David Santamarı́a
- Molecular Biology top 5%
- Oncology top 2%
- Cell Biology top 2%
- Pulmonary and Respiratory Medicine top 5%
- Cancer Research top 5%
- Co-authors
- Mariano BarbacidPierre DubusMarcos MalumbresSagrario OrtegaCédric BarrièreSarah L. HuntKathryn NewtonJavier F. Cáceres
- Topics
- Cancer-related Molecular Pathways (13 papers)Microtubule and mitosis dynamics (9 papers)Epigenetics and DNA Methylation (8 papers)
- Partner nations
- SpainFranceUnited States
In The Last Decade
David Santamarı́a
42 papers receiving 3.3k citations
Hit Papers
Peers
Comparison fields: 5 of 103
- Molecular Biology 2.3k
- Oncology 1.5k
- Cell Biology 753
- Pulmonary and Respiratory Medicine 727
- Cancer Research 452
Countries citing papers authored by David Santamarı́a
This map shows the geographic impact of David Santamarı́a's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Santamarı́a with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Santamarı́a more than expected).
Fields of papers citing papers by David Santamarı́a
This network shows the impact of papers produced by David Santamarı́a. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Santamarı́a. The network helps show where David Santamarı́a may publish in the future.
Co-authorship network of co-authors of David Santamarı́a
This figure shows the co-authorship network connecting the top 25 collaborators of David Santamarı́a. A scholar is included among the top collaborators of David Santamarı́a based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Santamarı́a. David Santamarı́a is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 3 | |
| 2 | 8 | |
| 3 | 4 | |
| 4 | 9 | |
| 5 | 1 | |
| 6 | 17 | |
| 7 | 15 | |
| 8 | 8 | |
| 9 | 17 | |
| 10 | 18 | |
| 11 | 91 | |
| 12 | 224 | |
| 13 | 331 | |
| 14 | 87 | |
| 15 | Cdk1 is sufficient to drive the mammalian cell cyclebreakdown → | 806 |
| 16 | 130 | |
| 17 | Mammalian Cells Cycle without the D-Type Cyclin-Dependent Kinases Cdk4 and Cdk6breakdown → | 609 |
| 18 | 29 | |
| 19 | 31 | |
| 20 | 3 |
About David Santamarı́a
David Santamarı́a is a scholar working on Cell Biology, Oncology and Cancer Research, having authored 43 papers that have together received 3.3k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (13 papers), Microtubule and mitosis dynamics (9 papers) and Epigenetics and DNA Methylation (8 papers). The work is most often cited by research in Oncology (1.5k citations), Cell Biology (753 citations) and Molecular Biology (2.3k citations). David Santamarı́a has collaborated with scholars based in Spain, France and United States. Frequent co-authors include Mariano Barbacid, Pierre Dubus, Marcos Malumbres, Sagrario Ortega, Cédric Barrière, Sarah L. Hunt, Kathryn Newton, Javier F. Cáceres, Claudine Tardy and Rocı́o Sotillo. Their work appears in journals such as Nature, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.