Ronald Cohen

4.4k total citations
76 papers, 2.8k citations indexed

About

Ronald Cohen is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Physiology. According to data from OpenAlex, Ronald Cohen has authored 76 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 29 papers in Endocrinology, Diabetes and Metabolism and 25 papers in Physiology. Recurrent topics in Ronald Cohen's work include Adipose Tissue and Metabolism (24 papers), Retinoids in leukemia and cellular processes (21 papers) and Estrogen and related hormone effects (18 papers). Ronald Cohen is often cited by papers focused on Adipose Tissue and Metabolism (24 papers), Retinoids in leukemia and cellular processes (21 papers) and Estrogen and related hormone effects (18 papers). Ronald Cohen collaborates with scholars based in United States, Japan and Germany. Ronald Cohen's co-authors include Fredric E. Wondisford, Anthony N. Hollenberg, Koshi Hashimoto, Samuel Refetoff, Matthew Brady, Christine Yu, Kathleen R. Markan, Wei Zheng, Juan Kong and Pan Wei and has published in prestigious journals such as Proceedings of the National Academy of Sciences, JAMA and Journal of Biological Chemistry.

In The Last Decade

Ronald Cohen

75 papers receiving 2.7k citations

Peers

Ronald Cohen
Sara H. Windahl Sweden
Hideaki Sowa Japan
R. Clay Bunn United States
James G. Simmons United States
Dike N. Kalu United States
Robert Brommage United States
Toshio Tsushima Japan
John F. Kuemmerle United States
Shigeru Sakurai Japan
Clara Crescioli Italy
Sara H. Windahl Sweden
Ronald Cohen
Citations per year, relative to Ronald Cohen Ronald Cohen (= 1×) peers Sara H. Windahl

Countries citing papers authored by Ronald Cohen

Since Specialization
Citations

This map shows the geographic impact of Ronald Cohen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald Cohen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald Cohen more than expected).

Fields of papers citing papers by Ronald Cohen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ronald Cohen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald Cohen. The network helps show where Ronald Cohen may publish in the future.

Co-authorship network of co-authors of Ronald Cohen

This figure shows the co-authorship network connecting the top 25 collaborators of Ronald Cohen. A scholar is included among the top collaborators of Ronald Cohen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ronald Cohen. Ronald Cohen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nordgren, Rachel, Ronald Cohen, David Sarne, et al.. (2025). Prevalence and Clinical Impact of BRAF p.V600E Mutation in Papillary Thyroid Carcinoma. Endocrine Pathology. 36(1). 13–13. 2 indexed citations
2.
Cohen, Ronald, et al.. (2024). Atomic force microscopy characterization of white and beige adipocyte differentiation. In Vitro Cellular & Developmental Biology - Animal. 60(8). 842–852. 1 indexed citations
3.
Nanda, Rita, Ronald Cohen, Zoe Quandt, et al.. (2024). Incidence and time to onset of immunotherapy-related adrenal insufficiency in the I-SPY2 trial.. Journal of Clinical Oncology. 42(16_suppl). 584–584.
4.
Cohen, Ronald, et al.. (2021). Investigation of insulin resistance through a multiorgan microfluidic organ-on-chip. Biomedical Materials. 17(2). 25002–25002. 8 indexed citations
5.
Sharma, Aishwarya, Yan Li, Alexandria Hoffman, et al.. (2020). SMRT Regulates Metabolic Homeostasis and Adipose Tissue Macrophage Phenotypes in Tandem. Endocrinology. 161(10). 4 indexed citations
6.
Shimizu, Hiroaki, Yu Lu, Kristen R. Vella, et al.. (2019). Nuclear corepressor SMRT is a strong regulator of body weight independently of its ability to regulate thyroid hormone action. PLoS ONE. 14(8). e0220717–e0220717. 12 indexed citations
7.
Nanda, Rita, Erica Stringer-Reasor, Masha Kocherginsky, et al.. (2016). A randomized phase I trial of nanoparticle albumin-bound paclitaxel with or without mifepristone for advanced breast cancer. SpringerPlus. 5(1). 947–947. 30 indexed citations
8.
Goldfarb, Yael, Noam Kadouri, Ben Levi, et al.. (2016). HDAC3 Is a Master Regulator of mTEC Development. Cell Reports. 15(3). 651–665. 26 indexed citations
9.
Emont, Margo P., et al.. (2015). Silencing Mediator of Retinoid and Thyroid Hormone Receptors (SMRT) regulates glucocorticoid action in adipocytes. Molecular and Cellular Endocrinology. 407. 52–56. 8 indexed citations
10.
Carmean, Christopher M., Ronald Cohen, & Matthew Brady. (2013). Systemic regulation of adipose metabolism. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1842(3). 424–430. 16 indexed citations
11.
Biyashev, Dauren, et al.. (2010). Natural Angiogenesis Inhibitor Signals through Erk5 Activation of Peroxisome Proliferator-activated Receptor γ (PPARγ). Journal of Biological Chemistry. 285(18). 13517–13524. 24 indexed citations
12.
13.
Kim, Helen H., Andrew Wolfe, Ronald Cohen, et al.. (2006). In VivoIdentification of a 107-Base Pair Promoter Element Mediating Neuron-Specific Expression of Mouse Gonadotropin-Releasing Hormone. Molecular Endocrinology. 21(2). 457–471. 20 indexed citations
14.
Ortiga-Carvalho, Tânia Maria, Nobuyuki Shibusawa, Karen Jesus Oliveira, et al.. (2005). Negative regulation by thyroid hormone receptor requires an intact coactivator-binding surface. Journal of Clinical Investigation. 115(9). 2517–2523. 55 indexed citations
15.
Hashimoto, Koshi, Ronald Cohen, Masanobu Yamada, et al.. (2005). Cross-talk between Thyroid Hormone Receptor and Liver X Receptor Regulatory Pathways Is Revealed in a Thyroid Hormone Resistance Mouse Model. Journal of Biological Chemistry. 281(1). 295–302. 63 indexed citations
16.
Shibusawa, Nobuyuki, Koshi Hashimoto, M. Charles Liberman, et al.. (2003). Thyroid hormone action in the absence of thyroid hormone receptor DNA-binding in vivo. Journal of Clinical Investigation. 112(4). 588–597. 92 indexed citations
17.
Pohlenz, Joachim, A Dumitrescu, Ursula Martiné, et al.. (2002). Partial deficiency of Thyroid transcription factor 1 produces predominantly neurological defects in humans and mice. Journal of Clinical Investigation. 109(4). 469–473. 140 indexed citations
18.
Cohen, Ronald, et al.. (2000). The Nuclear Corepressors Recognize Distinct Nuclear Receptor Complexes. Molecular Endocrinology. 14(6). 900–914. 82 indexed citations
19.
Cohen, Ronald, Fredric E. Wondisford, & Anthony N. Hollenberg. (1998). Two Separate NCoR (Nuclear Receptor Corepressor) Interaction Domains Mediate Corepressor Action on Thyroid Hormone Response Elements. Molecular Endocrinology. 12(10). 1567–1581. 64 indexed citations
20.
Safer, Joshua D., et al.. (1997). Isoform Variable Action among Thyroid Hormone Receptor Mutants Provides Insight into Pituitary Resistance to Thyroid Hormone. Molecular Endocrinology. 11(1). 16–26. 49 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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