Frank Eßmann

6.2k total citations · 1 hit paper
79 papers, 4.5k citations indexed

About

Frank Eßmann is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Frank Eßmann has authored 79 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 55 papers in Molecular Biology, 26 papers in Oncology and 15 papers in Immunology. Recurrent topics in Frank Eßmann's work include Cell death mechanisms and regulation (25 papers), Cancer-related Molecular Pathways (18 papers) and Immune Response and Inflammation (8 papers). Frank Eßmann is often cited by papers focused on Cell death mechanisms and regulation (25 papers), Cancer-related Molecular Pathways (18 papers) and Immune Response and Inflammation (8 papers). Frank Eßmann collaborates with scholars based in Germany, United States and United Kingdom. Frank Eßmann's co-authors include Klaus Schulze‐Osthoff, Reiner U. Jänicke, Peter T. Daniel, Bernd Dörken, Thomas Wieder, Dennis Sohn, Susanne Feil, Robert Łukowski, Birgit Fehrenbacher and Martin Schaller and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Angewandte Chemie International Edition.

In The Last Decade

Frank Eßmann

79 papers receiving 4.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Transdifferentiation of Vascular Smooth Muscle Cells to Macrophage-Like Cells During Atherogenesis

2014 413 citations Frank Eßmann, Klaus Schulze‐Osthoff et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Frank Eßmann Germany	40	2.7k	1.1k	840	685	459	79	4.5k
Latifa Bakiri Austria	37	2.5k 0.9×	1.0k 0.9×	699 0.8×	799 1.2×	473 1.0×	79	4.2k
Yaoqin Gong China	40	3.5k 1.3×	781 0.7×	663 0.8×	951 1.4×	373 0.8×	146	5.2k
Lin Xiao China	31	2.5k 0.9×	1.0k 1.0×	582 0.7×	598 0.9×	554 1.2×	87	4.2k
Yongjun Dang China	33	3.3k 1.2×	1.1k 1.0×	657 0.8×	724 1.1×	645 1.4×	101	5.2k
Hee Gu Lee South Korea	37	2.0k 0.7×	729 0.7×	854 1.0×	522 0.8×	454 1.0×	140	3.8k
Ping Wang China	37	2.2k 0.8×	806 0.8×	1.2k 1.4×	1.0k 1.5×	327 0.7×	206	4.5k
Joan Gil Spain	38	2.9k 1.0×	918 0.9×	557 0.7×	509 0.7×	441 1.0×	110	4.9k
Dooil Jeoung South Korea	42	3.0k 1.1×	693 0.7×	1.1k 1.3×	967 1.4×	286 0.6×	166	4.8k
Punita Dhawan United States	38	2.5k 0.9×	1.2k 1.2×	622 0.7×	1.0k 1.5×	275 0.6×	84	4.8k
Dae‐Yeul Yu South Korea	42	3.5k 1.3×	624 0.6×	886 1.1×	915 1.3×	842 1.8×	129	5.6k

Countries citing papers authored by Frank Eßmann

Since Specialization

Citations

This map shows the geographic impact of Frank Eßmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frank Eßmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frank Eßmann more than expected).

Fields of papers citing papers by Frank Eßmann

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Frank Eßmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frank Eßmann. The network helps show where Frank Eßmann may publish in the future.

Co-authorship network of co-authors of Frank Eßmann

This figure shows the co-authorship network connecting the top 25 collaborators of Frank Eßmann. A scholar is included among the top collaborators of Frank Eßmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Frank Eßmann. Frank Eßmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Nguyen, Dang, Bernhard Gillissen, Walter E. Aulitzky, et al.. (2024). BCL-2 and BOK regulate apoptosis by interaction of their C-terminal transmembrane domains. EMBO Reports. 25(9). 3896–3924. 7 indexed citations

Böpple, Kathrin, Yaara Oren, Whitney S. Henry, et al.. (2024). ATF3 characterizes aggressive drug-tolerant persister cells in HGSOC. Cell Death and Disease. 15(4). 290–290. 7 indexed citations

Eßmann, Frank, Heike Horn, Julia Schüler, et al.. (2023). Indolyl-chalcone derivatives trigger apoptosis in cisplatin-resistant mesothelioma cells through aberrant tubulin polymerization and deregulation of microtubule-associated proteins. Frontiers in Oncology. 13. 1190988–1190988. 3 indexed citations

Weller, Sandra, Kathrin Böpple, Ute Hofmann, et al.. (2022). The BCL-2 inhibitor ABT-199/venetoclax synergizes with proteasome inhibition via transactivation of the MCL-1 antagonist NOXA. Cell Death Discovery. 8(1). 215–215. 21 indexed citations

