Andrew A. Sproul

3.0k total citations · 1 hit paper
35 papers, 2.0k citations indexed

About

Andrew A. Sproul is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Physiology. According to data from OpenAlex, Andrew A. Sproul has authored 35 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Molecular Biology, 10 papers in Cellular and Molecular Neuroscience and 10 papers in Physiology. Recurrent topics in Andrew A. Sproul's work include Pluripotent Stem Cells Research (15 papers), Alzheimer's disease research and treatments (8 papers) and CRISPR and Genetic Engineering (7 papers). Andrew A. Sproul is often cited by papers focused on Pluripotent Stem Cells Research (15 papers), Alzheimer's disease research and treatments (8 papers) and CRISPR and Genetic Engineering (7 papers). Andrew A. Sproul collaborates with scholars based in United States, Spain and Germany. Andrew A. Sproul's co-authors include Scott Noggle, Samson Jacob, Dominik Paquet, Dylan Kwart, Marc Tessier‐Lavigne, Andrew Gregg, Shaun Teo, Ottavio Arancio, Lloyd A. Greene and Matthew Zimmer and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Andrew A. Sproul

32 papers receiving 2.0k citations

Hit Papers

Efficient introduction of specific homozygous and heteroz... 2016 2026 2019 2022 2016 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andrew A. Sproul United States 19 1.5k 444 325 249 199 35 2.0k
Kristopher L. Nazor United States 17 1.9k 1.3× 489 1.1× 335 1.0× 223 0.9× 266 1.3× 23 2.5k
Kristine Freude Denmark 28 1.7k 1.1× 539 1.2× 391 1.2× 552 2.2× 110 0.6× 92 2.8k
Dominik Paquet Germany 15 1.4k 0.9× 482 1.1× 390 1.2× 231 0.9× 48 0.2× 26 2.0k
Cheryl Herrera United States 9 1.1k 0.7× 604 1.4× 418 1.3× 119 0.5× 215 1.1× 10 1.6k
Poul Hyttel Denmark 27 1.6k 1.1× 269 0.6× 245 0.8× 509 2.0× 108 0.5× 104 2.3k
Katja Hebestreit United States 15 1.6k 1.1× 260 0.6× 124 0.4× 201 0.8× 58 0.3× 19 2.4k
Fei Yi United States 27 2.3k 1.5× 208 0.5× 308 0.9× 332 1.3× 150 0.8× 62 3.0k
Francesco Paolo Di Giorgio Italy 14 1.5k 1.0× 299 0.7× 486 1.5× 144 0.6× 214 1.1× 26 2.3k
Andrew J. Petersen United States 14 770 0.5× 257 0.6× 345 1.1× 82 0.3× 69 0.3× 22 1.3k
Cécile Martinat France 21 1.7k 1.2× 288 0.6× 852 2.6× 204 0.8× 107 0.5× 55 2.7k

Countries citing papers authored by Andrew A. Sproul

Since Specialization
Citations

This map shows the geographic impact of Andrew A. Sproul's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andrew A. Sproul with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andrew A. Sproul more than expected).

Fields of papers citing papers by Andrew A. Sproul

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andrew A. Sproul. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andrew A. Sproul. The network helps show where Andrew A. Sproul may publish in the future.

