Jonathan D. Licht

31.2k citations

255 papers · 17.9k indexed · 5 hit papers · h-index 71

Impact in

Hematology top 0.1%
- Acute Myeloid Leukemia Research
Molecular Biology top 0.1%
- Retinoids in leukemia and cellular processes
- Epigenetics and DNA Methylation
- Histone Deacetylase Inhibitors Research
- Protein Degradation and Inhibitors
- Renal and related cancers
- Cancer-related gene regulation
- Genomics and Chromatin Dynamics

Papers in

Hematology 87
- Acute Myeloid Leukemia Research 63
Molecular Biology 212
- Retinoids in leukemia and cellular processes 52
- Epigenetics and DNA Methylation 48
- Protein Degradation and Inhibitors 45
- Histone Deacetylase Inhibitors Research 44
- Genomics and Chromatin Dynamics 26
- Renal and related cancers 25
- Cancer-related gene regulation 24

Co-authors: Ari Melnick Dong‐Er Zhang Robert Hromas Daniel G. Tenen Ricky W. Johnstone M. Albert Basson Teresa Ezponda Richard L. Bennett
Journals: Blood (55 papers)Journal of Biological Chemistry (18 papers)Oncogene (14 papers)Molecular and Cellular Biology (11 papers)Proceedings of the National Academy of Sciences (9 papers)
Partner nations: United States United Kingdom Canada

In The Last Decade

Jonathan D. Licht

250 papers receiving 17.6k citations

Hit Papers

5 papers align trajectories log scale

Mitochondria Are Required for Antigen-Specific T Cell Activation through Reactive Oxygen Species Signaling 2013 · 986 citations

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if any of the following hold:

it has ≥500 total citations;
it reaches ≥1.5× the top-1% citation threshold for papers in the same subfield and year (the threshold is the minimum needed to enter the top 1%, not the average within it);
it reaches the top citation threshold in at least one of its specific research topics.

2013 Immunity
2003 Nature Genetics
1999 Blood
1997 Blood
1997 Blood

Peers

Countries citing papers authored by Jonathan D. Licht

Since Specialization

Citations

This map shows the geographic impact of Jonathan D. Licht's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan D. Licht with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan D. Licht more than expected).

Fields of papers citing papers by Jonathan D. Licht

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan D. Licht. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan D. Licht. The network helps show where Jonathan D. Licht may publish in the future.

