David S. Weiss

20.9k total citations · 3 hit papers
83 papers, 15.7k citations indexed

About

David S. Weiss is a scholar working on Molecular Biology, Molecular Medicine and Endocrinology. According to data from OpenAlex, David S. Weiss has authored 83 papers receiving a total of 15.7k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 31 papers in Molecular Medicine and 24 papers in Endocrinology. Recurrent topics in David S. Weiss's work include Antibiotic Resistance in Bacteria (31 papers), Bacterial Genetics and Biotechnology (18 papers) and Vibrio bacteria research studies (17 papers). David S. Weiss is often cited by papers focused on Antibiotic Resistance in Bacteria (31 papers), Bacterial Genetics and Biotechnology (18 papers) and Vibrio bacteria research studies (17 papers). David S. Weiss collaborates with scholars based in United States, Germany and Sweden. David S. Weiss's co-authors include Yvette Weinrauch, Arturo Zychlinsky, Christian Goosmann, Beatrix Fauler, Volker Brinkmann, Denise M. Monack, Sanjeev Mariathasan, Vishva M. Dixit, Timothy R. Sampson and Jacqueline McBride and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

David S. Weiss

82 papers receiving 15.5k citations

Hit Papers

Neutrophil Extracellular Traps Kill Bacteria 2004 2026 2011 2018 2004 2006 2019 2.5k 5.0k 7.5k

Peers

David S. Weiss
Robert K. Ernst United States
Brad T. Cookson United States
Denise M. Monack United States
Kelly D. Smith United States
Petr Brož Switzerland
Simon J. Foster United Kingdom
David S. Weiss
Citations per year, relative to David S. Weiss David S. Weiss (= 1×) peers Ivo G. Boneca

Countries citing papers authored by David S. Weiss

Since Specialization
Citations

This map shows the geographic impact of David S. Weiss's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David S. Weiss with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David S. Weiss more than expected).

Fields of papers citing papers by David S. Weiss

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David S. Weiss. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David S. Weiss. The network helps show where David S. Weiss may publish in the future.

Co-authorship network of co-authors of David S. Weiss

This figure shows the co-authorship network connecting the top 25 collaborators of David S. Weiss. A scholar is included among the top collaborators of David S. Weiss based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David S. Weiss. David S. Weiss is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Choby, Jacob E., Jennifer M. Colquhoun, Sarah W. Satola, et al.. (2025). Copy number flexibility facilitates heteroresistance to increasing antibiotic pressure and threatens the beta-lactam pipeline. Nature Communications. 16(1). 5721–5721. 1 indexed citations
2.
Levin, Bruce R., Teresa Gil‐Gil, Jacob E. Choby, et al.. (2024). Theoretical considerations and empirical predictions of the pharmaco- and population dynamics of heteroresistance. Proceedings of the National Academy of Sciences. 121(16). e2318600121–e2318600121. 5 indexed citations
3.
Babiker, Ahmed, et al.. (2024). Heteroresistance to piperacillin/tazobactam in Klebsiella pneumoniae is mediated by increased copy number of multiple β-lactamase genes. JAC-Antimicrobial Resistance. 6(2). dlae057–dlae057. 5 indexed citations
4.
Weiss, David S. & Martin Maurer. (2024). Inhomogeneous Focal Fat Distribution as Pseudolesions in a Patient with Pancreatic Carcinoma. SHILAP Revista de lepidopterología. 9(1). 11–13. 1 indexed citations
5.
Zhai, Bing, Chen Liao, Yuanyuan Tang, et al.. (2024). Antifungal heteroresistance causes prophylaxis failure and facilitates breakthrough Candida parapsilosis infections. Nature Medicine. 30(11). 3163–3172. 24 indexed citations
6.
Hufnagel, David A., Jacob E. Choby, Samantha Hao, et al.. (2021). Antibiotic-Selected Gene Amplification Heightens Metal Resistance. mBio. 12(1). 5 indexed citations
7.
Chande, Aroon T., Eileen M. Burd, Victor I. Band, et al.. (2020). Absence of mgrB Alleviates Negative Growth Effects of Colistin Resistance in Enterobacter cloacae. Antibiotics. 9(11). 825–825. 5 indexed citations
8.
Price, Aryn A., Arash Grakoui, & David S. Weiss. (2016). Harnessing the Prokaryotic Adaptive Immune System as a Eukaryotic Antiviral Defense. Trends in Microbiology. 24(4). 294–306. 24 indexed citations
9.
Band, Victor I., Emily K. Crispell, Brooke A. Napier, et al.. (2016). Antibiotic failure mediated by a resistant subpopulation in Enterobacter cloacae. Nature Microbiology. 1(6). 16053–16053. 165 indexed citations
10.
Heeke, Arielle L., Henry M. Blumberg, David S. Weiss, et al.. (2015). Disseminated emm Type 12 Group A Streptococcus and Review of Invasive Disease. The American Journal of the Medical Sciences. 350(5). 429–431. 2 indexed citations
11.
Sampson, Timothy R. & David S. Weiss. (2014). CRISPR-Cas systems: new players in gene regulation and bacterial physiology. Frontiers in Cellular and Infection Microbiology. 4. 37–37. 75 indexed citations
12.
Ravindran, Rajesh, Nooruddin Khan, Helder I. Nakaya, et al.. (2013). Vaccine Activation of the Nutrient Sensor GCN2 in Dendritic Cells Enhances Antigen Presentation. Science. 343(6168). 313–317. 161 indexed citations
13.
Napier, Brooke A., Eileen M. Burd, Sarah W. Satola, et al.. (2013). Clinical Use of Colistin Induces Cross-Resistance to Host Antimicrobials in Acinetobacter baumannii. mBio. 4(3). e00021–13. 87 indexed citations
14.
Sampson, Timothy R. & David S. Weiss. (2013). Exploiting CRISPR/Cas systems for biotechnology. BioEssays. 36(1). 34–38. 46 indexed citations
15.
Sampson, Timothy R. & David S. Weiss. (2013). Alternative Roles for CRISPR/Cas Systems in Bacterial Pathogenesis. PLoS Pathogens. 9(10). e1003621–e1003621. 33 indexed citations
16.
Pierini, Roberto, et al.. (2012). AIM2/ASC triggers caspase-8-dependent apoptosis in Francisella-infected caspase-1-deficient macrophages. Cell Death and Differentiation. 19(10). 1709–1721. 207 indexed citations
17.
Llewellyn, Anna C., Crystal L. Jones, Brooke A. Napier, James E. Bina, & David S. Weiss. (2011). Macrophage Replication Screen Identifies a Novel Francisella Hydroperoxide Resistance Protein Involved in Virulence. PLoS ONE. 6(9). e24201–e24201. 50 indexed citations
18.
Weiss, David S., Thomas Henry, & Denise M. Monack. (2007). Francisella Tularensis: Activation of the Inflammasome. Annals of the New York Academy of Sciences. 1105(1). 219–237. 42 indexed citations
19.
Mariathasan, Sanjeev, David S. Weiss, Vishva M. Dixit, & Denise M. Monack. (2005). Innate immunity against Francisella tularensis is dependent on the ASC/caspase-1 axis. The Journal of Experimental Medicine. 202(8). 1043–1049. 345 indexed citations
20.
Weiss, David S., Bärbel Raupach, Kohsuke Takeda, Shizuo Akira, & Arturo Zychlinsky. (2004). Toll-Like Receptors Are Temporally Involved in Host Defense. The Journal of Immunology. 172(7). 4463–4469. 176 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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