Emilio Boada-Romero

7.9k total citations · 1 hit paper
14 papers, 1.4k citations indexed

About

Emilio Boada-Romero is a scholar working on Epidemiology, Immunology and Molecular Biology. According to data from OpenAlex, Emilio Boada-Romero has authored 14 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Epidemiology, 8 papers in Immunology and 4 papers in Molecular Biology. Recurrent topics in Emilio Boada-Romero's work include Autophagy in Disease and Therapy (9 papers), Phagocytosis and Immune Regulation (5 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Emilio Boada-Romero is often cited by papers focused on Autophagy in Disease and Therapy (9 papers), Phagocytosis and Immune Regulation (5 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Emilio Boada-Romero collaborates with scholars based in United States, Spain and United Kingdom. Emilio Boada-Romero's co-authors include Douglas R. Green, Bradlee L. Heckmann, Jennifer Martinez, Larissa D. Cunha, Joëlle Magné, Felipe X. Pimentel‐Muiños, Clifford S. Guy, Mao Yang, Michal Letek and Charles Gawad and has published in prestigious journals such as Cell, Nature Communications and Nature Reviews Molecular Cell Biology.

In The Last Decade

Emilio Boada-Romero

13 papers receiving 1.4k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The clearance of dead cells by efferocytosis

2020 548 citations Emilio Boada-Romero, Jennifer Martinez et al. Nature Reviews Molecular Cell Biology profile →

Author Peers

Peers are selected by citation overlap in the author's most active subfields. citations · hero ref

Author						Papers	Cites
Emilio Boada-Romero	659	540	499	209	133	14	1.4k
Ivan Peng	535 0.8×	277 0.5×	444 0.9×	78 0.4×	86 0.6×	12	1.2k
Wei Jia	434 0.7×	548 1.0×	436 0.9×	62 0.3×	116 0.9×	22	1.1k
Thomas Riffelmacher	450 0.7×	440 0.8×	486 1.0×	89 0.4×	50 0.4×	18	1.1k
Zuojia Chen	817 1.2×	289 0.5×	628 1.3×	129 0.6×	52 0.4×	36	1.6k
Xavier Michelet	737 1.1×	477 0.9×	347 0.7×	252 1.2×	231 1.7×	26	1.4k
Ignacio Benedicto	237 0.4×	348 0.6×	642 1.3×	91 0.4×	192 1.4×	37	1.6k
Hirotada Kojima	663 1.0×	225 0.4×	791 1.6×	147 0.7×	54 0.4×	18	1.4k
Yuexi Gu	308 0.5×	805 1.5×	649 1.3×	159 0.8×	331 2.5×	16	1.4k
Kathleen A. McPhillips	1.4k 2.2×	198 0.4×	573 1.1×	386 1.8×	172 1.3×	9	1.9k
Heather H. Pua	909 1.4×	1.2k 2.2×	1.1k 2.2×	202 1.0×	155 1.2×	36	2.4k

Countries citing papers authored by Emilio Boada-Romero

Since Specialization

Citations

This map shows the geographic impact of Emilio Boada-Romero's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emilio Boada-Romero with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emilio Boada-Romero more than expected).

Fields of papers citing papers by Emilio Boada-Romero

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Emilio Boada-Romero. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emilio Boada-Romero. The network helps show where Emilio Boada-Romero may publish in the future.

Co-authorship network of co-authors of Emilio Boada-Romero

This figure shows the co-authorship network connecting the top 25 collaborators of Emilio Boada-Romero. A scholar is included among the top collaborators of Emilio Boada-Romero based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Emilio Boada-Romero. Emilio Boada-Romero is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Sharma, Piyush, Ao Guo, Suresh Poudel, et al.. (2025). Early methionine availability attenuates T cell exhaustion. Nature Immunology. 26(8). 1384–1396. 4 indexed citations

Boada-Romero, Emilio, Clifford S. Guy, Luigi Mari, et al.. (2025). Membrane receptors cluster phosphatidylserine to activate LC3-associated phagocytosis. Nature Cell Biology. 27(10). 1676–1687.

Heckmann, Bradlee L., Brett J.W. Teubner, Emilio Boada-Romero, et al.. (2020). Noncanonical function of an autophagy protein prevents spontaneous Alzheimer’s disease. Science Advances. 6(33). eabb9036–eabb9036. 79 indexed citations

Boada-Romero, Emilio, Simon R. Carding, Ulríke Mayer, et al.. (2020). Regulation of cytokine signaling through direct interaction between cytokine receptors and the ATG16L1 WD40 domain. Nature Communications. 11(1). 5919–5919. 12 indexed citations

Boada-Romero, Emilio, Jennifer Martinez, Bradlee L. Heckmann, & Douglas R. Green. (2020). The clearance of dead cells by efferocytosis. Nature Reviews Molecular Cell Biology. 21(7). 398–414. 548 indexed citations breakdown →

Heckmann, Bradlee L., Brett J.W. Teubner, Bart Tummers, et al.. (2020). LC3-Associated Endocytosis Facilitates β-Amyloid Clearance and Mitigates Neurodegeneration in Murine Alzheimer’s Disease. Cell. 183(6). 1733–1734. 24 indexed citations

Lechauve, Christophe, Eugene Khandros, Kalin Mayberry, et al.. (2019). The autophagy-activating kinase ULK1 mediates clearance of free α-globin in β-thalassemia. Science Translational Medicine. 11(506). 45 indexed citations

Slowicka, Karolina, Emilio Boada-Romero, Arne Martens, et al.. (2019). Physical and functional interaction between A20 and ATG16L1-WD40 domain in the control of intestinal homeostasis. Nature Communications. 10(1). 1834–1834. 31 indexed citations

Cunha, Larissa D., Mao Yang, Robert Carter, et al.. (2018). LC3-Associated Phagocytosis in Myeloid Cells Promotes Tumor Immune Tolerance. Cell. 175(2). 429–441.e16. 242 indexed citations

10.

Heckmann, Bradlee L., Emilio Boada-Romero, Larissa D. Cunha, Joëlle Magné, & Douglas R. Green. (2017). LC3-Associated Phagocytosis and Inflammation. Journal of Molecular Biology. 429(23). 3561–3576. 205 indexed citations

11.

Boada-Romero, Emilio, et al.. (2016). The T300A Crohn’s disease risk polymorphism impairs function of the WD40 domain of ATG16L1. Nature Communications. 7(1). 11821–11821. 55 indexed citations

12.

Boada-Romero, Emilio, et al.. (2016). Unconventional autophagy mediated by the WD40 domain of ATG16L1 is derailed by the T300A Crohn disease risk polymorphism. Autophagy. 12(11). 2254–2255. 4 indexed citations

13.

Pimentel‐Muiños, Felipe X. & Emilio Boada-Romero. (2014). Selective autophagy against membranous compartments. Autophagy. 10(3). 397–407. 20 indexed citations

14.

Boada-Romero, Emilio, et al.. (2013). TMEM59 defines a novel ATG16L1‐binding motif that promotes local activation of LC3. The EMBO Journal. 32(4). 566–582. 91 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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