Nathan Mise

3.7k total citations · 1 hit paper
52 papers, 2.6k citations indexed

About

Nathan Mise is a scholar working on Molecular Biology, Health, Toxicology and Mutagenesis and Genetics. According to data from OpenAlex, Nathan Mise has authored 52 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 14 papers in Health, Toxicology and Mutagenesis and 13 papers in Genetics. Recurrent topics in Nathan Mise's work include Heavy Metal Exposure and Toxicity (12 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (10 papers) and Epigenetics and DNA Methylation (7 papers). Nathan Mise is often cited by papers focused on Heavy Metal Exposure and Toxicity (12 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (10 papers) and Epigenetics and DNA Methylation (7 papers). Nathan Mise collaborates with scholars based in Japan, United States and United Kingdom. Nathan Mise's co-authors include Kuniya Abe, Sahoko Ichihara, Md. Shiblur Rahaman, Gaku Ichihara, Masaaki Kurasaki, Md. Mostafizur Rahman, Md. Khabir Uddin, Md. Tajuddin Sikder, Yuichi Obata and Susumu Tonegawa and has published in prestigious journals such as Science, Cell and Nucleic Acids Research.

In The Last Decade

Nathan Mise

52 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Environmental arsenic exposure and its contribution to human diseases, toxicity mechanism and management

2021 386 citations Md. Shiblur Rahaman, Nathan Mise et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Nathan Mise Japan	24	1.2k	534	319	309	305	52	2.6k
Keith R. Shockley United States	32	1.2k 1.0×	434 0.8×	75 0.2×	381 1.2×	189 0.6×	91	3.0k
Su Liu China	34	1.4k 1.2×	419 0.8×	101 0.3×	391 1.3×	801 2.6×	158	3.9k
Na He China	26	728 0.6×	647 1.2×	333 1.0×	325 1.1×	251 0.8×	125	2.7k
Jennifer L. Freeman United States	39	2.1k 1.8×	763 1.4×	281 0.9×	1.3k 4.3×	377 1.2×	110	5.3k
Ronghui Li China	24	1.0k 0.8×	222 0.4×	142 0.4×	58 0.2×	121 0.4×	94	2.0k
T. W. Sadler United States	39	1.6k 1.3×	628 1.2×	125 0.4×	288 0.9×	291 1.0×	95	4.8k
Bogdan J. Wlodarczyk United States	32	917 0.8×	471 0.9×	179 0.6×	289 0.9×	123 0.4×	87	2.4k
Matthew Brady United States	38	2.4k 2.0×	403 0.8×	35 0.1×	349 1.1×	202 0.7×	107	5.1k
Scott M. Belcher United States	39	1.2k 1.0×	827 1.5×	529 1.7×	3.0k 9.7×	416 1.4×	87	5.4k
Anna K. Wójtowicz Poland	30	628 0.5×	325 0.6×	107 0.3×	1.0k 3.3×	150 0.5×	75	2.6k

Countries citing papers authored by Nathan Mise

Since Specialization

Citations

This map shows the geographic impact of Nathan Mise's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan Mise with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan Mise more than expected).

Fields of papers citing papers by Nathan Mise

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan Mise. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan Mise. The network helps show where Nathan Mise may publish in the future.

Co-authorship network of co-authors of Nathan Mise

This figure shows the co-authorship network connecting the top 25 collaborators of Nathan Mise. A scholar is included among the top collaborators of Nathan Mise based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathan Mise. Nathan Mise is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Rahaman, Md. Shiblur, Nathan Mise, Akihiko Ikegami, et al.. (2023). The mechanism of low-level arsenic exposure-induced hypertension: Inhibition of the activity of the angiotensin-converting enzyme 2. Chemosphere. 318. 137911–137911. 10 indexed citations

Miyaji, Yumiko, Kiwako Yamamoto‐Hanada, Limin Yang, et al.. (2023). Sex steroid hormones and allergic diseases in children: a pilot birth cohort study in the Japan Environment and Children’s Study cohort. BMC Pediatrics. 23(1). 479–479. 2 indexed citations

Irahara, Makoto, Kiwako Yamamoto‐Hanada, Mayako Saito‐Abe, et al.. (2022). Fluctuations of aeroallergen-specific immunoglobulins and children's allergic profiles: Japan Environment & Children's Study of a pilot cohort. Allergology International. 71(3). 335–344. 5 indexed citations

Ichihara, Sahoko, Cai Zong, Kazuo Kinoshita, et al.. (2021). 1,2-Dichloropropane induces γ-H2AX expression in human cholangiocytes only in the presence of macrophages. Toxicology Letters. 349. 134–144. 6 indexed citations

