Chang‐Hun Rhee

690 total citations
17 papers, 601 citations indexed

About

Chang‐Hun Rhee is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Chang‐Hun Rhee has authored 17 papers receiving a total of 601 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Chang‐Hun Rhee's work include Cell death mechanisms and regulation (5 papers), Peptidase Inhibition and Analysis (3 papers) and Retinoids in leukemia and cellular processes (3 papers). Chang‐Hun Rhee is often cited by papers focused on Cell death mechanisms and regulation (5 papers), Peptidase Inhibition and Analysis (3 papers) and Retinoids in leukemia and cellular processes (3 papers). Chang‐Hun Rhee collaborates with scholars based in South Korea, United States and Ethiopia. Chang‐Hun Rhee's co-authors include Seok‐Il Hong, In‐Chul Park, Sang‐Hyeok Woo, Hyung‐Chahn Lee, Taeboo Choe, Doo‐Hyun Yoo, Hyeon‐Ok Jin, Sungkwan An, Seung‐Hoon Lee and Su‐Jae Lee and has published in prestigious journals such as Free Radical Biology and Medicine, Journal of neurosurgery and Biochemical Pharmacology.

In The Last Decade

Chang‐Hun Rhee

17 papers receiving 590 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Chang‐Hun Rhee South Korea 13 384 114 107 71 70 17 601
Doo‐Hyun Yoo South Korea 13 435 1.1× 124 1.1× 98 0.9× 93 1.3× 76 1.1× 13 706
Sung‐Keum Seo South Korea 14 432 1.1× 116 1.0× 124 1.2× 90 1.3× 75 1.1× 17 602
Sang‐Hyeok Woo South Korea 17 596 1.6× 159 1.4× 138 1.3× 110 1.5× 80 1.1× 21 847
Okkyung Rho United States 17 537 1.4× 132 1.2× 179 1.7× 89 1.3× 48 0.7× 29 746
José Luís Oliva Spain 15 425 1.1× 95 0.8× 101 0.9× 53 0.7× 31 0.4× 23 596
Hengyi Xiao China 12 516 1.3× 139 1.2× 118 1.1× 76 1.1× 65 0.9× 24 717
Ali Zekri Iran 17 353 0.9× 114 1.0× 138 1.3× 119 1.7× 49 0.7× 38 583
Fangliang Zhou China 11 391 1.0× 182 1.6× 67 0.6× 52 0.7× 60 0.9× 19 627
Steve L. Abrams United States 5 444 1.2× 82 0.7× 135 1.3× 42 0.6× 38 0.5× 8 625
In‐Chul Park South Korea 9 237 0.6× 106 0.9× 125 1.2× 55 0.8× 29 0.4× 10 459

Countries citing papers authored by Chang‐Hun Rhee

Since Specialization
Citations

This map shows the geographic impact of Chang‐Hun Rhee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chang‐Hun Rhee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chang‐Hun Rhee more than expected).

Fields of papers citing papers by Chang‐Hun Rhee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chang‐Hun Rhee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chang‐Hun Rhee. The network helps show where Chang‐Hun Rhee may publish in the future.

