John F. Hancock
- Cell Biology top 0.02%
- Cellular transport and secretion 51
- Caveolin-1 and cellular processes 29
- Molecular Biology top 0.1%
- Protein Kinase Regulation and GTPase Signaling 93
- Lipid Membrane Structure and Behavior 37
- PI3K/AKT/mTOR signaling in cancer 17
- Cell death mechanisms and regulation 16
- Melanoma and MAPK Pathways 14
- Immunology and Allergy top 0.5%
- Biophysics top 0.5%
- Oncology top 1%
-
- Erythrocyte Function and Pathophysiology 29
- Co-authors
- Robert G. PartonIan A. PriorAnthony I. MageeHugh PatersonChristopher J. MarshallYong ZhouKaren A. CadwalladerSarah J. Plowman
- Partner nations
- United StatesAustraliaUnited Kingdom
In The Last Decade
John F. Hancock
208 papers receiving 21.2k citations
Hit Papers
Peers
Comparison fields: 5 of 161
- Cell Biology 7.7k
- Molecular Biology 17.8k
- Immunology and Allergy 658
- Biophysics 549
- Oncology 2.4k
Countries citing papers authored by John F. Hancock
This map shows the geographic impact of John F. Hancock's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John F. Hancock with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John F. Hancock more than expected).
Fields of papers citing papers by John F. Hancock
This network shows the impact of papers produced by John F. Hancock. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John F. Hancock. The network helps show where John F. Hancock may publish in the future.
Co-authorship network
The 25 scholars most cited alongside John F. Hancock, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 1 | |
| 2 | 2023 | 11 | |
| 3 | 2023 | 14 | |
| 4 | 2021 | 8 | |
| 5 | 2020 | 43 | |
| 6 | 2019 | 17 | |
| 7 | 2019 | 30 | |
| 8 | 2017 | 44 | |
| 9 | 2017 | 34 | |
| 10 | 2017 | 63 | |
| 11 | 2017 | 39 | |
| 12 | 2016 | 43 | |
| 13 | 2014 | 98 | |
| 14 | 2013 | 64 | |
| 15 | 2012 | 36 | |
| 16 | 2009 | 30 | |
| 17 | 2005 | 341 | |
| 18 | 1998 | 118 | |
| 19 | 1995 | 21 | |
| 20 | 1954 | 1 |
About John F. Hancock
John F. Hancock is a scholar working on Cell Biology, Molecular Biology and Physiology, having authored 210 papers that have together received 21.6k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (93 papers), Cellular transport and secretion (51 papers), Lipid Membrane Structure and Behavior (37 papers), Erythrocyte Function and Pathophysiology (29 papers), Caveolin-1 and cellular processes (29 papers), PI3K/AKT/mTOR signaling in cancer (17 papers), Cell death mechanisms and regulation (16 papers) and Melanoma and MAPK Pathways (14 papers). The work is most often cited by research in Cell Biology (7.7k citations), Molecular Biology (17.8k citations) and Immunology and Allergy (658 citations). John F. Hancock has collaborated with scholars based in United States, Australia and United Kingdom. Frequent co-authors include Robert G. Parton, Ian A. Prior, Anthony I. Magee, Hugh Paterson, Christopher J. Marshall, Yong Zhou, Karen A. Cadwallader, Sarah J. Plowman, Christopher J. Marshall and Alemayehu A. Gorfe. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.