Akinori Endo

1.6k total citations
30 papers, 1.0k citations indexed

About

Akinori Endo is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Akinori Endo has authored 30 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 7 papers in Cell Biology and 6 papers in Oncology. Recurrent topics in Akinori Endo's work include Ubiquitin and proteasome pathways (14 papers), Endoplasmic Reticulum Stress and Disease (6 papers) and RNA modifications and cancer (5 papers). Akinori Endo is often cited by papers focused on Ubiquitin and proteasome pathways (14 papers), Endoplasmic Reticulum Stress and Disease (6 papers) and RNA modifications and cancer (5 papers). Akinori Endo collaborates with scholars based in Japan, United Kingdom and Germany. Akinori Endo's co-authors include Yasushi Saeki, Masayuki Komada, Toshifumi Inada, Keiji Tanaka, Angus I. Lamond, Naomi Kitamura, Fumiaki Ohtake, Ken Ikeuchi, Hikaru Tsuchiya and Shigeo Murata and has published in prestigious journals such as Nature, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Akinori Endo

30 papers receiving 1.0k citations

Peers

Countries citing papers authored by Akinori Endo

Since Specialization

Citations

This map shows the geographic impact of Akinori Endo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Akinori Endo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Akinori Endo more than expected).

Fields of papers citing papers by Akinori Endo

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Akinori Endo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Akinori Endo. The network helps show where Akinori Endo may publish in the future.

Co-authorship network of co-authors of Akinori Endo

This figure shows the co-authorship network connecting the top 25 collaborators of Akinori Endo. A scholar is included among the top collaborators of Akinori Endo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Akinori Endo. Akinori Endo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Title	Journal	Authors	Indexed citations
1	Ubiquitination-activated TAB–TAK1–IKK–NF-κB axis modulates gene expression for cell survival in the lysosomal damage response	eLife	Akinori Endo, Naoko Ishibashi et al.	1
2	Combinatorial ubiquitin code degrades deubiquitylation-protected substrates	Nature Communications	Takuya Tomita, Akinori Endo et al.	2
3	USP8 prevents aberrant NF-κB and Nrf2 activation by counteracting ubiquitin signals from endosomes	The Journal of Cell Biology	Akinori Endo, Toshiaki Fukushima et al.	7
4	Sugar-mediated non-canonical ubiquitination impairs Nrf1/NFE2L1 activation	Molecular Cell	Yukiko Yoshida, Tsuyoshi Takahashi et al.	11
5	Ubiquitin‐mediated endosomal stress: A novel organelle stress of early endosomes that initiates cellular signaling pathways	BioEssays	Akinori Endo, Masayuki Komada et al.	1
6	Senescent cells form nuclear foci that contain the 26S proteasome	Cell Reports	Masakazu Kato, Hidetaka Kosako et al.	11
7	cIAP1-based degraders induce degradation via branched ubiquitin architectures	Nature Chemical Biology	Yuki Mori, Shivani Dixit et al.	25
8	Molecular basis of ubiquitin-specific protease 8 autoinhibition by the WW-like domain	Communications Biology	Kei Moritsugu, Masahide Kubo et al.	11
9	TRIP12 promotes small-molecule-induced degradation through K29/K48-branched ubiquitin chains	Molecular Cell	Katsuhide Igarashi, Akinori Endo et al.	66
10	Discovery of a Highly Potent and Selective Degrader Targeting Hematopoietic Prostaglandin D Synthase via In Silico Design	Journal of Medicinal Chemistry	Hidetomo Yokoo, Norihito Shibata et al.	27
11	RQT complex dissociates ribosomes collided on endogenous RQC substrate SDD1	Nature Structural & Molecular Biology	Yoshitaka Matsuo, Petr Těšina et al.	105
12	Two distinct modes of DNMT1 recruitment ensure stable maintenance DNA methylation	Nature Communications	Atsuya Nishiyama, Christopher B. Mulholland et al.	96
13	Stress- and ubiquitylation-dependent phase separation of the proteasome	Nature	Sayaka Yasuda, Hikaru Tsuchiya et al.	208
14	Ubiquitin-specific protease 8 deubiquitinates Sec31A and decreases large COPII carriers and collagen IV secretion	Biochemical and Biophysical Research Communications	Kohei Kawaguchi, Akinori Endo et al.	10
15	Human RIF 1 and protein phosphatase 1 stimulate DNA replication origin licensing but suppress origin activation	EMBO Reports	Shin‐ichiro Hiraga, Tony Ly et al.	105
16	The Chromatin Assembly Factor Complex 1 (CAF1) and 5-Azacytidine (5-AzaC) Affect Cell Motility in Src-transformed Human Epithelial Cells	Journal of Biological Chemistry	Akinori Endo, Tony Ly et al.	7
17	Analysis of Human Protein Replacement Stable Cell Lines Established using snoMEN-PR Vector	PLoS ONE	Motoharu Ono, Kayo Yamada et al.	7
18	Human box C/D snoRNA processing conservation across multiple cell types	Nucleic Acids Research	Michelle S. Scott, Motoharu Ono et al.	81
19	Arrhythmogenic superior vena cava caused by partial anomalous pulmonary venous return: Panel A–F	Heart	Kotaro Fukumoto, Seiji Takatsuki et al.	1
20	Nucleophosmin/B23 Regulates Ubiquitin Dynamics in Nucleoli by Recruiting Deubiquitylating Enzyme USP36	Journal of Biological Chemistry	Akinori Endo, Naomi Kitamura et al.	44

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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