Koji Yamano

7.3k total citations · 3 hit papers
55 papers, 5.6k citations indexed

About

Koji Yamano is a scholar working on Molecular Biology, Epidemiology and Neurology. According to data from OpenAlex, Koji Yamano has authored 55 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 41 papers in Molecular Biology, 25 papers in Epidemiology and 12 papers in Neurology. Recurrent topics in Koji Yamano's work include Mitochondrial Function and Pathology (31 papers), Autophagy in Disease and Therapy (24 papers) and ATP Synthase and ATPases Research (17 papers). Koji Yamano is often cited by papers focused on Mitochondrial Function and Pathology (31 papers), Autophagy in Disease and Therapy (24 papers) and ATP Synthase and ATPases Research (17 papers). Koji Yamano collaborates with scholars based in Japan, United States and Germany. Koji Yamano's co-authors include Richard J. Youle, Toshiya Endo, Noriyuki Matsuda, Mashun Onishi, Miyuki Sato, Koji Okamoto, Adam I. Fogel, Lesley A. Kane, Shireen A. Sarraf and Keiji Tanaka and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Koji Yamano

54 papers receiving 5.6k citations

Hit Papers

PINK1 phosphorylates ubiq... 2013 2026 2017 2021 2014 2021 2013 250 500 750

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Koji Yamano Japan 32 4.0k 2.5k 972 786 725 55 5.6k
Der‐Fen Suen Taiwan 14 4.1k 1.0× 3.1k 1.2× 1.4k 1.5× 811 1.0× 999 1.4× 17 6.2k
Lesley A. Kane United States 19 4.1k 1.0× 3.8k 1.5× 1.4k 1.4× 868 1.1× 1.0k 1.4× 22 6.4k
Shireen A. Sarraf United States 11 3.3k 0.8× 3.3k 1.3× 1.1k 1.2× 764 1.0× 735 1.0× 16 5.2k
Tomotake Kanki Japan 34 3.1k 0.8× 2.4k 0.9× 462 0.5× 863 1.1× 502 0.7× 75 4.5k
Dionisia P. Sideris Greece 17 2.4k 0.6× 1.8k 0.7× 543 0.6× 607 0.8× 464 0.6× 18 3.6k
Yushan Zhu China 33 3.1k 0.8× 2.3k 0.9× 382 0.4× 814 1.0× 600 0.8× 66 4.8k
Jakob D. Wikström Sweden 27 4.1k 1.0× 1.9k 0.8× 289 0.3× 669 0.9× 1.5k 2.1× 40 6.1k
Atsushi Tanaka Japan 15 5.0k 1.3× 4.4k 1.7× 2.2k 2.2× 1.1k 1.4× 1.3k 1.8× 30 7.8k
Jean‐François Trempe Canada 28 2.7k 0.7× 1.7k 0.7× 934 1.0× 674 0.9× 432 0.6× 68 4.0k
Toshihiko Oka Japan 23 3.3k 0.8× 1.1k 0.4× 435 0.4× 425 0.5× 538 0.7× 41 4.0k

Countries citing papers authored by Koji Yamano

Since Specialization
Citations

This map shows the geographic impact of Koji Yamano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Koji Yamano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Koji Yamano more than expected).

Fields of papers citing papers by Koji Yamano

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Koji Yamano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Koji Yamano. The network helps show where Koji Yamano may publish in the future.

