D. Schenk

464 total citations
5 papers, 348 citations indexed

About

D. Schenk is a scholar working on Physiology, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, D. Schenk has authored 5 papers receiving a total of 348 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Physiology, 2 papers in Molecular Biology and 1 paper in Pathology and Forensic Medicine. Recurrent topics in D. Schenk's work include Alzheimer's disease research and treatments (3 papers), Advanced Glycation End Products research (1 paper) and Prion Diseases and Protein Misfolding (1 paper). D. Schenk is often cited by papers focused on Alzheimer's disease research and treatments (3 papers), Advanced Glycation End Products research (1 paper) and Prion Diseases and Protein Misfolding (1 paper). D. Schenk collaborates with scholars based in United States, France and Germany. D. Schenk's co-authors include Jennifer Johnston, Lars Lannfelt, Nikolaos Venizelos, D. J. Selkoe, Martin Citron, Carmen Vigo‐Pelfrey, David B. Teplow, Bengt Winblad, Christine Miller and Eliezer Masliah and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Molecular Psychiatry and Journal of Neuropathology & Experimental Neurology.

In The Last Decade

D. Schenk

5 papers receiving 333 citations

Peers

D. Schenk
Comparison fields: 5 of 46
  • Physiology 314
  • Molecular Biology 183
  • Cellular and Molecular Neuroscience 91
  • Pharmacology 70
  • Computational Theory and Mathematics 50
Replace Kristina Sennvik with:
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Chris De Jonghe Belgium
Hiroki Akashiba Japan
Sharie J. Haugabook United States
Hanjun Guan United States
Jenny Frånberg Sweden
David Demedts Belgium
Geereesh Shankar Ireland
Tatyana Melnikova United States
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Kristina Sennvik Sweden View profile →
Citations per field, relative to D. Schenk
D. Schenk · 1×
Citations per year, relative to D. Schenk
D. Schenk · 1×

Countries citing papers authored by D. Schenk

Since Specialization
Citations

This map shows the geographic impact of D. Schenk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Schenk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Schenk more than expected).

Fields of papers citing papers by D. Schenk

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by D. Schenk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Schenk. The network helps show where D. Schenk may publish in the future.

Co-authorship network of co-authors of D. Schenk

This figure shows the co-authorship network connecting the top 25 collaborators of D. Schenk. A scholar is included among the top collaborators of D. Schenk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D. Schenk. D. Schenk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

5 of 5 papers shown
# Work Indexed citations
1 27
2 44
3 272
4 3
5 2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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