Joseph E. Qualls

3.9k total citations
39 papers, 2.9k citations indexed

About

Joseph E. Qualls is a scholar working on Immunology, Epidemiology and Infectious Diseases. According to data from OpenAlex, Joseph E. Qualls has authored 39 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Immunology, 7 papers in Epidemiology and 5 papers in Infectious Diseases. Recurrent topics in Joseph E. Qualls's work include Immune cells in cancer (16 papers), Immune Cell Function and Interaction (12 papers) and Immune Response and Inflammation (12 papers). Joseph E. Qualls is often cited by papers focused on Immune cells in cancer (16 papers), Immune Cell Function and Interaction (12 papers) and Immune Response and Inflammation (12 papers). Joseph E. Qualls collaborates with scholars based in United States, United Kingdom and Netherlands. Joseph E. Qualls's co-authors include Peter J. Murray, Amber M. Smith, Alan M. Kaplan, Donald A. Cohen, Nico van Rooijen, Geoffrey Neale, Gilla Kaplan, Karim C. El Kasmi, Thomas A. Wynn and Luke O'neill and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Joseph E. Qualls

39 papers receiving 2.9k citations

Peers

Joseph E. Qualls

IMMUN

EPIDE

Noah S. Butler United States

You‐Me Kim South Korea

Céline Eidenschenk United States

Jiusheng Deng United States

Christelle Faveeuw France

Dominic De Nardo Australia

Antje Blumenthal Australia

Jonathan P. Moorman United States

Kenneth C. Malcolm United States

Noah S. Butler United States

Joseph E. Qualls

2.4k ×1.5

IMMUN

980 ×1.1

523 ×1.2

EPIDE

405 ×1.0

200 ×0.7

Citations per year, relative to Joseph E. Qualls Joseph E. Qualls (= 1×) peers Noah S. Butler

Countries citing papers authored by Joseph E. Qualls

Since Specialization

Citations

This map shows the geographic impact of Joseph E. Qualls's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph E. Qualls with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph E. Qualls more than expected).

Fields of papers citing papers by Joseph E. Qualls

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph E. Qualls. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph E. Qualls. The network helps show where Joseph E. Qualls may publish in the future.

Co-authorship network of co-authors of Joseph E. Qualls

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph E. Qualls. A scholar is included among the top collaborators of Joseph E. Qualls based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph E. Qualls. Joseph E. Qualls is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zafar, Farhan, Bal Krishan Sharma, Daniel Leino, et al.. (2023). Factor XII promotes the thromboinflammatory response in a rat model of venoarterial extracorporeal membrane oxygenation. Journal of Thoracic and Cardiovascular Surgery. 168(2). e37–e53. 4 indexed citations

Qualls, Joseph E., et al.. (2021). Metabolic Regulation of Immune Responses to Mycobacterium tuberculosis: A Spotlight on L-Arginine and L-Tryptophan Metabolism. Frontiers in Immunology. 11. 628432–628432. 25 indexed citations

Woo, Vivienne, Emily M. Eshleman, Seika Hashimoto‐Hill, et al.. (2021). Commensal segmented filamentous bacteria-derived retinoic acid primes host defense to intestinal infection. Cell Host & Microbe. 29(12). 1744–1756.e5. 66 indexed citations

Schmidt, Stephanie, et al.. (2021). Promotion of Anti-Tuberculosis Macrophage Activity by L-Arginine in the Absence of Nitric Oxide. Frontiers in Immunology. 12. 653571–653571. 17 indexed citations

Huamán, Moisés A., Joseph E. Qualls, Shinsmon Jose, et al.. (2020). Mycobacterium bovis Bacille-Calmette-Guérin Infection Aggravates Atherosclerosis. Frontiers in Immunology. 11. 607957–607957. 12 indexed citations

Oates, Jarren R., Maria E. Moreno‐Fernandez, Michelle S. M. A. Damen, et al.. (2019). Macrophage Function in the Pathogenesis of Non-alcoholic Fatty Liver Disease: The Mac Attack. Frontiers in Immunology. 10. 2893–2893. 55 indexed citations

