Louise Howell

1.8k total citations · 1 hit paper
15 papers, 967 citations indexed

About

Louise Howell is a scholar working on Molecular Biology, Hematology and Oncology. According to data from OpenAlex, Louise Howell has authored 15 papers receiving a total of 967 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 5 papers in Hematology and 3 papers in Oncology. Recurrent topics in Louise Howell's work include Acute Myeloid Leukemia Research (5 papers), Histone Deacetylase Inhibitors Research (5 papers) and Retinoids in leukemia and cellular processes (4 papers). Louise Howell is often cited by papers focused on Acute Myeloid Leukemia Research (5 papers), Histone Deacetylase Inhibitors Research (5 papers) and Retinoids in leukemia and cellular processes (4 papers). Louise Howell collaborates with scholars based in United Kingdom, United States and Germany. Louise Howell's co-authors include Arthur Zelent, Kevin Petrie, John E. Dick, Fabien Guidez, Mark D. Minden, Andreea C. Popescu, Carsten Müller‐Tidow, Robert A. Casero, Ken Mills and Patrick M. Woster and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Genes & Development.

In The Last Decade

Louise Howell

15 papers receiving 954 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Inhibition of the LSD1 (KDM1A) demethylase reactivates the all-trans-retinoic acid differentiation pathway in acute myeloid leukemia

2012 502 citations Tino Schenk, Weihsu Claire Chen et al. Nature Medicine profile →

Peers

Louise Howell

HEMAT

ONCOL

IMMUN

GENET

James T. Lynch United Kingdom

Tomoyoshi Nakadai Japan

Dilshad H. Khan Canada

Cecilia Grimaldi Italy

Ling Bei United States

Louis R. Ghanem United States

Alla Dolnikov Australia

Lior Golomb Israel

Emery H. Bresnick United States

Andrea Basile Italy

James T. Lynch United Kingdom

Louise Howell

935 ×1.2

255 ×1.3

HEMAT

137 ×1.3

ONCOL

98 ×1.0

IMMUN

122 ×1.3

GENET

Citations per year, relative to Louise Howell Louise Howell (= 1×) peers James T. Lynch

Countries citing papers authored by Louise Howell

Since Specialization

Citations

This map shows the geographic impact of Louise Howell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Louise Howell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Louise Howell more than expected).

Fields of papers citing papers by Louise Howell

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Louise Howell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Louise Howell. The network helps show where Louise Howell may publish in the future.

Co-authorship network of co-authors of Louise Howell

This figure shows the co-authorship network connecting the top 25 collaborators of Louise Howell. A scholar is included among the top collaborators of Louise Howell based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Louise Howell. Louise Howell is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Krásný, Lukáš, Jessica Burns, Emma Perkins, et al.. (2024). Clinical Implications and Molecular Features of Extracellular Matrix Networks in Soft Tissue Sarcomas. Clinical Cancer Research. 30(15). 3229–3242. 3 indexed citations

Anbalagan, Selvakumar, Carol Box, Sheng Yu, et al.. (2024). Tumour reoxygenation after intratumoural hydrogen peroxide (KORTUC) injection: a novel approach to enhance radiosensitivity. SHILAP Revista de lepidopterología. 2(1). 78–78. 3 indexed citations

Urban‐Wójciuk, Zuzanna, Amy C. Graham, Karen Barker, et al.. (2021). The biguanide polyamine analog verlindamycin promotes differentiation in neuroblastoma via induction of antizyme. Cancer Gene Therapy. 29(7). 940–950. 4 indexed citations

Andrejeva, Gabriela, Sharon Gowan, Gigin Lin, et al.. (2019). De novo phosphatidylcholine synthesis is required for autophagosome membrane formation and maintenance during autophagy. Autophagy. 16(6). 1044–1060. 76 indexed citations

Ahmad, Zai, Veronica Gil, Louise Howell, et al.. (2015). Molecular and In Vivo Characterization of Cancer-Propagating Cells Derived from MYCN-Dependent Medulloblastoma. PLoS ONE. 10(3). e0119834–e0119834. 16 indexed citations

Bansal, Nidhi, Kevin Petrie, Rossitza Christova, et al.. (2015). Targeting the SIN3A-PF1 interaction inhibits epithelial to mesenchymal transition and maintenance of a stem cell phenotype in triple negative breast cancer. Oncotarget. 6(33). 34087–34105. 24 indexed citations

Palmi, Chiara, Grazia Fazio, Angela Maria Savino, et al.. (2014). Cytoskeletal Regulatory Gene Expression and Migratory Properties of B-cell Progenitors Are Affected by the ETV6–RUNX1 Rearrangement. Molecular Cancer Research. 12(12). 1796–1806. 7 indexed citations

Cubedo, Elena, Veronica Gil, Chae Hwa Kim, et al.. (2014). HDAC9 Expression Is Deregulated in Malignant B-Cell Lymphomas in Particular in Diffuse Large B Cell Lymphoma and Mantle Cell Lymphoma. Blood. 124(21). 5374–5374. 1 indexed citations

Schenk, Tino, Weihsu Claire Chen, Stefanie Göllner, et al.. (2012). Inhibition of the LSD1 (KDM1A) demethylase reactivates the all-trans-retinoic acid differentiation pathway in acute myeloid leukemia. Nature Medicine. 18(4). 605–611. 502 indexed citations breakdown →

10.

Doulatov, Sergei, Faiyaz Notta, Kim L. Rice, et al.. (2009). PLZF is a regulator of homeostatic and cytokine-induced myeloid development. Genes & Development. 23(17). 2076–2087. 79 indexed citations

11.

Petrie, Kevin, Fabien Guidez, Jiang Zhu, et al.. (2008). Retinoblastoma protein and the leukemia-associated PLZF transcription factor interact to repress target gene promoters. Oncogene. 27(39). 5260–5266. 8 indexed citations

12.

Gil, Veronica, Louise Howell, Jenny Yeung, et al.. (2007). Ectopic Expression of HDAC9 in Murine Lymphoid System Leads to Altered Lymphocyte Numbers and Proliferation as Well as Predisposition to Tumorigenesis.. Blood. 110(11). 376–376. 1 indexed citations

13.

Guidez, Fabien, Louise Howell, Mark Isalan, et al.. (2005). Histone Acetyltransferase Activity of p300 Is Required for Transcriptional Repression by the Promyelocytic Leukemia Zinc Finger Protein. Molecular and Cellular Biology. 25(13). 5552–5566. 90 indexed citations

14.

Ivins, Sarah, et al.. (2003). Regulation of Hoxb2 by APL-associated PLZF protein. Oncogene. 22(24). 3685–3697. 38 indexed citations

15.

Petrie, Kevin, Fabien Guidez, Louise Howell, et al.. (2003). The Histone Deacetylase 9 Gene Encodes Multiple Protein Isoforms. Journal of Biological Chemistry. 278(18). 16059–16072. 115 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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