Matthew J. Emmett

2.0k total citations
17 papers, 1.3k citations indexed

About

Matthew J. Emmett is a scholar working on Molecular Biology, Physiology and Cancer Research. According to data from OpenAlex, Matthew J. Emmett has authored 17 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 8 papers in Physiology and 5 papers in Cancer Research. Recurrent topics in Matthew J. Emmett's work include Adipose Tissue and Metabolism (8 papers), Circadian rhythm and melatonin (4 papers) and Adipokines, Inflammation, and Metabolic Diseases (4 papers). Matthew J. Emmett is often cited by papers focused on Adipose Tissue and Metabolism (8 papers), Circadian rhythm and melatonin (4 papers) and Adipokines, Inflammation, and Metabolic Diseases (4 papers). Matthew J. Emmett collaborates with scholars based in United States, Greece and Israel. Matthew J. Emmett's co-authors include Mitchell A. Lazar, Dan Feng, David J. Steger, Erika R. Briggs, Patrick Seale, Jennifer Jager, Raymond E. Soccio, Marine Adlanmérini, Bin Fang and Zachary Gerhart‐Hines and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Matthew J. Emmett

16 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthew J. Emmett United States 13 562 549 427 158 155 17 1.3k
Jeongkyung Lee United States 20 731 1.3× 419 0.8× 484 1.1× 209 1.3× 79 0.5× 26 1.5k
Erika R. Briggs United States 11 454 0.8× 668 1.2× 556 1.3× 70 0.4× 247 1.6× 11 1.4k
Anne Bugge Denmark 13 622 1.1× 760 1.4× 760 1.8× 106 0.7× 208 1.3× 17 1.6k
Min‐Dian Li China 19 901 1.6× 463 0.8× 401 0.9× 120 0.8× 152 1.0× 33 1.6k
Manashree Damle United States 13 338 0.6× 290 0.5× 281 0.7× 107 0.7× 94 0.6× 17 758
M. N. Yurova Russia 12 862 1.5× 570 1.0× 233 0.5× 163 1.0× 131 0.8× 32 1.6k
Giorgio Ramadori United States 18 441 0.8× 800 1.5× 595 1.4× 168 1.1× 322 2.1× 30 1.7k
Marie‐France Champy France 22 1.1k 2.0× 896 1.6× 257 0.6× 180 1.1× 467 3.0× 28 2.2k
Kristopher Burkewitz United States 9 647 1.2× 494 0.9× 349 0.8× 93 0.6× 183 1.2× 16 1.3k
Giorgia Benegiamo Switzerland 17 467 0.8× 416 0.8× 669 1.6× 38 0.2× 107 0.7× 33 1.3k

Countries citing papers authored by Matthew J. Emmett

Since Specialization
Citations

This map shows the geographic impact of Matthew J. Emmett's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Emmett with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Emmett more than expected).

Fields of papers citing papers by Matthew J. Emmett

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Emmett. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Emmett. The network helps show where Matthew J. Emmett may publish in the future.