John, Katharina, Franziska Wandrer, Florian W. R. Vondran, et al.. (2021). BH3-only protein expression determines hepatocellular carcinoma response to sorafenib-based treatment. Cell Death and Disease. 12(8). 736–736. 12 indexed citations

Bucher, Philip, Anja Schmitt, Caroline Schönfeld, et al.. (2020). Threonine Phosphorylation of IκBζ Mediates Inhibition of Selective Proinflammatory Target Genes. Journal of Investigative Dermatology. 140(9). 1805–1814.e6. 6 indexed citations

Zhang, Yi, et al.. (2017). A New Acetylenic Compound and Other Bioactive Metabolites from a Shark Gill-derived Penicillium Strain. Records of Natural Products. 11(1). 7 indexed citations

Hailfinger, Stephan, Klaus Schulze‐Osthoff, Frank Eßmann, et al.. (2016). The Atypical Inhibitor of NF-kappa B, I kappa B zeta, Controls Macrophage Interleukin-10 Expression. Journal of Biological Chemistry. 291(24). 1 indexed citations

Sindlinger, Julia, Klaus Schulze‐Osthoff, Markus Hartl, et al.. (2015). Interrogating Substrate Selectivity and Composition of Endogenous Histone Deacetylase Complexes with Chemical Probes. Angewandte Chemie International Edition. 55(3). 1192–1195. 20 indexed citations

10.

Bareiss, Petra M., Anna Paczulla, Hui Wang, et al.. (2013). SOX2 Expression Associates with Stem Cell State in Human Ovarian Carcinoma. Cancer Research. 73(17). 5544–5555. 126 indexed citations

11.

Venturelli, Sascha, Alexander Berger, Timo Weiland, et al.. (2013). Differential Induction of Apoptosis and Senescence by the DNA Methyltransferase Inhibitors 5-Azacytidine and 5-Aza-2′-Deoxycytidine in Solid Tumor Cells. Molecular Cancer Therapeutics. 12(10). 2226–2236. 86 indexed citations

12.

Hildebrand, Dominic G., Eva Alexander, Simon Lehle, et al.. (2013). I kappa B zeta Is a Transcriptional Key Regulator of CCL2/MCP-1. The Journal of Immunology. 190(9). 3 indexed citations

13.

Lehle, Simon, Dominic G. Hildebrand, Michael Becker, et al.. (2013). LORD-Q: a long-run real-time PCR-based DNA-damage quantification method for nuclear and mitochondrial genome analysis. Nucleic Acids Research. 42(6). e41–e41. 45 indexed citations

14.

Weiland, Timo, Alexander Berger, Frank Eßmann, et al.. (2011). Kinetic Tracking of Therapy-Induced Senescence Using the Real-Time Cell Analyzer Single Plate System. Assay and Drug Development Technologies. 10(3). 289–295. 13 indexed citations

15.

Werdehausen, Robert, Sebastian Braun, Frank Eßmann, et al.. (2007). Lidocaine Induces Apoptosis via the Mitochondrial Pathway Independently of Death Receptor Signaling. Anesthesiology. 107(1). 136–143. 111 indexed citations

16.

Sohn, Dennis, Frank Eßmann, Klaus Schulze‐Osthoff, & Reiner U. Jänicke. (2006). p21 Blocks Irradiation-Induced Apoptosis Downstream of Mitochondria by Inhibition of Cyclin-Dependent Kinase–Mediated Caspase-9 Activation. Cancer Research. 66(23). 11254–11262. 103 indexed citations

17.

Eßmann, Frank, et al.. (2005). Irradiation-induced Translocation of p53 to Mitochondria in the Absence of Apoptosis. Journal of Biological Chemistry. 280(44). 37169–37177. 52 indexed citations

18.

Eßmann, Frank, Ingo H. Engels, Gudrun Totzke, Klaus Schulze‐Osthoff, & Reiner U. Jänicke. (2004). Apoptosis Resistance of MCF-7 Breast Carcinoma Cells to Ionizing Radiation Is Independent of p53 and Cell Cycle Control but Caused by the Lack of Caspase-3 and a Caffeine-Inhibitable Event. Cancer Research. 64(19). 7065–7072. 94 indexed citations

19.

Eßmann, Frank, Heike Bantel, Gudrun Totzke, et al.. (2003). Staphylococcus aureus α-toxin-induced cell death: predominant necrosis despite apoptotic caspase activation. Cell Death and Differentiation. 10(11). 1260–1272. 101 indexed citations

20.

Daniel, Peter T., Christian Schölz, Frank Eßmann, et al.. (1999). CD95/Fas-triggered apoptosis of activated T lymphocytes is prevented by dendritic cells through a CD58-dependent mechanism. Experimental Hematology. 27(9). 1402–1408. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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