Boucher, Alexander A., Bal Krishan Sharma, Adam Lane, et al.. (2019). Cell type‐specific mechanisms coupling protease‐activated receptor‐1 to infectious colitis pathogenesis. Journal of Thrombosis and Haemostasis. 18(1). 91–103. 13 indexed citations

Schmidt, Stephanie, Vandana Chaturvedi, Jeremy M. Kinder, et al.. (2017). l-Citrulline Metabolism in Mice Augments CD4+ T Cell Proliferation and Cytokine Production In Vitro, and Accumulation in the Mycobacteria-Infected Lung. Frontiers in Immunology. 8. 1561–1561. 17 indexed citations

Kratochvill, Franz, Nina Gratz, Joseph E. Qualls, et al.. (2015). Tristetraprolin Limits Inflammatory Cytokine Production in Tumor-Associated Macrophages in an mRNA Decay–Independent Manner. Cancer Research. 75(15). 3054–3064. 36 indexed citations

10.

Qualls, Joseph E. & Peter J. Murray. (2015). Immunometabolism within the tuberculosis granuloma: amino acids, hypoxia, and cellular respiration. Seminars in Immunopathology. 38(2). 139–152. 65 indexed citations

11.

Kratochvill, Franz, Geoffrey Neale, Jessica M. Haverkamp, et al.. (2015). TNF Counterbalances the Emergence of M2 Tumor Macrophages. Cell Reports. 12(11). 1902–1914. 227 indexed citations

12.

Haverkamp, Jessica M., Amber M. Smith, Ricardo Weinlich, et al.. (2014). Myeloid-Derived Suppressor Activity Is Mediated by Monocytic Lineages Maintained by Continuous Inhibition of Extrinsic and Intrinsic Death Pathways. Immunity. 41(6). 947–959. 126 indexed citations

13.

Qualls, Joseph E., Geoffrey Neale, Jessica M. Haverkamp, et al.. (2012). MyD88 and Stat3 signaling are fundamental to tumor associated macrophage function (162.34). The Journal of Immunology. 188(1_Supplement). 162.34–162.34. 1 indexed citations

14.

Qualls, Joseph E., Chitra Subramanian, Wasiulla Rafi, et al.. (2012). Sustained Generation of Nitric Oxide and Control of Mycobacterial Infection Requires Argininosuccinate Synthase 1. Cell Host & Microbe. 12(3). 313–323. 136 indexed citations

15.

Qualls, Joseph E. & Peter J. Murray. (2010). A double agent in cancer: Stopping macrophages wounds tumors. Nature Medicine. 16(8). 863–864. 10 indexed citations

16.

McCoy, Claire E., Frederick J. Sheedy, Joseph E. Qualls, et al.. (2010). IL-10 Inhibits miR-155 Induction by Toll-like Receptors. Journal of Biological Chemistry. 285(27). 20492–20498. 217 indexed citations

17.

Qualls, Joseph E., et al.. (2009). Direct and indirect type-1 arginase (Arg1) induction following Mycobacterium bovis (BCG) infection (43.1). The Journal of Immunology. 182(Supplement_1). 43.1–43.1. 2 indexed citations

18.

Moreira, Lilian O., et al.. (2008). Modulation of adaptive immunity by different adjuvant–antigen combinations in mice lacking Nod2. Vaccine. 26(46). 5808–5813. 32 indexed citations

19.

Kasmi, Karim C. El, Joseph E. Qualls, John Pesce, et al.. (2008). Toll-like receptor–induced arginase 1 in macrophages thwarts effective immunity against intracellular pathogens. Nature Immunology. 9(12). 1399–1406. 491 indexed citations

20.

Pager, Cara T., et al.. (2003). Host‐guest scale of left‐handed polyproline II helix formation. Proteins Structure Function and Bioinformatics. 53(1). 68–75. 116 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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