Elizabeth A. Pollina
- Molecular Biology top 5%
- Physiology top 5%
- Cancer Research top 5%
- Aging top 0.5%
- Genetics top 10%
- Co-authors
- Anne BrunetBérénice A. BenayounSalah MahmoudiThomas A. RandoMichael E. GreenbergCindy LinDragana RoguljaAlexandra Vaccaro
- Topics
- Epigenetics and DNA Methylation (4 papers)Genetics, Aging, and Longevity in Model Organisms (3 papers)Neurogenesis and neuroplasticity mechanisms (3 papers)
- Partner nations
- United StatesSwitzerlandSingapore
In The Last Decade
Elizabeth A. Pollina
15 papers receiving 2.7k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Molecular Biology 1.7k
- Physiology 457
- Cancer Research 334
- Aging 289
- Genetics 259
Countries citing papers authored by Elizabeth A. Pollina
This map shows the geographic impact of Elizabeth A. Pollina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Elizabeth A. Pollina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Elizabeth A. Pollina more than expected).
Fields of papers citing papers by Elizabeth A. Pollina
This network shows the impact of papers produced by Elizabeth A. Pollina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Elizabeth A. Pollina. The network helps show where Elizabeth A. Pollina may publish in the future.
Co-authorship network of co-authors of Elizabeth A. Pollina
This figure shows the co-authorship network connecting the top 25 collaborators of Elizabeth A. Pollina. A scholar is included among the top collaborators of Elizabeth A. Pollina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Elizabeth A. Pollina. Elizabeth A. Pollina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 61 | |
| 3 | 12 | |
| 4 | Sleep Loss Can Cause Death through Accumulation of Reactive Oxygen Species in the Gutbreakdown → | 340 |
| 5 | 218 | |
| 6 | 34 | |
| 7 | Lysosome activation clears aggregates and enhances quiescent neural stem cell activation during agingbreakdown → | 382 |
| 8 | 404 | |
| 9 | 384 | |
| 10 | 119 | |
| 11 | 128 | |
| 12 | 92 | |
| 13 | 317 | |
| 14 | 57 | |
| 15 | 135 |
About Elizabeth A. Pollina
Elizabeth A. Pollina is a scholar working on Aging, Developmental Neuroscience and Biological Psychiatry, having authored 15 papers that have together received 2.7k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (4 papers), Genetics, Aging, and Longevity in Model Organisms (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). The work is most often cited by research in Aging (289 citations), Developmental Neuroscience (216 citations) and Molecular Biology (1.7k citations). Elizabeth A. Pollina has collaborated with scholars based in United States, Switzerland and Singapore. Frequent co-authors include Anne Brunet, Bérénice A. Benayoun, Salah Mahmoudi, Thomas A. Rando, Michael E. Greenberg, Cindy Lin, Dragana Rogulja, Alexandra Vaccaro, Ben W. Dulken and Anshul Kundaje. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.