Eva M. Pålsson‐McDermott

13.4k citations

35 papers · 7.2k indexed · 6 hit papers · h-index 27

Immunology top 0.5%
- Immune cells in cancer 12
- Immune Response and Inflammation 11
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 5
- interferon and immune responses 4
- Immune Cell Function and Interaction 4
Cancer Research top 0.5%
- NF-κB Signaling Pathways 6
- Cancer, Hypoxia, and Metabolism 4
Biological Psychiatry top 2%
Endocrine and Autonomic Systems top 2%
- Circadian rhythm and melatonin 3
Neurology top 2%

Co-authors: Luke O'neill Frederick J. Sheedy Ciana Diskin Derek Johnson Elizabeth J. Hennessy Qingguo Ruan Youhai Chen Annie M. Curtis
Cited by: Immunology Cancer Research Biological Psychiatry
Journals: Nature (1 paper)Proceedings of the National Academy of Sciences (4 papers)Journal of Biological Chemistry (3 papers)
Partner nations: Ireland United States United Kingdom

In The Last Decade

Eva M. Pålsson‐McDermott

34 papers receiving 7.2k citations

Hit Papers

6 papers align trajectories log scale

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

2025 Cell Metabolism
2020 Cell Research
2015 Cell Metabolism
2009 Nature Immunology
2004 Immunology
2001 Nature

Peers

Countries citing papers authored by Eva M. Pålsson‐McDermott

Since Specialization

Citations

This map shows the geographic impact of Eva M. Pålsson‐McDermott's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva M. Pålsson‐McDermott with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva M. Pålsson‐McDermott more than expected).

Fields of papers citing papers by Eva M. Pålsson‐McDermott

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva M. Pålsson‐McDermott. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva M. Pålsson‐McDermott. The network helps show where Eva M. Pålsson‐McDermott may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Eva M. Pålsson‐McDermott, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Eva M. Pålsson‐McDermott Line = papers co-authored together Eva M. Pålsson‐McDermott links everyone, so they are left out of the graph.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