Co-authors

The 25 scholars most cited alongside Jonathan D. Licht, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jonathan D. Licht Line = papers co-authored together Jonathan D. Licht links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Association of Glucagon‐Like Peptide‐1 Receptor Agonists With Cancer Risk in Older Adults With Type 2 Diabetes Obesity ·充洋池原, Hao Dai, Huilin Tang, William T. Donahoo, Thomas J. George, Ramon C. Sun, Sizun Jiang, Aik Choon Tan, Yi Guo, Jonathan D. Licht, John M. Allen, Д. М. Копанева, Jingchuan Guo, Jiang Bian	2025	1
2	KDM6A regulates immune response genes in multiple myeloma Blood ·Kouhei Sugita, Alberto Riva, Benjamin G. Barwick, Sayantan Maji, Chen Jun-na, Jianping Li, Umasankar De, Amin Sobh, Lib Web, Sara Japhet, Marta Kulis, Teresa Ezponda, José I. Martı́n-Subero, Giovanni Tonon, Weizhou Zhang, Constantine S. Mitsiades, Lawrence Boise, Richard L. Bennett, Jonathan D. Licht	2024	5
3	A novel regimen for pancreatic ductal adenocarcinoma targeting MEK, BCL-xL, and EGFR Neoplasia ·Song Han, Ono Kyosuke, Peiyi Zhang, Guangrong Zheng, Daohong Zhou, Zhiguang Huo, Jonathan D. Licht, Thomas J. George, Carmen J. Allegra, José G. Treviño, Steven J. Hughes	2024	3
4	The C-terminal PHDVC5HCH tandem domain of NSD2 is a combinatorial reader of unmodified H3K4 and tri-methylated H3K27 that regulates transcription of cell adhesion genes in multiple myeloma Nucleic Acids Research ·Amy E. Kupferman, 安文, Michela Ghitti, Giacomo Quilici, V. A. Khyuvenen, Jianping Li, 陳慧臻, 祐輔木村, Stefan Knapp, Jonathan D. Licht, Giovanna Musco	2024	2
5	Discovery of a Potent and Selective Targeted NSD2 Degrader for the Reduction of H3K36me2 Journal of the American Chemical Society ·Ronan P. Hanley, Yan Nie, Nanang Purniawan, Fengling Li, Amin Sobh, Chenxi Xu, Natalie K. Barker, David Dilworth, Taraneh Hajian, Elisa Gibson, Magdalena M. Szewczyk, Peter J. Brown, Dalia Baršytė-Lovejoy, Laura E. Herring, Gang Greg Wang, Jonathan D. Licht, Masoud Vedadi, C.H. Arrowsmith, Lindsey I. James	2023	32
6	Low muscle mass is associated with a higher risk of all–cause and cardiovascular disease–specific mortality in cancer survivors Nutrition ·Dongyu Zhang, Jin Bing-Liang, Sergio Setién, Demetra D. Christou, Shama D. Karanth, Stephen D. Anton, Christiaan Leeuwenburgh, Muxuan Liang, James H. Gawron, Danting Yang, الفت جمال محمد محمد زناتي, Robert T. Mankowski, Ting‐Yuan David Cheng, Hanchao Zhang, Erin M. Siegel, Frank J. Penedo, Jonathan D. Licht, Dejana Braithwaite	2022	8
7	BAP1 Loss Promotes Suppressive Tumor Immune Microenvironment via Upregulation of PROS1 in Class 2 Uveal Melanomas Cancers ·P. S. Landa, James J. Dollar, M.S. D’Agostino, Ilhama Namazova, Margaret I. Sanchez, Christina L. Decatur, Jonathan D. Licht, Keiran S.M. Smalley, Zélia M. Corrêa, Stefan Kurtenbach, J. William Harbour	2022	23
8	HDAC Inhibition Enhances the In Vivo Efficacy of MEK Inhibitor Therapy in Uveal Melanoma Clinical Cancer Research ·Fernanda Faião‐Flores, Michael F. Emmons, Michael Durante, Fumi Kinose, Biswarup Saha, Bin Fang, John M. Koomen, Srikumar Chellappan, Silvya Stuchi Maria–Engler, Uwe Rix, Jonathan D. Licht, J. William Harbour, Keiran S.M. Smalley	2019	69
9	A Mutation in Histone H2B Represents a New Class of Oncogenic Driver Cancer Discovery ·Richard L. Bennett, Akinteye Ibrahim Akinyemi, Eliza C. Small, Christine Will, Behnam Nabet, Jon A. Oyer, Xiaoxiao Huang, Rajarshi P. Ghosh, Adrian T. Grzybowski, Tao Yu, Qiao Zhang, Alberto Riva, Tanmay P. Lele, George C. Schatz, Neil L. Kelleher, Alexander J. Ruthenburg, Jan Liphardt, Jonathan D. Licht	2019	65
10	HDAC8 Regulates a Stress Response Pathway in Melanoma to Mediate Escape from BRAF Inhibitor Therapy Cancer Research ·Michael F. Emmons, Fernanda Faião‐Flores, Ritin Sharma, Ram Thapa, Jane L. Messina, Jürgen C. Becker, Dirk Schadendorf, Edward Seto, Vernon K. Sondak, John M. Koomen, Y. Ann Chen, Eric K. Lau, Lixin Wan, Jonathan D. Licht, Keiran S.M. Smalley	2019	52
11	DISORDERED HISTONE METHYLATION IN HEMATOLOGICAL MALIGNANCIES THE CASE OF UTX/KDM6A. PubMed ·Jonathan D. Licht	2018	1
12	UTX/KDM6A Loss Enhances the Malignant Phenotype of Multiple Myeloma and Sensitizes Cells to EZH2 inhibition Cell Reports ·Teresa Ezponda, Daphné Dupéré-Richer, Christine Will, Eliza C. Small, Nobish Varghese, Stacy Sue Rosello, Behnam Nabet, Relja Popovic, Jon A. Oyer, Marinka Bulic, Yupeng Zheng, Xiaoxiao Huang, Mrinal Y. Shah, Sayantan Maji, Alberto Riva, Hakan Oğuz Arı, Giovanni Tonon, Neil L. Kelleher, Jonathan J. Keats, Jonathan D. Licht	2017	92
13	Unabridged Analysis of Human Histone H3 by Differential Top-Down Mass Spectrometry Reveals Hypermethylated Proteoforms from MMSET/NSD2 Overexpression Molecular & Cellular Proteomics ·Yupeng Zheng, Luca Fornelli, Philip D. Compton, S. Sharma, Jesse D. Canterbury, Christopher Mullen, Vlad Zabrouskov, Ryan T. Fellers, Paul M. Thomas, Jonathan D. Licht, Michael W. Senko, Neil L. Kelleher	2015	58
14	WHSC1 Promotes Oncogenesis through Regulation of NIMA-Related Kinase-7 in Squamous Cell Carcinoma of the Head and Neck Molecular Cancer Research ·Vassiliki Saloura, Hyun‐Soo Cho, Kazuma Kiyotani, Houda Alachkar, Zhixiang Zuo, Makoto Nakakido, Tatsuhiko Tsunoda, Tanguy Y. Seiwert, Mark W. Lingen, Jonathan D. Licht, Yusuke Nakamura, Ryuji Hamamoto	2014	83
15	Expression of Leukemia-Associated Fusion Proteins Increases Sensitivity to Histone Deacetylase Inhibitor–Induced DNA Damage and Apoptosis Molecular Cancer Therapeutics ·Luca A. Petruccelli, Filippa Pettersson, Sonia V. del Rincón, Hidalgo‐Lara Maria Eugenia, Jonathan D. Licht, Wilson H. Miller	2013	21
16	Transcriptional Profiling of Polycythemia Vera Identifies Gene Expression Patterns Both Dependent and Independent from the Action of JAK2V617F Clinical Cancer Research ·Windy Berkofsky‐Fessler, Monica Buzzai, Lori A. Houser, Steven Fruchtman, Vesna Najfeld, Mori Tooru, Fabrício F. Costa, Jared M. Bischof, Marcelo B. Soares, Melanie J. McConnell, Weijia Zhang, Ross L. Levine, D. Gary Gilliland, Raffaele Calogero, Jonathan D. Licht	2010	30
17	MDS and secondary AML display unique patterns and abundance of aberrant DNA methylation Blood ·María E. Figueroa, Lucy Skrabanek, Yushan Li, Anchalee Jiemjit, Tamer E. Fandy, Elisabeth Paietta, Hugo F. Fernández, Martin S. Tallman, John M. Greally, Hetty E. Carraway, Jonathan D. Licht, Steven D. Gore, Ari Melnick	2009	231
18	Histone Acetyltransferase Activity of p300 Is Required for Transcriptional Repression by the Promyelocytic Leukemia Zinc Finger Protein Molecular and Cellular Biology ·Fabien Guidez, Louise Howell, Mark Isalan, Marek Cebrat, Rhoda M. Alani, Sarah Ivins, Itsaso Hormaeche, Melanie J. McConnell, Sarah B. Pierce, Philip A. Cole, Jonathan D. Licht, Arthur Zelent	2005	90
19	Growth Suppression by Acute PromyelocyticLeukemia-Associated Protein PLZF Is Mediated by Repression of c-myc Expression Molecular and Cellular Biology ·Melanie J. McConnell, Nathalie Chevallier, Windy Berkofsky‐Fessler, Jena M. Giltnane, Kikis Patsalos, Louis M. Staudt, Jonathan D. Licht	2003	111
20	The Transcriptional Effect of WT1 Is Modulated by Choice of Expression Vector Journal of Biological Chemistry ·Josina C. Reddy, Seiyu Hosono, Jonathan D. Licht	1995	43

About Jonathan D. Licht

Jonathan D. Licht is a scholar working on Hematology, Molecular Biology, Genetics, Oncology and Cancer Research, having authored 255 papers that have together received 17.9k indexed citations. Recurring topics across this work include Acute Myeloid Leukemia Research (63 papers), Retinoids in leukemia and cellular processes (52 papers), Epigenetics and DNA Methylation (48 papers), Protein Degradation and Inhibitors (45 papers), Histone Deacetylase Inhibitors Research (44 papers), Genomics and Chromatin Dynamics (26 papers), Renal and related cancers (25 papers) and Cancer-related gene regulation (24 papers). The work is most often cited by research in Hematology (4.1k citations), Molecular Biology (14.1k citations), Immunology (2.8k citations), Cancer Research (1.8k citations) and Oncology (2.4k citations). Jonathan D. Licht has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Ari Melnick, Dong‐Er Zhang, Robert Hromas, Daniel G. Tenen, Ricky W. Johnstone, M. Albert Basson, Teresa Ezponda, Richard L. Bennett, D. Morrison and Arthur Zelent. Their work appears in journals such as Blood, Journal of Biological Chemistry, Oncogene, Molecular and Cellular Biology and Proceedings of the National Academy of Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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