Co-authorship network of co-authors of Andrew A. Sproul

This figure shows the co-authorship network connecting the top 25 collaborators of Andrew A. Sproul. A scholar is included among the top collaborators of Andrew A. Sproul based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andrew A. Sproul. Andrew A. Sproul is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Haage, Verena, Charles C. White, Ronak Patel, et al.. (2025). HDAC inhibitors engage MITF and the disease-associated microglia signature to enhance amyloid β uptake. Brain Behavior and Immunity. 129. 279–293. 1 indexed citations
4.
Qu, Xiaoyi, et al.. (2023). Impaired Microtubule Dynamics Are a Driver of Tau Phosphorylation. Alzheimer s & Dementia. 19(S13). 1 indexed citations
5.
Blaze, Jennifer, Mehmet İlyas Coşacak, Atul Kumar, et al.. (2022). RNA methyltransferase NSun2 deficiency promotes neurodegeneration through epitranscriptomic regulation of tau phosphorylation. Acta Neuropathologica. 145(1). 29–48. 21 indexed citations
6.
Lee, Annie, Chandana Kondapalli, Tommy L. Lewis, et al.. (2022). Aβ42 oligomers trigger synaptic loss through CAMKK2-AMPK-dependent effectors coordinating mitochondrial fission and mitophagy. Nature Communications. 13(1). 4444–4444. 69 indexed citations
7.
Hernández, José Martínez, Aditi Sharma, Jean-Marc Soleilhac, et al.. (2022). Crosstalk between acetylation and the tyrosination/detyrosination cycle of α-tubulin in Alzheimer’s disease. Frontiers in Cell and Developmental Biology. 10. 926914–926914. 17 indexed citations
8.
Coric, Pascale, Ju Youn Kim, Ronak Patel, et al.. (2022). Genuine selective caspase-2 inhibition with new irreversible small peptidomimetics. Cell Death and Disease. 13(11). 959–959. 7 indexed citations
9.
Lu, Tyler M., Sean Houghton, Tarig Magdeldin, et al.. (2021). Pluripotent stem cell-derived epithelium misidentified as brain microvascular endothelium requires ETS factors to acquire vascular fate. Proceedings of the National Academy of Sciences. 118(8). 146 indexed citations
10.
Sun, Jichao, Jared Carlson-Stevermer, Utpal Das, et al.. (2018). CRISPR/Cas9 editing of APP C-terminus attenuates β-cleavage and promotes α-cleavage. Nature Communications. 10(1). 53–53. 79 indexed citations
11.
Chung, Kyung Min, et al.. (2018). Alzheimer’s disease and the autophagic-lysosomal system. Neuroscience Letters. 697. 49–58. 50 indexed citations
12.
Ortiz‐Virumbrales, Maitane, Cesar L. Moreno, Ilya Kruglikov, et al.. (2017). CRISPR/Cas9-Correctable mutation-related molecular and physiological phenotypes in iPSC-derived Alzheimer’s PSEN2 N141I neurons. Acta Neuropathologica Communications. 5(1). 77–77. 122 indexed citations
13.
Sproul, Andrew A.. (2015). Being human: The role of pluripotent stem cells in regenerative medicine and humanizing Alzheimer's disease models. Molecular Aspects of Medicine. 43-44. 54–65. 23 indexed citations
14.
Sproul, Andrew A., Samson Jacob, Deborah Prè, et al.. (2014). Characterization and Molecular Profiling of PSEN1 Familial Alzheimer's Disease iPSC-Derived Neural Progenitors. PLoS ONE. 9(1). e84547–e84547. 140 indexed citations
15.
Prè, Deborah, Michael W. Nestor, Andrew A. Sproul, et al.. (2014). A Time Course Analysis of the Electrophysiological Properties of Neurons Differentiated from Human Induced Pluripotent Stem Cells (iPSCs). PLoS ONE. 9(7). e103418–e103418. 95 indexed citations
16.
Nestor, Michael W., et al.. (2013). Differentiation of serum-free embryoid bodies from human induced pluripotent stem cells into networks. Stem Cell Research. 10(3). 454–463. 17 indexed citations
17.
Kahler, David J., Haiqing Hua, Dorota Moroziewicz, et al.. (2013). Improved Methods for Reprogramming Human Dermal Fibroblasts Using Fluorescence Activated Cell Sorting. PLoS ONE. 8(3). e59867–e59867. 49 indexed citations
18.
Sproul, Andrew A., et al.. (2009). Cbl negatively regulates JNK activation and cell death. Cell Research. 19(8). 950–961. 11 indexed citations
19.
Biswas, Subhas C., Yijie Shi, Andrew A. Sproul, & Lloyd A. Greene. (2007). Pro-apoptotic Bim Induction in Response to Nerve Growth Factor Deprivation Requires Simultaneous Activation of Three Different Death Signaling Pathways. Journal of Biological Chemistry. 282(40). 29368–29374. 84 indexed citations
20.
Xu, Zhiheng, et al.. (2005). Siah1 Interacts with the Scaffold Protein POSH to Promote JNK Activation and Apoptosis. Journal of Biological Chemistry. 281(1). 303–312. 55 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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