#	Work
1	Gang of 3: How the Krebs cycle-linked metabolites itaconate, succinate, and fumarate regulate macrophages and inflammationbreakdown → Cell Metabolism ·Eva M. Pålsson‐McDermott,Luke O'neill	2025	20
2	Beyond energy: how TCA cycle-derived metabolites regulate gene expression and inflammation in the nucleus Journal of Inflammation ·J. S. Randhawa,Eva M. Pålsson‐McDermott	2025	0
3	Metabolic reprogramming of macrophages by PKM2 promotes IL-10 production via adenosine Cell Reports ·Juliana E. Toller-Kawahisa,Paula Ramos Viacava,Eva M. Pålsson‐McDermott,Daniele C. Nascimento,Mariana P. Cervantes‐Silva,Shane M. O’Carroll,Alessia Zotta,Luis Eduardo Alves Damasceno,Gabriel Azevedo Públio,Pedro K K Forti,João Paulo Mesquita Luiz,Bruno Marcel Silva de Melo,Timna Varela Martins,Vítor M. Faça,Annie M. Curtis,Thiago M. Cunha,Fernando Q. Cunha,Luke O'neill,José C. Alves‐Filho	2025	11
4	Targeting immunometabolism as an anti-inflammatory strategybreakdown → Cell Research ·Eva M. Pålsson‐McDermott,Luke O'neill	2020	364
5	Specific Inhibition of the NLRP3 Inflammasome as an Antiinflammatory Strategy in Cystic Fibrosis American Journal of Respiratory and Critical Care Medicine ·Oliver J. McElvaney,Zbigniew Zasłona,Katrin Anne Becker-Flegler,Eva M. Pålsson‐McDermott,Fiona Boland,Cedric Gunaratnam,Erich Gulbins,Luke O'neill,Emer P. Reeves,Noel G. McElvaney	2019	75
6	Malonylation of GAPDH is an inflammatory signal in macrophages Nature Communications ·Silvia Galván-Peña,Richard G. Carroll,Carla F. Newman,Elizabeth C. Hinchy,Eva M. Pålsson‐McDermott,Elektra K. Robinson,Sergio Covarrubias,Alan Nadin,Andrew M. James,Moritz Haneklaus,Susan Carpenter,Vincent P. Kelly,Michael P. Murphy,Louise K. Modis,Luke O'neill	2019	155
7	Metabolic Modulation in Macrophage Effector Function Frontiers in Immunology ·Ciana Diskin,Eva M. Pålsson‐McDermott	2018	227
8	Pyruvate Kinase M2: A Potential Target for Regulating Inflammation Frontiers in Immunology ·José C. Alves‐Filho,Eva M. Pålsson‐McDermott	2016	137
9	Cutting Edge: Mycobacterium tuberculosis Induces Aerobic Glycolysis in Human Alveolar Macrophages That Is Required for Control of Intracellular Bacillary Replication The Journal of Immunology ·Laura E. Gleeson,Frederick J. Sheedy,Eva M. Pålsson‐McDermott,Denise Triglia,Seónadh M. O’Leary,Mary P. O’Sullivan,Luke O'neill,Joseph Keane	2016	220
10	Pyruvate Kinase M2 Regulates Hif-1α Activity and IL-1β Induction and Is a Critical Determinant of the Warburg Effect in LPS-Activated Macrophagesbreakdown → Cell Metabolism ·Eva M. Pålsson‐McDermott,Annie M. Curtis,Gautam Goel,Mario Lauterbach,Frederick J. Sheedy,Laura E. Gleeson,Mirjam W.M. van den Bosch,Susan R. Quinn,Raquel Domingo-Fernandéz,Daniel Johnston,Jian‐kang Jiang,William J. Israelsen,Joseph Keane,Craig J. Thomas,Clary B. Clish,Matthew G. Vander Heiden,Ramnik J. Xavier,Luke O'neill	2015	1029
11	LPS Induces the Degradation of Programmed Cell Death Protein 4 (PDCD4) to Release Twist2, Activating c-Maf Transcription to Promote Interleukin-10 Production Journal of Biological Chemistry ·Mirjam W.M. van den Bosch,Eva M. Pålsson‐McDermott,Derek Johnson,Luke O'neill	2014	48
12	The Warburg effect then and now: From cancer to inflammatory diseases BioEssays ·Eva M. Pålsson‐McDermott,Luke O'neill	2013	404
13	MyD88 adaptor-like (Mal) functions in the epithelial barrier and contributes to intestinal integrity via protein kinase C Mucosal Immunology ·Sinéad C. Corr,Eva M. Pålsson‐McDermott,Irina Grishina,Sean Barry,Gabriella Aviello,Nicholas J. Bernard,Patrick G. Casey,Joseph B. J. Ward,Stephen J. Keely,Satya Dandekar,Padraic G. Fallon,Luke O'neill	2013	28
14	TAG, a splice variant of the adaptor TRAM, negatively regulates the adaptor MyD88–independent TLR4 pathway Nature Immunology ·Eva M. Pålsson‐McDermott,Sarah Doyle,Anne F. McGettrick,Matthew P. Hardy,Harald Husebye,Kathy Banahan,Mei Gong,Douglas T. Golenbock,Terje Espevik,Luke O'neill	2009	113
15	Glucocorticoids Inhibit IRF3 Phosphorylation in Response to Toll-like Receptor-3 and -4 by Targeting TBK1 Activation Journal of Biological Chemistry ·Claire E. McCoy,Susan Carpenter,Eva M. Pålsson‐McDermott,Linden J. Gearing,Luke O'neill	2008	68
16	The potential of targeting Toll-like receptor 2 in autoimmune and inflammatory diseases Irish Journal of Medical Science (1971 -) ·Eva M. Pålsson‐McDermott,Luke O'neill	2007	38
17	Trif-related adapter molecule is phosphorylated by PKCε during Toll-like receptor 4 signaling Proceedings of the National Academy of Sciences ·Anne F. McGettrick,Elizabeth Brint,Eva M. Pålsson‐McDermott,Daniel C. Rowe,Douglas T. Golenbock,Nicholas J. Gay,Katherine A. Fitzgerald,Luke O'neill	2006	112
18	Signal transduction by the lipopolysaccharide receptor, Toll‐like receptor‐4breakdown → Immunology ·Eva M. Pålsson‐McDermott,Luke O'neill	2004	1012
19	Ras Participates in the Activation of p38 MAPK by Interleukin-1 by Associating with IRAK, IRAK2, TRAF6, and TAK-1 Journal of Biological Chemistry ·Eva M. Pålsson‐McDermott,Luke O'neill	2002	102
20	Mal (MyD88-adapter-like) is required for Toll-like receptor-4 signal transductionbreakdown → Nature ·Katherine A. Fitzgerald,Eva M. Pålsson‐McDermott,Andrew Bowie,Caroline A. Jefferies,Ashley Mansell,Gareth Brady,Elizabeth Brint,Aisling Dunne,Pearl Gray,Mary T. Harte,Diane McMurray,Dirk E. Smith,John E. Sims,Timothy A. Bird,Luke O'neill	2001	1006

About Eva M. Pålsson‐McDermott

Eva M. Pålsson‐McDermott is a scholar working on Immunology, Biological Psychiatry and Cancer Research, having authored 35 papers that have together received 7.2k indexed citations. Recurring topics across this work include Immune cells in cancer (12 papers), Immune Response and Inflammation (11 papers), NF-κB Signaling Pathways (6 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (5 papers), interferon and immune responses (4 papers), Cancer, Hypoxia, and Metabolism (4 papers), Immune Cell Function and Interaction (4 papers) and Circadian rhythm and melatonin (3 papers). The work is most often cited by research in Immunology (3.7k citations), Cancer Research (1.7k citations) and Biological Psychiatry (174 citations). Eva M. Pålsson‐McDermott has collaborated with scholars based in Ireland, United States and United Kingdom. Frequent co-authors include Luke O'neill, Frederick J. Sheedy, Ciana Diskin, Derek Johnson, Elizabeth J. Hennessy, Qingguo Ruan, Youhai Chen, Annie M. Curtis, Cara Martin and John O’Leary. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact