Bernd Wollnik

13.2k citations
143 papers · 4.4k · h-index 36

Impact in

Papers in

    • Connexins and lens biology 10
    • Hedgehog Signaling Pathway Studies 10
    • Ion channel regulation and function 9
    • Genomic variations and chromosomal abnormalities 12
    • Genomics and Rare Diseases 8

Bernd Wollnik

136 papers receiving 4.3k citations

Peers

Bernd Wollnik
Comparison fields: 5 of 113
  • Sensory Systems 384
  • Developmental Biology 121
  • Genetics 1.5k
  • Molecular Biology 2.5k
  • Cardiology and Cardiovascular Medicine 505
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Citations per year

Countries citing papers authored by Bernd Wollnik

Since Specialization
Citations

This map shows the geographic impact of Bernd Wollnik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bernd Wollnik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bernd Wollnik more than expected).

Fields of papers citing papers by Bernd Wollnik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bernd Wollnik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bernd Wollnik. The network helps show where Bernd Wollnik may publish in the future.

Co-authors

The 25 scholars most cited alongside Bernd Wollnik, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Bernd Wollnik Line = papers co-authored together Bernd Wollnik links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 143 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2003488
2 2011252
3 2012187
4 1997150
5 2011139
6 2012103
7 2010103
8 2001100
9 200497
10 200995
11 201893
12 200590
13 200990
14 199888
15 200388
16 201779
17 200763
18 199763
19 201062
20 200260

About Bernd Wollnik

Bernd Wollnik is a scholar working on Molecular Biology, Genetics, Cardiology and Cardiovascular Medicine, Genetics and Sensory Systems, having authored 143 papers that have together received 4.4k indexed citations. Recurring topics across this work include Genomic variations and chromosomal abnormalities (12 papers), Hearing, Cochlea, Tinnitus, Genetics (12 papers), Connexins and lens biology (10 papers), Hedgehog Signaling Pathway Studies (10 papers), Cardiac electrophysiology and arrhythmias (9 papers), Ion channel regulation and function (9 papers), Genomics and Rare Diseases (8 papers) and Cardiomyopathy and Myosin Studies (8 papers). The work is most often cited by research in Sensory Systems (384 citations), Developmental Biology (121 citations), Genetics (1.5k citations), Molecular Biology (2.5k citations) and Cardiology and Cardiovascular Medicine (505 citations). Bernd Wollnik has collaborated with scholars based in Germany, Türkiye and United States. Frequent co-authors include Nina Bögershausen, Christian Kubisch, Zehra Oya Uyguner, William A. Paznekas, Simeon A. Boyadjiev, Robert E. Shapiro, Ethylin Wang Jabs, Mary Beth Dinulos, Otto Daniëls and Jeffrey W. Innis. Their work appears in journals such as Clinical Genetics, Human Molecular Genetics, Human Genetics, The American Journal of Human Genetics and Human Mutation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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