Ingo H. Engels

12.0k total citations · 1 hit paper
23 papers, 2.7k citations indexed

About

Ingo H. Engels is a scholar working on Molecular Biology, Oncology and Immunology. According to data from OpenAlex, Ingo H. Engels has authored 23 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 7 papers in Oncology and 4 papers in Immunology. Recurrent topics in Ingo H. Engels's work include Cell death mechanisms and regulation (11 papers), DNA Repair Mechanisms (4 papers) and RNA Interference and Gene Delivery (3 papers). Ingo H. Engels is often cited by papers focused on Cell death mechanisms and regulation (11 papers), DNA Repair Mechanisms (4 papers) and RNA Interference and Gene Delivery (3 papers). Ingo H. Engels collaborates with scholars based in Germany, United States and Singapore. Ingo H. Engels's co-authors include Klaus Schulze‐Osthoff, Sebastian Wesselborg, Reiner U. Jänicke, Marek Łoś, Kirsten Lauber, Christoph P. Berg, Christopher Stroh, Quinn L. Deveraux, Heike Bantel and Gudrun Totzke and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

Ingo H. Engels

23 papers receiving 2.7k citations

Hit Papers

Chemical activation of the mechanotransduction channel Pi... 2015 2026 2018 2022 2015 100 200 300 400

Peers

Ingo H. Engels
Tzipora Goldkorn United States
Ganesh M. Sathe United States
Lily Huang United States
Raghothama Chaerkady United States
Doris A. Wall United States
Mary Ann Accavitti United States
Ryuji Higashikubo United States
Ingo H. Engels
Citations per year, relative to Ingo H. Engels Ingo H. Engels (= 1×) peers Yaël Zermati

Countries citing papers authored by Ingo H. Engels

Since Specialization
Citations

This map shows the geographic impact of Ingo H. Engels's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ingo H. Engels with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ingo H. Engels more than expected).

Fields of papers citing papers by Ingo H. Engels

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ingo H. Engels. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ingo H. Engels. The network helps show where Ingo H. Engels may publish in the future.

