Matthias Drosten
- Molecular Biology top 5%
- Oncology top 5%
- Pathology and Forensic Medicine top 5%
- Cancer Research top 10%
- Pulmonary and Respiratory Medicine top 10%
- Co-authors
- Mariano BarbacidBrigitte M. PützerCarmen G. LechugaCarmen GuerraEleanor Y. M. SumLucía Simón-CarrascoEugenio SantosJelena Urosevic
- Topics
- Protein Kinase Regulation and GTPase Signaling (12 papers)Cancer-related Molecular Pathways (12 papers)Melanoma and MAPK Pathways (9 papers)
- Partner nations
- SpainGermanyUnited States
In The Last Decade
Matthias Drosten
43 papers receiving 2.0k citations
Hit Papers
Peers
Comparison fields: 5 of 91
- Molecular Biology 1.4k
- Oncology 777
- Pathology and Forensic Medicine 301
- Cancer Research 291
- Pulmonary and Respiratory Medicine 261
Countries citing papers authored by Matthias Drosten
This map shows the geographic impact of Matthias Drosten's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthias Drosten with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthias Drosten more than expected).
Fields of papers citing papers by Matthias Drosten
This network shows the impact of papers produced by Matthias Drosten. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthias Drosten. The network helps show where Matthias Drosten may publish in the future.
Co-authorship network of co-authors of Matthias Drosten
This figure shows the co-authorship network connecting the top 25 collaborators of Matthias Drosten. A scholar is included among the top collaborators of Matthias Drosten based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthias Drosten. Matthias Drosten is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | 1 | |
| 3 | 1 | |
| 4 | 9 | |
| 5 | 4 | |
| 6 | 9 | |
| 7 | 22 | |
| 8 | 25 | |
| 9 | 12 | |
| 10 | 18 | |
| 11 | 15 | |
| 12 | Targeting the MAPK Pathway in KRAS-Driven Tumorsbreakdown → | 330 |
| 13 | 87 | |
| 14 | 35 | |
| 15 | 38 | |
| 16 | 45 | |
| 17 | 349 | |
| 18 | 19 | |
| 19 | 7 | |
| 20 | 57 |
About Matthias Drosten
Matthias Drosten is a scholar working on Oncology, Cell Biology and Molecular Biology, having authored 43 papers that have together received 2.0k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (12 papers), Cancer-related Molecular Pathways (12 papers) and Melanoma and MAPK Pathways (9 papers). The work is most often cited by research in Oncology (777 citations), Molecular Biology (1.4k citations) and Cancer Research (291 citations). Matthias Drosten has collaborated with scholars based in Spain, Germany and United States. Frequent co-authors include Mariano Barbacid, Brigitte M. Pützer, Carmen G. Lechuga, Carmen Guerra, Eleanor Y. M. Sum, Lucía Simón-Carrasco, Eugenio Santos, Jelena Urosevic, Luis Miguel Pedrero Esteban and Esther Castellano. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Genes & Development.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.