Matthew J. Flick

10.0k total citations · 2 hit papers
120 papers, 7.2k citations indexed

About

Matthew J. Flick is a scholar working on Hematology, Pulmonary and Respiratory Medicine and Molecular Biology. According to data from OpenAlex, Matthew J. Flick has authored 120 papers receiving a total of 7.2k indexed citations (citations by other indexed papers that have themselves been cited), including 61 papers in Hematology, 43 papers in Pulmonary and Respiratory Medicine and 23 papers in Molecular Biology. Recurrent topics in Matthew J. Flick's work include Blood properties and coagulation (42 papers), Blood Coagulation and Thrombosis Mechanisms (36 papers) and Platelet Disorders and Treatments (31 papers). Matthew J. Flick is often cited by papers focused on Blood properties and coagulation (42 papers), Blood Coagulation and Thrombosis Mechanisms (36 papers) and Platelet Disorders and Treatments (31 papers). Matthew J. Flick collaborates with scholars based in United States, Denmark and Canada. Matthew J. Flick's co-authors include Jay L. Degen, Keith W. Kombrinck, Joseph S. Palumbo, Thomas H. Bugge, Cynthia C. Daugherty, Kathryn E. Talmage, James P. Luyendyk, Stephen F. Konieczny, Markéta Jiroušková and Jonathan G. Schoenecker and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Matthew J. Flick

113 papers receiving 7.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Platelets and fibrin(ogen) increase metastatic potential by impeding natural killer cell–mediated elimination of tumor cells

2004 779 citations Joseph S. Palumbo, Matthew J. Flick et al. Blood profile →
The multifaceted role of fibrinogen in tissue injury and inflammation

2018 358 citations James P. Luyendyk, Matthew J. Flick et al. Blood profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew J. Flick United States	41	2.3k	1.8k	1.6k	1.4k	1.3k	120	7.2k
Henri H. Versteeg Netherlands	37	2.4k 1.0×	1.7k 1.0×	706 0.5×	755 0.5×	872 0.7×	129	7.1k
Joseph E. Italiano United States	50	5.1k 2.3×	2.9k 1.6×	761 0.5×	1.6k 1.1×	1.6k 1.3×	133	9.7k
Perumal Thiagarajan United States	43	2.7k 1.2×	2.9k 1.6×	776 0.5×	1.0k 0.7×	361 0.3×	138	7.5k
Eduardo Anglés‐Cano France	36	1.1k 0.5×	1.1k 0.6×	1.4k 0.9×	1.3k 0.9×	714 0.6×	151	6.1k
D.C. Rijken Netherlands	45	3.3k 1.4×	1.4k 0.8×	3.4k 2.2×	2.2k 1.5×	597 0.5×	134	7.3k
H.R. Lijnen Belgium	44	1.8k 0.8×	1.5k 0.9×	2.7k 1.7×	1.2k 0.8×	604 0.5×	147	5.5k
José Sampol France	44	1.3k 0.6×	3.5k 2.0×	856 0.6×	702 0.5×	866 0.7×	92	7.4k
Samuel A. Santoro United States	53	2.4k 1.0×	2.5k 1.4×	1.6k 1.0×	811 0.6×	947 0.7×	120	8.3k
Alexander Brill Israel	34	2.0k 0.9×	2.3k 1.3×	553 0.4×	1.1k 0.8×	737 0.6×	84	8.1k
Florea Lupu United States	60	2.8k 1.2×	4.3k 2.4×	2.7k 1.7×	1.6k 1.1×	1.2k 0.9×	182	12.3k

Countries citing papers authored by Matthew J. Flick

Since Specialization

Citations

This map shows the geographic impact of Matthew J. Flick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew J. Flick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew J. Flick more than expected).

Fields of papers citing papers by Matthew J. Flick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew J. Flick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew J. Flick. The network helps show where Matthew J. Flick may publish in the future.

Co-authorship network of co-authors of Matthew J. Flick

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew J. Flick. A scholar is included among the top collaborators of Matthew J. Flick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew J. Flick. Matthew J. Flick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Münster, Anna‐Marie Bloch, Mette Munk Lauridsen, Matthew J. Flick, et al.. (2025). Plasma and tissue fibrin formation associates with severity of obesity and metabolic liver disease: 2-year follow-up after bariatric surgery. Journal of Thrombosis and Haemostasis. 23. S5–S6.

