Delfina Larrea

895 citations
18 papers · 643 indexed · h-index 12
Topics
Mitochondrial Function and Pathology (9 papers)Endoplasmic Reticulum Stress and Disease (6 papers)Alzheimer's disease research and treatments (5 papers)
Partner nations
United StatesSpainItaly

In The Last Decade

Delfina Larrea

17 papers receiving 625 citations

Peers

Delfina Larrea
Comparison fields: 5 of 70
  • Molecular Biology 399
  • Physiology 277
  • Cell Biology 109
  • Cellular and Molecular Neuroscience 84
  • Genetics 77
Replace Javier Calvo‐Garrido with:
Javier Calvo‐Garrido Sweden
Ara B. Hwang South Korea
Chinatsu Maita Japan
Magdalena Czeredys Poland
M. Pierre France
Xueying Wu China
Zhengyu Zhu China
Lihong Zhao United States
Myriam Mirza Germany
Nick Dang United States
Delfina Larrea relative to Javier Calvo‐Garrido Sweden Javier Calvo‐Garrido's profile →
Citations per field
00.5×2.6×
Javier Calvo‐Garrido · 1×
Citations per year

Countries citing papers authored by Delfina Larrea

Since Specialization
Citations

This map shows the geographic impact of Delfina Larrea's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Delfina Larrea with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Delfina Larrea more than expected).

Fields of papers citing papers by Delfina Larrea

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Delfina Larrea. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Delfina Larrea. The network helps show where Delfina Larrea may publish in the future.

Co-authorship network of co-authors of Delfina Larrea

This figure shows the co-authorship network connecting the top 25 collaborators of Delfina Larrea. A scholar is included among the top collaborators of Delfina Larrea based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Delfina Larrea. Delfina Larrea is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
#WorkIndexed citations
1 14
2 0
3 8
4 8
5 10
6 38
7 29
8 34
9 27
10 29
11 91
12 78
13 178
14 5
15 13
16 31
17 9
18 41

About Delfina Larrea

Delfina Larrea is a scholar working on Cell Biology, Physiology and Molecular Biology, having authored 18 papers that have together received 643 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (9 papers), Endoplasmic Reticulum Stress and Disease (6 papers) and Alzheimer's disease research and treatments (5 papers). The work is most often cited by research in Physiology (277 citations), Endocrinology (50 citations) and Biological Psychiatry (21 citations). Delfina Larrea has collaborated with scholars based in United States, Spain and Italy. Frequent co-authors include Estela Área-Gómez, Marta Pera, Rishi R. Agrawal, Jorge Montesinos, Kevin Velasco, Yimeng Xu, Cristina Guardia‐Laguarta, Eric A. Schon, Matxalen Llosa and Robin Chan. Their work appears in journals such as Nature Communications, The EMBO Journal and Scientific Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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