Frank J. Kaiser

6.0k citations

83 papers · 1.9k indexed · h-index 26

Molecular Biology top 10%
Genetics top 5%
Immunology top 10%
Cellular and Molecular Neuroscience top 10%
Neurology top 5%

Co-authors: Brian E. Henderson Diana Braunholz Ilaria Parenti Jens Zerrahn Erwan Watrin Kerstin S. Wendt Steven C. Ley Gabriele Gillessen‐Kaesbach
Topics: Genomics and Chromatin Dynamics (21 papers)RNA Research and Splicing (20 papers)Genomic variations and chromosomal abnormalities (11 papers)
Cited by: Sensory Systems Immunology Neurology
Journals: Nucleic Acids Research Journal of Biological Chemistry Angewandte Chemie International Edition
Partner nations: Germany United Kingdom United States

In The Last Decade

Frank J. Kaiser

82 papers receiving 1.9k citations

Peers

Countries citing papers authored by Frank J. Kaiser

Since Specialization

Citations

This map shows the geographic impact of Frank J. Kaiser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frank J. Kaiser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frank J. Kaiser more than expected).

Fields of papers citing papers by Frank J. Kaiser

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frank J. Kaiser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frank J. Kaiser. The network helps show where Frank J. Kaiser may publish in the future.

