Benjamin Dannenmann

460 total citations
22 papers, 238 citations indexed

About

Benjamin Dannenmann is a scholar working on Genetics, Molecular Biology and Oncology. According to data from OpenAlex, Benjamin Dannenmann has authored 22 papers receiving a total of 238 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Genetics, 9 papers in Molecular Biology and 9 papers in Oncology. Recurrent topics in Benjamin Dannenmann's work include Blood disorders and treatments (11 papers), Neutropenia and Cancer Infections (8 papers) and CRISPR and Genetic Engineering (6 papers). Benjamin Dannenmann is often cited by papers focused on Blood disorders and treatments (11 papers), Neutropenia and Cancer Infections (8 papers) and CRISPR and Genetic Engineering (6 papers). Benjamin Dannenmann collaborates with scholars based in Germany, Austria and Japan. Benjamin Dannenmann's co-authors include Klaus Schulze‐Osthoff, Julia Skokowa, Frank Eßmann, Simon Lehle, Oliver C. Rothfuss, Karl Welte, Katharina Holzer, Dominic G. Hildebrand, Maksim Klimiankou and Yun Xu and has published in prestigious journals such as Blood, Cardiovascular Research and Molecular Therapy.

In The Last Decade

Benjamin Dannenmann

20 papers receiving 236 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Benjamin Dannenmann Germany 9 133 36 35 32 32 22 238
Tomohiro Miyai Japan 6 117 0.9× 45 1.3× 43 1.2× 71 2.2× 29 0.9× 9 383
Deborah Lavin United Kingdom 7 120 0.9× 32 0.9× 32 0.9× 12 0.4× 16 0.5× 12 229
F. Colleoni Italy 11 169 1.3× 26 0.7× 33 0.9× 15 0.5× 18 0.6× 18 352
Taro Miyagawa Japan 8 135 1.0× 22 0.6× 39 1.1× 10 0.3× 15 0.5× 21 234
Gülay Güleç Ceylan Türkiye 9 149 1.1× 43 1.2× 13 0.4× 12 0.4× 63 2.0× 34 268
Dianna C. Martin Canada 7 142 1.1× 17 0.5× 18 0.5× 11 0.3× 27 0.8× 7 248
Prethish Sreenivas United States 6 170 1.3× 20 0.6× 18 0.5× 8 0.3× 37 1.2× 8 223
Alice Rousseau France 6 126 0.9× 20 0.6× 19 0.5× 28 0.9× 27 0.8× 9 233
Timna Varela Martins Brazil 6 122 0.9× 109 3.0× 37 1.1× 10 0.3× 22 0.7× 9 277

Countries citing papers authored by Benjamin Dannenmann

Since Specialization
Citations

This map shows the geographic impact of Benjamin Dannenmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Dannenmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Dannenmann more than expected).

Fields of papers citing papers by Benjamin Dannenmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Dannenmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Dannenmann. The network helps show where Benjamin Dannenmann may publish in the future.

