 9 indexed citations
5.
Ziegler‐Waldkirch, Stephanie, Wilfried Reichardt, Daniel Erny, et al.. (2022). Seed-induced Aβ deposits in the corpus callosum disrupt white matter integrity in a mouse model of Alzheimer’s disease. Frontiers in Cellular Neuroscience. 16. 862918–862918. 2 indexed citations
6.
Ziegler‐Waldkirch, Stephanie, Paolo d’Errico, Charlotte Mezö, et al.. (2021). Distinct Aβ pathology in the olfactory bulb and olfactory deficits in a mouse model of Aβ and α‐syn co‐pathology. Brain Pathology. 32(3). e13032–e13032. 4 indexed citations
7.
Masuda, Takahiro, Lukas Amann, Roman Sankowski, et al.. (2020). Novel Hexb-based tools for studying microglia in the CNS. Nature Immunology. 21(7). 802–815. 210 indexed citations
8.
Mezö, Charlotte, Nikolaos Dokalis, Omar Mossad, et al.. (2020). Different effects of constitutive and induced microbiota modulation on microglia in a mouse model of Alzheimer’s disease. Acta Neuropathologica Communications. 8(1). 119–119. 96 indexed citations
9.
Ravi, Vidhya M., Kevin Joseph, Simon P. Behringer, et al.. (2019). Human organotypic brain slice culture: a novel framework for environmental research in neuro-oncology. Life Science Alliance. 2(4). e201900305–e201900305. 45 indexed citations
10.
Datta, Moumita, Ori Staszewski, Elena Raschi, et al.. (2018). Histone Deacetylases 1 and 2 Regulate Microglia Function during Development, Homeostasis, and Neurodegeneration in a Context-Dependent Manner. Immunity. 48(3). 514–529.e6. 144 indexed citations
11.
Ziegler‐Waldkirch, Stephanie & Melanie Meyer‐Luehmann. (2018). The Role of Glial Cells and Synapse Loss in Mouse Models of Alzheimer’s Disease. Frontiers in Cellular Neuroscience. 12. 473–473. 23 indexed citations
12.
Ziegler‐Waldkirch, Stephanie, Paolo d’Errico, Jonas‐Frederic Sauer, et al.. (2017). Seed‐induced Aβ deposition is modulated by microglia under environmental enrichment in a mouse model of Alzheimer's disease. The EMBO Journal. 37(2). 167–182. 87 indexed citations
13.
Meyer‐Luehmann, Melanie & Marco Prinz. (2015). Myeloid Cells in Alzheimer's Disease: Culprits, Victims or Innocent Bystanders?. Trends in Neurosciences. 38(10). 659–668. 47 indexed citations
14.
Hellwig, Sabine, Annette Masuch, Sigrun Nestel, et al.. (2015). Forebrain microglia from wild-type but not adult 5xFAD mice prevent amyloid-β plaque formation in organotypic hippocampal slice cultures. Scientific Reports. 5(1). 14624–14624. 70 indexed citations
15.
Liebscher, Sabine, Edith Winkler, Kyle P. Quinn, et al.. (2013). Chronic γ-secretase inhibition reduces amyloid plaque-associated instability of pre- and postsynaptic structures. Molecular Psychiatry. 19(8). 937–946. 27 indexed citations
16.
Meyer‐Luehmann, Melanie, J. Rodrigo Mora, Tara L. Spires‐Jones, et al.. (2011). T cell mediated cerebral hemorrhages and microhemorrhages during passive Aβ immunization in APPPS1 transgenic mice. Molecular Neurodegeneration. 6(1). 22–22. 13 indexed citations
17.
Koenigsknecht-Talboo, Jessica, Melanie Meyer‐Luehmann, Maia Parsadanian, et al.. (2008). Rapid Microglial Response Around Amyloid Pathology after Systemic Anti-Aβ Antibody Administration in PDAPP Mice. Journal of Neuroscience. 28(52). 14156–14164. 117 indexed citations
18.
Meyer‐Luehmann, Melanie, Tara L. Spires‐Jones, Claudia Prada, et al.. (2008). Rapid appearance and local toxicity of amyloid-β plaques in a mouse model of Alzheimer’s disease. Nature. 451(7179). 720–724. 814 indexed citations breakdown →
19.
Spires‐Jones, Tara L., Melanie Meyer‐Luehmann, Edward A. Stern, et al.. (2005). Dendritic Spine Abnormalities in Amyloid Precursor Protein Transgenic Mice Demonstrated by Gene Transfer and Intravital Multiphoton Microscopy. Journal of Neuroscience. 25(31). 7278–7287. 448 indexed citations
20.
Mueggler, Thomas, Melanie Meyer‐Luehmann, Martin Rausch, et al.. (2004). Restricted diffusion in the brain of transgenic mice with cerebral amyloidosis. European Journal of Neuroscience. 20(3). 811–817. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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