Sang, Yaqiu, Marzia Menegatti, Jennifer A. Brody, et al.. (2025). Plasminogen activation and plasmin activity are not necessary to prevent venous thrombosis/thromboembolism. Blood. 146(11). 1346–1358. 1 indexed citations

Lee, Robert H., Paul Y. Kim, Rafał Pawliński, et al.. (2024). 4D intravital imaging studies identify platelets as the predominant cellular procoagulant surface in a mouse hemostasis model. Blood. 144(10). 1116–1126. 7 indexed citations

Song, Zhenfeng, Mauricio Torres, Shuangcheng Alivia Wu, et al.. (2024). Regulation of hepatic inclusions and fibrinogen biogenesis by SEL1L-HRD1 ERAD. Nature Communications. 15(1). 9244–9244. 6 indexed citations

Donahue, Deborah L., Aleksandra Klim, Michael G. Caparon, et al.. (2024). Fibrinolytic-deficiencies predispose hosts to septicemia from a catheter-associated UTI. Nature Communications. 15(1). 2704–2704. 9 indexed citations

Sang, Yaqiu, Robert H. Lee, Ëva Katona, et al.. (2024). Activated platelets retain and protect most of their factor XIII-A cargo from proteolytic activation and degradation. Blood Advances. 8(19). 5072–5085. 2 indexed citations

Hernandez, Gloria E., Feiyang Ma, Guadalupe Martínez, et al.. (2022). Aortic intimal resident macrophages are essential for maintenance of the non-thrombogenic intravascular state. Nature Cardiovascular Research. 1(1). 67–84. 33 indexed citations

Paul, David S., Sarah E. Rowe, Jay L. Degen, et al.. (2022). Fibrin(ogen) engagement of S. aureus promotes the host antimicrobial response and suppression of microbe dissemination following peritoneal infection. PLoS Pathogens. 18(1). e1010227–e1010227. 14 indexed citations

Henderson, Michael W., Erica Sparkenbaugh, Shaobin Wang, et al.. (2021). Plasmin-mediated cleavage of high-molecular-weight kininogen contributes to acetaminophen-induced acute liver failure. Blood. 138(3). 259–272. 20 indexed citations

10.

Yang, Yi, Nadia A. Lanman, Brett P. Monia, et al.. (2019). Thrombin Signaling Promotes Pancreatic Adenocarcinoma through PAR-1–Dependent Immune Evasion. Cancer Research. 79(13). 3417–3430. 60 indexed citations

11.

Silva, Lakmali M., Zhen Gao, Matthew J. Flick, et al.. (2019). Plasmin-mediated fibrinolysis enables macrophage migration in a murine model of inflammation. Blood. 134(3). 291–303. 33 indexed citations

12.

Shukla, Samriddhi, David Milewski, Arun Pradhan, et al.. (2019). The FOXM1 Inhibitor RCM-1 Decreases Carcinogenesis and Nuclear β-Catenin. Molecular Cancer Therapeutics. 18(7). 1217–1229. 44 indexed citations

13.

Anokhin, Boris A., William L. Dean, Kerrie A. Smith, et al.. (2019). Proteolytic and nonproteolytic activation mechanisms result in conformationally and functionally different forms of coagulation factor XIII A. FEBS Journal. 287(3). 452–464. 15 indexed citations

14.

Ley, Carsten Dan, et al.. (2018). Absence of functional compensation between coagulation factor VIII and plasminogen in double-knockout mice. Blood Advances. 2(22). 3126–3136. 6 indexed citations

15.

Kopec, Anna K., Sherry Thornton, Joseph S. Palumbo, et al.. (2017). Thrombin promotes diet-induced obesity through fibrin-driven inflammation. Journal of Clinical Investigation. 127(8). 3152–3166. 92 indexed citations

16.

Shaw, Maureen A., Keith W. Kombrinck, Kathryn McElhinney, et al.. (2016). Limiting prothrombin activation to meizothrombin is compatible with survival but significantly alters hemostasis in mice. Blood. 128(5). 721–731. 10 indexed citations

17.

Adams, Gregory N., Keith W. Kombrinck, Kathryn McElhinney, et al.. (2015). Colon Cancer Growth and Dissemination Relies upon Thrombin, Stromal PAR-1, and Fibrinogen. Cancer Research. 75(19). 4235–4243. 98 indexed citations

18.

Turpin, Brian, Keith W. Kombrinck, Matthew J. Flick, et al.. (2014). Thrombin Drives Tumorigenesis in Colitis-Associated Colon Cancer. Cancer Research. 74(11). 3020–3030. 33 indexed citations

19.

Adams, Ryan A., Jan Bauer, Matthew J. Flick, et al.. (2007). The fibrin-derived γ377-395 peptide inhibits microglia activation and suppresses relapsing paralysis in central nervous system autoimmune disease. The Journal of Experimental Medicine. 204(3). 571–582. 268 indexed citations

20.

Flick, Matthew J., David P. Witte, Markéta Jiroušková, et al.. (2004). Leukocyte engagement of fibrin(ogen) via the integrin receptor αMβ2/Mac-1 is critical for host inflammatory response in vivo. Journal of Clinical Investigation. 113(11). 1596–1606. 342 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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