Co-authorship network of co-authors of Matthew J. Emmett

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Emmett. A scholar is included among the top collaborators of Matthew J. Emmett based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Emmett. Matthew J. Emmett is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Emmett, Matthew J., Christos Adamopoulos, Stuart A. Aaronson, et al.. (2025). RAS Mutation–Specific Responses to Paralog- and State-Selective RAS Inhibitors. Molecular Cancer Research. 24(1). 60–71.
2.
Inoue, Shin‐ichi, Matthew J. Emmett, Hee‐Woong Lim, et al.. (2024). Short-term cold exposure induces persistent epigenomic memory in brown fat. Cell Metabolism. 36(8). 1764–1778.e9. 16 indexed citations
3.
Lu, Chenyue, Michael J. Raabe, Ann Liu, et al.. (2024). HCC spatial transcriptomic profiling reveals significant and potentially targetable cancer-endothelial interactions. Hepatology Communications. 8(10). 1 indexed citations
4.
Santoleri, Dominic, Hee‐Woong Lim, Matthew J. Emmett, et al.. (2022). Global-run on sequencing identifies Gm11967 as an Akt-dependent long noncoding RNA involved in insulin sensitivity. iScience. 25(6). 104410–104410. 3 indexed citations
5.
Richter, Hannah J., Amy K. Hauck, Kirill Batmanov, et al.. (2022). Balanced control of thermogenesis by nuclear receptor corepressors in brown adipose tissue. Proceedings of the National Academy of Sciences. 119(33). e2205276119–e2205276119. 5 indexed citations
6.
Angueira, Anthony R., Suzanne N. Shapira, Jeff Ishibashi, et al.. (2020). Early B Cell Factor Activity Controls Developmental and Adaptive Thermogenic Gene Programming in Adipocytes. Cell Reports. 30(9). 2869–2878.e4. 46 indexed citations
7.
Dierickx, Pieterjan, Matthew J. Emmett, Chunjie Jiang, et al.. (2019). SR9009 has REV-ERB–independent effects on cell proliferation and metabolism. Proceedings of the National Academy of Sciences. 116(25). 12147–12152. 118 indexed citations
8.
Emmett, Matthew J. & Mitchell A. Lazar. (2018). Integrative regulation of physiology by histone deacetylase 3. Nature Reviews Molecular Cell Biology. 20(2). 102–115. 134 indexed citations
9.
Emmett, Matthew J., et al.. (2018). β‐Adrenergic receptors control brown adipose UCP‐1 tone and cold response without affecting its circadian rhythmicity. The FASEB Journal. 32(10). 5640–5646. 34 indexed citations
10.
Emmett, Matthew J., Hee‐Woong Lim, Jennifer Jager, et al.. (2017). Histone deacetylase 3 prepares brown adipose tissue for acute thermogenic challenge. Nature. 546(7659). 544–548. 142 indexed citations
11.
Teng, Xin, Matthew J. Emmett, Mitchell A. Lazar, Erwin Goldberg, & Joshua D. Rabinowitz. (2016). Lactate Dehydrogenase C Produces S-2-Hydroxyglutarate in Mouse Testis. ACS Chemical Biology. 11(9). 2420–2427. 34 indexed citations
12.
Soccio, Raymond E., Eric R. Chen, Satyajit Rajapurkar, et al.. (2015). Genetic Variation Determines PPARγ Function and Anti-diabetic Drug Response In Vivo. Cell. 162(1). 33–44. 90 indexed citations
13.
Koren, Shlomit, Lisa M. DiPilato, Matthew J. Emmett, et al.. (2015). The role of mouse Akt2 in insulin-dependent suppression of adipocyte lipolysis in vivo. Diabetologia. 58(5). 1063–1070. 22 indexed citations
14.
Zhang, Yuxiang, Bin Fang, Matthew J. Emmett, et al.. (2015). Discrete functions of nuclear receptor Rev-erbα couple metabolism to the clock. Science. 348(6242). 1488–1492. 247 indexed citations
15.
Finkbeiner, Stacy R., Matthew J. Emmett, Feven Tameire, et al.. (2014). De Novo Formation of Insulin-Producing “Neo-β Cell Islets” from Intestinal Crypts. Cell Reports. 6(6). 1046–1058. 122 indexed citations
16.
Gerhart‐Hines, Zachary, Dan Feng, Matthew J. Emmett, et al.. (2013). The nuclear receptor Rev-erbα controls circadian thermogenic plasticity. Nature. 503(7476). 410–413. 213 indexed citations
17.
Madison, Blair, Qi Liu, Xue Zhong, et al.. (2013). LIN28B promotes growth and tumorigenesis of the intestinal epithelium via Let-7. Genes & Development. 27(20). 2233–2245. 101 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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