Co-authorship network of co-authors of Ingo H. Engels

This figure shows the co-authorship network connecting the top 25 collaborators of Ingo H. Engels. A scholar is included among the top collaborators of Ingo H. Engels based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ingo H. Engels. Ingo H. Engels is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Syeda, Ruhma, Jie Xu, Adrienne E. Dubin, et al.. (2015). Chemical activation of the mechanotransduction channel Piezo1. eLife. 4. 487 indexed citations breakdown →
2.
Johnson, Steven M., Puiying A. Mak, Ingo H. Engels, et al.. (2014). A biochemical screen for GroEL/GroES inhibitors. Bioorganic & Medicinal Chemistry Letters. 24(3). 786–789. 34 indexed citations
3.
Ramadoss, Nitya S., John N. Alumasa, Lin Cheng, et al.. (2013). Small molecule inhibitors of trans -translation have broad-spectrum antibiotic activity. Proceedings of the National Academy of Sciences. 110(25). 10282–10287. 63 indexed citations
4.
Vargas, Leonardo, Michael Garcia, Yury J. Sigal, et al.. (2010). HTS-Compatible Patient-Derived Cell-Based Assay to Identify Small Molecule Modulators of Aberrant Splicing in Myotonic Dystrophy Type 1. PubMed. 4. 9–18. 16 indexed citations
5.
Engels, Ingo H., Truc T. Huynh, Hugo D. Urbina, et al.. (2009). A time-resolved fluorescence resonance energy transfer-based assay for DEN1 peptidase activity. Analytical Biochemistry. 390(1). 85–87. 16 indexed citations
6.
Anderson, Paul, Buu P. Tu, Genevieve Welch, et al.. (2009). Identification of Small Molecule and Genetic Modulators of AON-Induced Dystrophin Exon Skipping by High-Throughput Screening. PLoS ONE. 4(12). e8348–e8348. 30 indexed citations
7.
Buddenkotte, Joerg, Christopher Stroh, Ingo H. Engels, et al.. (2005). Agonists of Proteinase-Activated Receptor-2 Stimulate Upregulation of Intercellular Cell Adhesion Molecule-1 in Primary Human Keratinocytes via Activation of NF-kappa B. Journal of Investigative Dermatology. 124(1). 38–45. 110 indexed citations
8.
Wang, Yan, Ingo H. Engels, Deborah Knee, et al.. (2004). Synthetic lethal targeting of MYC by activation of the DR5 death receptor pathway. Cancer Cell. 5(5). 501–512. 131 indexed citations
9.
Müller, Frank U., Karin Loser, Joachim Neumann, et al.. (2004). Transcription factor AP-2α triggers apoptosis in cardiac myocytes. Cell Death and Differentiation. 11(5). 485–493. 17 indexed citations
10.
Eßmann, Frank, Ingo H. Engels, Gudrun Totzke, Klaus Schulze‐Osthoff, & Reiner U. Jänicke. (2004). Apoptosis Resistance of MCF-7 Breast Carcinoma Cells to Ionizing Radiation Is Independent of p53 and Cell Cycle Control but Caused by the Lack of Caspase-3 and a Caffeine-Inhibitable Event. Cancer Research. 64(19). 7065–7072. 94 indexed citations
11.
Wagner, Klaus W., Ingo H. Engels, & Quinn L. Deveraux. (2004). Caspase-2 Can Function Upstream of Bid Cleavage in the TRAIL Apoptosis Pathway. Journal of Biological Chemistry. 279(33). 35047–35052. 98 indexed citations
12.
Eßmann, Frank, Heike Bantel, Gudrun Totzke, et al.. (2003). Staphylococcus aureus α-toxin-induced cell death: predominant necrosis despite apoptotic caspase activation. Cell Death and Differentiation. 10(11). 1260–1272. 101 indexed citations
13.
Berg, Christoph P., Kirsten Lauber, Gerburg M. Stein, et al.. (2003). Tributyltin (TBT) induces ultra-rapid caspase activation independent of apoptosome formation in human platelets. Oncogene. 22(5). 775–780. 11 indexed citations
14.
Seeliger, Stephan, Thomas Vogl, Ingo H. Engels, et al.. (2003). Expression of Calcium-Binding Proteins MRP8 and MRP14 in Inflammatory Muscle Diseases. American Journal Of Pathology. 163(3). 947–956. 68 indexed citations
15.
Jänicke, Reiner U., Ingo H. Engels, Torsten Dunkern, et al.. (2001). Ionizing radiation but not anticancer drugs causes cell cycle arrest and failure to activate the mitochondrial death pathway in MCF-7 breast carcinoma cells. Oncogene. 20(36). 5043–5053. 71 indexed citations
16.
Stępczyńska, Anna, Kirsten Lauber, Ingo H. Engels, et al.. (2001). Staurosporine and conventional anticancer drugs induce overlapping, yet distinct pathways of apoptosis and caspase activation. Oncogene. 20(10). 1193–1202. 133 indexed citations
17.
Łoś, Marek, Christopher Stroh, Reiner U. Jänicke, Ingo H. Engels, & Klaus Schulze‐Osthoff. (2001). Caspases: more than just killers?. Trends in Immunology. 22(1). 31–34. 148 indexed citations
18.
Berg, Christoph P., Ingo H. Engels, Kirsten Lauber, et al.. (2001). Human mature red blood cells express caspase-3 and caspase-8, but are devoid of mitochondrial regulators of apoptosis. Cell Death and Differentiation. 8(12). 1197–1206. 316 indexed citations
19.
Engels, Ingo H., Christopher Stroh, Kirsten Lauber, et al.. (2000). Caspase-8/FLICE functions as an executioner caspase in anticancer drug-induced apoptosis. Oncogene. 19(40). 4563–4573. 231 indexed citations
20.
Bantel, Heike, Ingo H. Engels, Wolfgang Voelter, Klaus Schulze‐Osthoff, & Sebastian Wesselborg. (1999). Mistletoe lectin activates caspase-8/FLICE independently of death receptor signaling and enhances anticancer drug-induced apoptosis.. PubMed. 59(9). 2083–90. 169 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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