Co-authorship network of co-authors of Koji Yamano

This figure shows the co-authorship network connecting the top 25 collaborators of Koji Yamano. A scholar is included among the top collaborators of Koji Yamano based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Koji Yamano. Koji Yamano is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Endo, Akinori, Naoko Ishibashi, Yasumasa Nishito, et al.. (2025). Ubiquitination-activated TAB–TAK1–IKK–NF-κB axis modulates gene expression for cell survival in the lysosomal damage response. eLife. 14. 1 indexed citations
2.
Okatsu, Kei, Reika Kikuchi, Noriyuki Matsuda, Shuya Fukai, & Koji Yamano. (2025). Functional and Structural Insights Into Complex Formation Between OPTN Leucine Zipper Domain and RAB8A. Genes to Cells. 30(5). e70043–e70043.
3.
Saito, Muneyasu, Masaki Mishima, Masahiro Yamashina, et al.. (2025). The reaction mechanism for glycolysis side product degradation by Parkinson’s disease–linked DJ-1. The Journal of Cell Biology. 224(8). 1 indexed citations
4.
Yamano, Koji, Reika Kikuchi, Waka Kojima, et al.. (2024). Optineurin provides a mitophagy contact site for TBK1 activation. The EMBO Journal. 43(5). 754–779. 31 indexed citations
5.
Koyano, Fumika, Koji Yamano, Hidetaka Kosako, et al.. (2024). AAA+ ATPase chaperone p97/VCPFAF2 governs basal pexophagy. Nature Communications. 15(1). 9347–9347. 9 indexed citations
6.
Kikuchi, Reika, et al.. (2024). TBK1 adaptor AZI2/NAP1 regulates NDP52-driven mitochondrial autophagy. Journal of Biological Chemistry. 300(10). 107775–107775. 5 indexed citations
7.
Yamano, Koji, et al.. (2023). Mitochondrial lipid dynamics regulated by MITOL-mediated ubiquitination. The Journal of Biochemistry. 175(3). 217–219. 1 indexed citations
8.
Yamano, Koji, et al.. (2023). Mitochondrial quality control via organelle and protein degradation. The Journal of Biochemistry. 175(5). 487–494. 5 indexed citations
9.
Onishi, Mashun, Koji Yamano, Miyuki Sato, Noriyuki Matsuda, & Koji Okamoto. (2021). Molecular mechanisms and physiological functions of mitophagy. The EMBO Journal. 40(3). e104705–e104705. 932 indexed citations breakdown →
10.
Kojima, Waka, Koji Yamano, Hidetaka Kosako, et al.. (2021). Mammalian BCAS3 and C16orf70 associate with the phagophore assembly site in response to selective and non-selective autophagy. Autophagy. 17(8). 2011–2036. 14 indexed citations
11.
Queliconi, Bruno B., Waka Kojima, Mayumi Kimura, et al.. (2021). Unfolding is the driving force for mitochondrial import and degradation of the Parkinson's disease-related protein DJ-1. Journal of Cell Science. 134(22). 6 indexed citations
12.
Yamano, Koji, Reika Kikuchi, Waka Kojima, et al.. (2020). Critical role of mitochondrial ubiquitination and the OPTN–ATG9A axis in mitophagy. The Journal of Cell Biology. 219(9). 150 indexed citations
13.
Yamano, Koji & Richard J. Youle. (2020). Two different axes CALCOCO2-RB1CC1 and OPTN-ATG9A initiate PRKN-mediated mitophagy. Autophagy. 16(11). 2105–2107. 35 indexed citations
14.
Koyano, Fumika, Koji Yamano, Hidetaka Kosako, Keiji Tanaka, & Noriyuki Matsuda. (2019). Parkin recruitment to impaired mitochondria for nonselective ubiquitylation is facilitated by MITOL. Journal of Biological Chemistry. 294(26). 10300–10314. 90 indexed citations
15.
Koyano, Fumika, Koji Yamano, Hidetaka Kosako, et al.. (2019). Parkin‐mediated ubiquitylation redistributes MITOL/March5 from mitochondria to peroxisomes. EMBO Reports. 20(12). e47728–e47728. 42 indexed citations
16.
Yamano, Koji, Chunxin Wang, Shireen A. Sarraf, et al.. (2018). Endosomal Rab cycles regulate Parkin-mediated mitophagy. eLife. 7. 127 indexed citations
17.
Okatsu, Kei, Yusuke Sato, Koji Yamano, et al.. (2018). Structural insights into ubiquitin phosphorylation by PINK1. Scientific Reports. 8(1). 10382–10382. 32 indexed citations
18.
Kane, Lesley A., Michael Lazarou, Adam I. Fogel, et al.. (2014). PINK1 phosphorylates ubiquitin to activate Parkin E3 ubiquitin ligase activity. The Journal of Cell Biology. 205(2). 143–153. 991 indexed citations breakdown →
19.
Shiota, Takuya, et al.. (2011). In vivo protein-interaction mapping of a mitochondrial translocator protein Tom22 at work. Proceedings of the National Academy of Sciences. 108(37). 15179–15183. 101 indexed citations
20.
Tamura, Yasushi, Yoshihiro Harada, Takuya Shiota, et al.. (2009). Tim23–Tim50 pair coordinates functions of translocators and motor proteins in mitochondrial protein import. The Journal of Cell Biology. 184(1). 129–141. 113 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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