Co-authorship network of co-authors of Frank J. Kaiser

This figure shows the co-authorship network connecting the top 25 collaborators of Frank J. Kaiser. A scholar is included among the top collaborators of Frank J. Kaiser based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frank J. Kaiser. Frank J. Kaiser is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Skeletal muscle vulnerability in a child with Pitt-Hopkins syndrome 2024 ·Skeletal Muscle ·(unknown),(unknown),Christel Depienne,Corinna Preuße,Adela Della Marina,André Reis,Frank J. Kaiser,Kay Nolte,Andreas Hentschel,Ulrike Schara‐Schmidt,Heike Kölbel,Andreas Roos	1
2	Cardiac recovery from pressure overload is not altered by thyroid hormone status in old mice 2024 ·Frontiers in Endocrinology ·(unknown),(unknown),(unknown),Georg Sebastian Hönes,(unknown),Jens Mittag,(unknown),Frank J. Kaiser,Lars C. Moeller,Kristina Lorenz,Dagmar Führer	1
3	Mosaic divergent repeat interruptions in XDP influence repeat stability and disease onset 2022 ·Brain ·Joanne Trinh,(unknown),Susen Schaake,Björn‐Hergen Laabs,(unknown),(unknown),Jelena Pozojevic,(unknown),Inke R. König,Roland Dominic G. Jamora,Raymond L. Rosales,Norbert Brüggemann,Gerard Saranza,Cid Czarina E. Diesta,Frank J. Kaiser,Christel Depienne,Christopher E. Pearson,Ana Westenberger,Christine Klein	15
4	G Protein-Coupled Receptor 15 Expression Is Associated with Myocardial Infarction 2022 ·International Journal of Molecular Sciences ·(unknown),Christian Müller,Bastian Stoffers,(unknown),Mélanie Waldenberger,Frank J. Kaiser,Mahir Karakas,Sangwon V. Kim,(unknown),Philipp S. Wild,Karl J. Lackner,Jonas Andersson,Stefan Söderberg,Diana Lindner,Tanja Zeller	7
5	Endocrine Evaluation and Homeostatic Model Assessment in Patients with Cornelia de Lange Syndrome 2022 ·Journal of Clinical Research in Pediatric Endocrinology ·(unknown),Ana Latorre,Beatriz Puisac,(unknown),María Arnedo,Ilaria Parenti,(unknown),(unknown),Ángel Matute-Llorente,(unknown),Frank J. Kaiser,Feliciano J. Ramos,Juan Pié,Gloria Bueno	2
6	MFSD2A-associated primary microcephaly - Expanding the clinical and mutational spectrum of this ultra-rare disease 2021 ·European Journal of Medical Genetics ·(unknown),Gökhan Yigit,(unknown),Janine Altmüller,Hölger Thiele,Peter Nürnberg,Nursel Elçioğlu,(unknown),Ute Hehr,Anja Stein,Adela Della Marina,Angela Köninger,Christel Depienne,Frank J. Kaiser,Bernd Wollnik,Alma Kuechler	6
7	Heterozygous de novo variants in CSNK1G1 are associated with syndromic developmental delay and autism spectrum disorder 2020 ·Clinical Genetics ·Nina B. Gold,Dong Li,Anna Chassevent,Frank J. Kaiser,Ilaria Parenti,Tim M. Strom,Feliciano J. Ramos,Beatriz Puisac,Juan Pié,Kirsty McWalter,María J. Guillen Sacoto,Hong Cui,(unknown),Constance Smith‐Hicks,Lance H. Rodan,Edward Blair,Elizabeth Bhoj	10
8	DNA Methylation as a Potential Molecular Mechanism in X‐linked Dystonia‐Parkinsonism 2020 ·Movement Disorders ·(unknown),Susen Schaake,Karen Grütz,(unknown),(unknown),Inke R. König,Björn‐Hergen Laabs,Roland Dominic G. Jamora,Raymond L. Rosales,Cid Czarina E. Diesta,Jelena Pozojevic,Timo Gemoll,Ana Westenberger,Frank J. Kaiser,Christine Klein,Henriette Kirchner	6
9	Chromatinopathies: A focus on Cornelia de Lange syndrome 2019 ·Clinical Genetics ·Laura Avagliano,Ilaria Parenti,(unknown),(unknown),Chiara Parodi,Milena Mariani,Frank J. Kaiser,Angelo Selicorni,Cristina Gervasini,Valentina Massa	34
10	mRNA Quantification of NIPBL Isoforms A and B in Adult and Fetal Human Tissues, and a Potentially Pathological Variant Affecting Only Isoform A in Two Patients with Cornelia de Lange Syndrome 2017 ·International Journal of Molecular Sciences ·Beatriz Puisac,(unknown),(unknown),(unknown),(unknown),Torkild Visnes,Christopher Bot,Paulino Gómez‐Puertas,Frank J. Kaiser,Feliciano J. Ramos,Lena Ström,Juan Pié	3
11	Increased percentages of regulatory T cells are associated with inflammatory and neuroendocrine responses to acute psychological stress and poorer health status in older men and women 2015 ·Psychopharmacology ·Amy Ronaldson,Ahmad Mahfuz Gazali,(unknown),Frank J. Kaiser,Stephen Thompson,Brian E. Henderson,Andrew Steptoe,Lívia A. Carvalho	28
12	THAP1, the gene mutated in DYT6 dystonia, autoregulates its own expression 2014 ·Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms ·(unknown),(unknown),Philip Seibler,Diana Braunholz,(unknown),Reinhard Depping,Juliane Eckhold,Thora Lohnau,Gabriele Gillessen‐Kaesbach,Anne Grünewald,Aleksandar Raković,Katja Lohmann,Frank J. Kaiser	18
13	Coronary Heart Disease-Associated Variation in TCF21 Disrupts a miR-224 Binding Site and miRNA-Mediated Regulation 2014 ·PLoS Genetics ·Clint L. Miller,(unknown),(unknown),Nicholas J. Leeper,Ramendra K. Kundu,Bhagat Patlolla,Themistocles L. Assimes,Frank J. Kaiser,Ljubica Perisic,Ulf Hedin,Lars Mäegdefessel,Heribert Schunkert,Jeanette Erdmann,Thomas Quertermous,Georg Sczakiel	87
14	Exome sequencing identifies compound heterozygous mutations in C12orf57 in two siblings with severe intellectual disability, hypoplasia of the corpus callosum, chorioretinal coloboma, and intractable seizures 2014 ·American Journal of Medical Genetics Part A ·Konrad Platzer,Irina Hüning,(unknown),Thomas Schwarzmayr,Rainer Gabriel,Tim M. Strom,Gabriele Gillessen‐Kaesbach,Frank J. Kaiser	6
15	Do reciprocal interactions between cell stress proteins and cytokines create a new intra-/extra-cellular signalling nexus? 2013 ·Cell Stress and Chaperones ·Brian E. Henderson,Frank J. Kaiser	17
16	The dystonia gene DYT1 is repressed by the transcription factor THAP1 (DYT6) 2010 ·Annals of Neurology ·Frank J. Kaiser,(unknown),Aleksandar Raković,(unknown),(unknown),Diana Braunholz,Thora Lohnau,Slobodanka Orolicki,Melanie Albrecht,Gabriele Gillessen‐Kaesbach,Christine Klein,Katja Lohmann	64
17	09-P024 The transcription factor Trps1 interacts with the activator form of Gli3 to regulate chondrocyte proliferation and differentiation 2009 ·Mechanisms of Development ·Manuela Wuelling,Frank J. Kaiser,Diana Braunholz,Reinhard Depping,Andrea Vortkamp	2
18	The Cohesin loading factor NIPBL recruits histone deacetylases to mediate local chromatin modifications 2008 ·Nucleic Acids Research ·(unknown),Weizhen Xu,Manuela Wülling,Melanie Albrecht,Holger Gabriel,Gabriele Gillessen‐Kaesbach,Frank J. Kaiser	45
19	IIGP, a member of the IFN inducible and microbial defense mediating 47 kDa GTPase family, interacts with the microtubule binding protein hook3 2004 ·Journal of Cell Science ·Frank J. Kaiser,Stefan H. E. Kaufmann,Jens Zerrahn	26
20	Nuclear interaction of the dynein light chain LC8a with the TRPS1 transcription factor suppresses the transcriptional repression activity of TRPS1 2003 ·Human Molecular Genetics ·Frank J. Kaiser	57

About Frank J. Kaiser

Frank J. Kaiser is a scholar working on Genetics, Molecular Biology and Aging, having authored 83 papers that have together received 1.9k indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (21 papers), RNA Research and Splicing (20 papers) and Genomic variations and chromosomal abnormalities (11 papers). The work is most often cited by research in Sensory Systems (78 citations), Immunology (322 citations) and Neurology (228 citations). Frank J. Kaiser has collaborated with scholars based in Germany, United Kingdom and United States. Frequent co-authors include Brian E. Henderson, Diana Braunholz, Ilaria Parenti, Jens Zerrahn, Erwan Watrin, Kerstin S. Wendt, Steven C. Ley, Gabriele Gillessen‐Kaesbach, Stamatia Papoutsopoulou and Anne O’Garra. Their work appears in journals such as Nucleic Acids Research, Journal of Biological Chemistry and Angewandte Chemie International Edition.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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