Gregory J. Tesz
- Aging top 5%
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- Diet, Metabolism, and Disease 5
- Pharmaceutical Science top 5%
- Physiology top 10%
- Diet and metabolism studies 2
- Molecular Biology top 10%
- Metabolism, Diabetes, and Cancer 4
- RNA Interference and Gene Delivery 3
- Peroxisome Proliferator-Activated Receptors 3
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- Pancreatic function and diabetes 7
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- Liver Disease Diagnosis and Treatment 3
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- Regulation of Appetite and Obesity 2
- Co-authors
- Michael CzechMyriam AouadiSarah M. NicoloroErnesto R. SotoGary R. OstroffMengxi WangMy ChouinardJoshua D. Rabinowitz
- Partner nations
- United StatesNetherlandsSweden
In The Last Decade
Gregory J. Tesz
17 papers receiving 1.5k citations
Hit Papers
Peers
Comparison fields: 5 of 102
- Aging 68
- Endocrinology, Diabetes and Metabolism 399
- Pharmaceutical Science 84
- Physiology 345
- Molecular Biology 782
Countries citing papers authored by Gregory J. Tesz
This map shows the geographic impact of Gregory J. Tesz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gregory J. Tesz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gregory J. Tesz more than expected).
Fields of papers citing papers by Gregory J. Tesz
This network shows the impact of papers produced by Gregory J. Tesz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gregory J. Tesz. The network helps show where Gregory J. Tesz may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Gregory J. Tesz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 7 | |
| 2 | 2023 | 8 | |
| 3 | 2023 | 1 | |
| 4 | 2021 | 75 | |
| 5 | The Small Intestine Converts Dietary Fructose into Glucose and Organic Acidsbreakdown → | 2018 | 444 |
| 6 | 2011 | 81 | |
| 7 | 2011 | 98 | |
| 8 | 2009 | 140 | |
| 9 | 2009 | 45 | |
| 10 | 2009 | 461 | |
| 11 | 2007 | 77 | |
| 12 | 2005 | 40 | |
| 13 | 2004 | 25 | |
| 14 | 2004 | 33 | |
| 15 | 2003 | 7 | |
| 16 | 2001 | 28 | |
| 17 | 2001 | 12 |
About Gregory J. Tesz
Gregory J. Tesz is a scholar working on Aging, Endocrine and Autonomic Systems and Endocrinology, Diabetes and Metabolism, having authored 17 papers that have together received 1.6k indexed citations. Recurring topics across this work include Pancreatic function and diabetes (7 papers), Diet, Metabolism, and Disease (5 papers), Metabolism, Diabetes, and Cancer (4 papers), Liver Disease Diagnosis and Treatment (3 papers), RNA Interference and Gene Delivery (3 papers), Peroxisome Proliferator-Activated Receptors (3 papers), Diet and metabolism studies (2 papers) and Regulation of Appetite and Obesity (2 papers). The work is most often cited by research in Aging (68 citations), Endocrinology, Diabetes and Metabolism (399 citations) and Pharmaceutical Science (84 citations). Gregory J. Tesz has collaborated with scholars based in United States, Netherlands and Sweden. Frequent co-authors include Michael Czech, Myriam Aouadi, Sarah M. Nicoloro, Ernesto R. Soto, Gary R. Ostroff, Mengxi Wang, My Chouinard, Joshua D. Rabinowitz, Wenyun Lu and Morris J. Birnbaum. Their work appears in journals such as Nature, Cell and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.