Iwai‐Shimada, Miyuki, Yayoi Kobayashi, Tomohiko Isobe, et al.. (2021). Comparison of Simultaneous Quantitative Analysis of Methylmercury and Inorganic Mercury in Cord Blood Using LC-ICP-MS and LC-CVAFS: The Pilot Study of the Japan Environment and Children’s Study. Toxics. 9(4). 82–82. 11 indexed citations

Mise, Nathan, Yuka Suzuki, Saeko Tada‐Oikawa, et al.. (2018). MicroRNAs as Potential Mediators for Cigarette Smoking Induced Atherosclerosis. International Journal of Molecular Sciences. 19(4). 1097–1097. 23 indexed citations

Kobayashi, Motoi, Fumitake Usui‐Kawanishi, Tadayoshi Karasawa, et al.. (2017). The cardiac glycoside ouabain activates NLRP3 inflammasomes and promotes cardiac inflammation and dysfunction. PLoS ONE. 12(5). e0176676–e0176676. 35 indexed citations

Cui, Xiaoyi, Nathan Mise, Akihiko Ikegami, et al.. (2016). Heavy metal exposure, in combination with physical activity and aging, is related with oxidative stress in Japanese women from a rural agricultural community. SpringerPlus. 5(1). 885–885. 10 indexed citations

Kuroki, Shunsuke, Shogo Matoba, Mika Akiyoshi, et al.. (2013). Epigenetic Regulation of Mouse Sex Determination by the Histone Demethylase Jmjd1a. Science. 341(6150). 1106–1109. 197 indexed citations

10.

Ikeda, Ryohei, Hirosuke Shiura, Koji Numata, et al.. (2013). Large, Male Germ Cell-Specific Hypomethylated DNA Domains With Unique Genomic and Epigenomic Features on the Mouse X Chromosome. DNA Research. 20(6). 549–565. 10 indexed citations

11.

Kohara, Keigo, Michele Pignatelli, Takashi Kitamura, et al.. (2013). Cell type–specific genetic and optogenetic tools reveal hippocampal CA2 circuits. Nature Neuroscience. 17(2). 269–279. 363 indexed citations

12.

Okamura, Daiji, Makiko Ikeda, Keiko Ozato, et al.. (2012). Cell cycle gene-specific control of transcription has a critical role in proliferation of primordial germ cells. Genes & Development. 26(22). 2477–2482. 27 indexed citations

13.

Chow, Jennifer, Constance Ciaudo, Melissa Fazzari, et al.. (2010). LINE-1 Activity in Facultative Heterochromatin Formation during X Chromosome Inactivation. Cell. 141(6). 956–969. 241 indexed citations

14.

Ogonuki, Narumi, Kimiko Inoue, Michiko Hirose, et al.. (2009). A High-Speed Congenic Strategy Using First-Wave Male Germ Cells. PLoS ONE. 4(3). e4943–e4943. 34 indexed citations

15.

Kobayashi, Shin, Ayako Isotani, Nathan Mise, et al.. (2006). Comparison of Gene Expression in Male and Female Mouse Blastocysts Revealed Imprinting of the X-Linked Gene, Rhox5/Pem, at Preimplantation Stages. Current Biology. 16(2). 166–172. 121 indexed citations

16.

Kiyosawa, Hidenori, et al.. (2005). Disclosing hidden transcripts: Mouse natural sense–antisense transcripts tend to be poly(A) negative and nuclear localized. Genome Research. 15(4). 463–474. 114 indexed citations

17.

Drosten, Matthias, Gero Hilken, Michelle Bockmann, et al.. (2004). Role of MEN2A-Derived RET in Maintenance and Proliferation of Medullary Thyroid Carcinoma. JNCI Journal of the National Cancer Institute. 96(16). 1231–1239. 57 indexed citations

18.

Mise, Nathan & Philip Avner. (2004). BAC Modification Using a RecA Expressing Shuttle Vector System. Humana Press eBooks. 256. 77–88. 2 indexed citations

19.

Mise, Nathan, Yuji Goto, Noboru Nakajima, & Nobuo Takagi. (1999). Molecular Cloning of Antisense Transcripts of the MouseXistGene. Biochemical and Biophysical Research Communications. 258(3). 537–541. 35 indexed citations

20.

Mise, Nathan & Masami Wakahara. (1994). Dorsoventral polarization and formation of dorsal axial structures in Xenopus laevis: analyses using UV irradiation of the full-grown oocyte and after fertilization. The International Journal of Developmental Biology. 38(3). 447–453. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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