David Partida

691 total citations
8 papers, 540 citations indexed

About

David Partida is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, David Partida has authored 8 papers receiving a total of 540 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Cell Biology and 2 papers in Oncology. Recurrent topics in David Partida's work include Microtubule and mitosis dynamics (3 papers), Cancer-related Molecular Pathways (2 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (1 paper). David Partida is often cited by papers focused on Microtubule and mitosis dynamics (3 papers), Cancer-related Molecular Pathways (2 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (1 paper). David Partida collaborates with scholars based in Spain, United States and United Kingdom. David Partida's co-authors include Marcos Malumbres, Gonzalo Fernández‐Miranda, María Salazar‐Roa, Raúl Méndez, Manuel Pérez‐Martínez, Eduardo Rial, Miguel López, Guillermo Velasco, Esther Seco and Lorena Esteban‐Martínez and has published in prestigious journals such as Journal of Clinical Investigation, Blood and PLoS ONE.

In The Last Decade

David Partida

8 papers receiving 538 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
David Partida Spain	7	374	143	124	104	95	8	540
Sung‐Keum Seo South Korea	14	432 1.2×	90 0.6×	124 1.0×	116 1.1×	75 0.8×	17	602
Sang‐Hyeok Woo South Korea	17	596 1.6×	110 0.8×	138 1.1×	159 1.5×	80 0.8×	21	847
Chang‐Hun Rhee South Korea	13	384 1.0×	71 0.5×	107 0.9×	114 1.1×	70 0.7×	17	601
Francisco Iñesta-Vaquera United Kingdom	12	655 1.8×	124 0.9×	125 1.0×	98 0.9×	98 1.0×	19	997
Gang Hu China	16	769 2.1×	118 0.8×	169 1.4×	142 1.4×	82 0.9×	24	1.0k
Luena Papa United States	11	493 1.3×	112 0.8×	66 0.5×	95 0.9×	79 0.8×	16	744
Jinge Kong China	10	192 0.5×	69 0.5×	78 0.6×	142 1.4×	84 0.9×	13	518
Okkyung Rho United States	17	537 1.4×	89 0.6×	179 1.4×	132 1.3×	48 0.5×	29	746
Mahandranauth A. Chetram United States	10	421 1.1×	248 1.7×	213 1.7×	103 1.0×	31 0.3×	13	753

Countries citing papers authored by David Partida

Since Specialization

Citations

This map shows the geographic impact of David Partida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Partida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Partida more than expected).

Fields of papers citing papers by David Partida

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Partida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Partida. The network helps show where David Partida may publish in the future.

Co-authorship network of co-authors of David Partida

This figure shows the co-authorship network connecting the top 25 collaborators of David Partida. A scholar is included among the top collaborators of David Partida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Partida. David Partida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

Hurtado, Begoña, Marianna Trakala, Pilar Ximénez‐Embún, et al.. (2018). Thrombocytopenia-associated mutations in Ser/Thr kinase MASTL deregulate actin cytoskeletal dynamics in platelets. Journal of Clinical Investigation. 128(12). 5351–5367. 20 indexed citations

Álvarez‐Fernández, Mónica, María Salazar‐Roa, David Partida, et al.. (2017). Therapeutic relevance of the PP2A-B55 inhibitory kinase MASTL/Greatwall in breast cancer. Cell Death and Differentiation. 25(5). 828–840. 73 indexed citations

Trakala, Marianna, et al.. (2015). Activation of the endomitotic spindle assembly checkpoint and thrombocytopenia in Plk1-deficient mice. Blood. 126(14). 1707–1714. 16 indexed citations

Domènech, Elena B., Carolina Maestre, Lorena Esteban‐Martínez, et al.. (2015). AMPK and PFKFB3 mediate glycolysis and survival in response to mitophagy during mitotic arrest. Nature Cell Biology. 17(10). 1304–1316. 206 indexed citations

González‐Loyola, Alejandra, Gonzalo Fernández‐Miranda, Marianna Trakala, et al.. (2015). Aurora B Overexpression Causes Aneuploidy and p21^Cip1 Repression during Tumor Development. Molecular and Cellular Biology. 35(20). 3566–3578. 88 indexed citations

Rodríguez, Esther, Víctor Quereda, Florian Bellutti, et al.. (2014). Cdk4 and Cdk6 cooperate in counteracting the INK4 family of inhibitors during murine leukemogenesis. Blood. 124(15). 2380–2390. 24 indexed citations

Galmozzi, Andrea, et al.. (2013). Identification of Regulatory Elements That Control PPARγ Expression in Adipocyte Progenitors. PLoS ONE. 8(8). e72511–e72511. 6 indexed citations

Domínguez, Eduardo, Andrea Galmozzi, Ku‐Lung Hsu, et al.. (2013). Integrated phenotypic and activity-based profiling links Ces3 to obesity and diabetes. Nature Chemical Biology. 10(2). 113–121. 107 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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