Co-authorship network of co-authors of Benjamin Dannenmann

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin Dannenmann. A scholar is included among the top collaborators of Benjamin Dannenmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin Dannenmann. Benjamin Dannenmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kadam, Vaibhavi, Patrick Oeckl, Benjamin Dannenmann, et al.. (2025). Most L1CAM Is not Associated with Extracellular Vesicles in Human Biofluids and iPSC–Derived Neurons. Molecular Neurobiology. 62(8). 10427–10442. 4 indexed citations
2.
Dannenmann, Benjamin, et al.. (2024). Comparison of Gene-Editing Approaches for Severe Congenital Neutropenia-Causing Mutations in the ELANE Gene. The CRISPR Journal. 7(5). 258–271. 3 indexed citations
3.
Dannenmann, Benjamin, Yun Xu, Maksim Klimiankou, et al.. (2024). CRISPR-Cas9n-mediated ELANE promoter editing for gene therapy of severe congenital neutropenia. Molecular Therapy. 32(6). 1628–1642. 7 indexed citations
4.
Dannenmann, Benjamin, Frank Bernhard, Siarhei Kandabarau, et al.. (2024). Flavopiridol restores granulopoiesis in experimental models of severe congenital neutropenia. Molecular Therapy. 33(6). 2851–2871.
5.
Dannenmann, Benjamin, Maksim Klimiankou, Siarhei Kandabarau, et al.. (2023). Differential transcriptional control of hematopoiesis in congenital and cycling neutropenia patients harboring <i>ELANE</i> mutations. Haematologica. 109(5). 1393–1402. 1 indexed citations
6.
Dannenmann, Benjamin & Julia Skokowa. (2022). Generation, expansion, and drug treatment of hematopoietic progenitor cells derived from human iPSCs. STAR Protocols. 3(2). 101400–101400.
7.
8.
Klimiankou, Maksim, Benjamin Dannenmann, Ingeborg Steiert, et al.. (2020). Efficient Correction of HAX1 Mutations in Primary HSPCs of Severe Congenital Neutropenia Patients Using CRISPR/CAS9 GENE-Editing. Blood. 136(Supplement 1). 22–22. 1 indexed citations
9.
Dannenmann, Benjamin, et al.. (2020). CRISPR/Cas9 Genome Editing of Human-Induced Pluripotent Stem Cells Followed by Granulocytic Differentiation. Methods in molecular biology. 2115. 471–483. 10 indexed citations
10.
Rohlfing, Anne‐Katrin, Benjamin Dannenmann, Tatsiana Castor, et al.. (2020). The chemokine CXCL14 mediates platelet function and migration via direct interaction with CXCR4. Cardiovascular Research. 117(3). 903–917. 35 indexed citations
11.
Dannenmann, Benjamin, Maksim Klimiankou, Tatsuya Morishima, et al.. (2019). Human iPSC-based model of severe congenital neutropenia reveals elevated UPR and DNA damage in CD34+ cells preceding leukemic transformation. Experimental Hematology. 71. 51–60. 14 indexed citations
12.
Dannenmann, Benjamin, Yun Xu, Klaus Schulze‐Osthoff, et al.. (2019). Fluorescent labeling of CRISPR/Cas9 RNP for gene knockout in HSPCs and iPSCs reveals an essential role for GADD45b in stress response. Blood Advances. 3(1). 63–71. 15 indexed citations
13.
Klimiankou, Maksim, Narges Aghaallaei, Benjamin Dannenmann, et al.. (2019). Efficient Correction of ELANE mutations in Primary HSPCs of Severe Congenital Neutropenia Patients Using CRISPR/Cas9 and rAVV6 HDR Repair Templates. Blood. 134(Supplement_1). 1036–1036. 3 indexed citations
14.
Nocker, Andreas, et al.. (2018). Assessment of UV-C-induced water disinfection by differential PCR-based quantification of bacterial DNA damage. Journal of Microbiological Methods. 149. 89–95. 15 indexed citations
15.
Dannenmann, Benjamin, Maksim Klimiankou, Michael A. Rieger, et al.. (2018). GADD45b Plays an Essential Role in the G-CSF Triggered Granulocytic Differentiation of Human Hematopoietic Cells. Blood. 132(Supplement 1). 17–17. 1 indexed citations
16.
Dannenmann, Benjamin, Maksim Klimiankou, Christian Lindner, et al.. (2017). BAALC Is a Key Mediator of Leukemia Development in Congenital Neutropenia. Blood. 130. 541–541. 1 indexed citations
17.
Klimiankou, Maksim, Benjamin Dannenmann, Cornelia Zeidler, et al.. (2017). Effects of CSF3R mutations on Myeloid Differentiation and Proliferation of Hematopoietic Cells of Congenital Neutropenia Patients. Blood. 130. 2278–2278. 1 indexed citations
18.
Dannenmann, Benjamin, et al.. (2017). Simultaneous quantification of DNA damage and mitochondrial copy number by long-run DNA-damage quantification (LORD-Q). Oncotarget. 8(68). 112417–112425. 16 indexed citations
19.
Dannenmann, Benjamin, Simon Lehle, Dominic G. Hildebrand, et al.. (2015). High Glutathione and Glutathione Peroxidase-2 Levels Mediate Cell-Type-Specific DNA Damage Protection in Human Induced Pluripotent Stem Cells. Stem Cell Reports. 4(5). 886–898. 72 indexed citations
20.
Morishima, Tatsuya, et al.. (2015). SIRT2 Plays Essential Role in Early Hematopoiesis through Deacetylation of LMO2 Protein. Blood. 126(23). 3574–3574. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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