Co-authorship network of co-authors of Chang‐Hun Rhee

This figure shows the co-authorship network connecting the top 25 collaborators of Chang‐Hun Rhee. A scholar is included among the top collaborators of Chang‐Hun Rhee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chang‐Hun Rhee. Chang‐Hun Rhee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Jin, Hyeon‐Ok, Sung‐Keum Seo, Sang‐Hyeok Woo, et al.. (2009). Activating transcription factor 4 and CCAAT/enhancer-binding protein-β negatively regulate the mammalian target of rapamycin via Redd1 expression in response to oxidative and endoplasmic reticulum stress. Free Radical Biology and Medicine. 46(8). 1158–1167. 97 indexed citations
2.
Lee, Hyung‐Chahn, Sungkwan An, Hansoo Lee, et al.. (2008). Activation of Epidermal Growth Factor Receptor and Its Downstream Signaling Pathway by Nitric Oxide in Response to Ionizing Radiation. Molecular Cancer Research. 6(6). 996–1002. 42 indexed citations
3.
Jin, Hyeon‐Ok, Sung‐Keum Seo, Sang‐Hyeok Woo, et al.. (2008). A combination of sulindac and arsenic trioxide synergistically induces apoptosis in human lung cancer H1299 cells via c-Jun NH2-terminal kinase-dependent Bcl-xL phosphorylation. Lung Cancer. 61(3). 317–327. 29 indexed citations
4.
Seo, Sung‐Keum, Hyeon‐Ok Jin, Sang‐Hyeok Woo, et al.. (2007). Combined Effects of Sulindac and Suberoylanilide Hydroxamic Acid on Apoptosis Induction in Human Lung Cancer Cells. Molecular Pharmacology. 73(3). 1005–1012. 23 indexed citations
5.
Jin, Hyeon‐Ok, Sung‐Keum Seo, Hyung‐Chahn Lee, et al.. (2006). Synergistic induction of apoptosis by sulindac and arsenic trioxide in human lung cancer A549 cells via reactive oxygen species-dependent down-regulation of survivin. Biochemical Pharmacology. 72(10). 1228–1236. 47 indexed citations
6.
Seo, Sung‐Keum, Hyung‐Chahn Lee, Sang‐Hyeok Woo, et al.. (2006). Sulindac-derived reactive oxygen species induce apoptosis of human multiple myeloma cells via p38 mitogen activated protein kinase-induced mitochondrial dysfunction. APOPTOSIS. 12(1). 195–209. 34 indexed citations
7.
Jin, Hyeon‐Ok, In‐Chul Park, Sungkwan An, et al.. (2005). Up‐regulation of Bak and Bim via JNK downstream pathway in the response to nitric oxide in human glioblastoma cells. Journal of Cellular Physiology. 206(2). 477–486. 50 indexed citations
8.
Lee, Hyung‐Chahn, In‐Chul Park, Ki Hun Park, et al.. (2004). Sulindac and its metabolites inhibit invasion of glioblastoma cells via down‐regulation of Akt/PKB and MMP‐2. Journal of Cellular Biochemistry. 94(3). 597–610. 46 indexed citations
9.
Kim, Jong-Taek, Chang‐Hun Rhee, Myung‐Jin Park, et al.. (2004). Reverse Effects of Tetraarsenic Oxide on the Angiogenesis Induced by Nerve Growth Factor in the Rat Cornea. Journal of Veterinary Medical Science. 66(9). 1091–1095. 8 indexed citations
10.
Lee, Hyung‐Chahn, Dae Won Kim, In‐Chul Park, et al.. (2004). Increased expression of antioxidant enzymes in radioresistant variant from U251 human glioblastoma cell line. International Journal of Molecular Medicine. 73 indexed citations
11.
Park, In‐Chul, Sang‐Hyeok Woo, Myung‐Jin Park, et al.. (2003). Ionizing radiation and nitric oxide donor sensitize Fas-induced apoptosis via up-regulation of Fas in human cervical cancer cells.. PubMed. 10(3). 629–33. 11 indexed citations
12.
Park, Myung Jin, In‐Chul Park, Sang‐Hyeok Woo, et al.. (2002). Modulation of phorbol ester—induced regulation of matrix metalloproteinases and tissue inhibitors of metalloproteinases by SB203580, a specific inhibitor of p38 mitogen-activated protein kinase. Journal of neurosurgery. 97(1). 112–118. 36 indexed citations
13.
Lee, Seung-Hoon, Heeseog Kang, Chang‐Hun Rhee, et al.. (2001). Growth-inhibitory effect of adenovirus-mediated p53 gene transfer on medulloblastoma cell line, Daoy, harboring mutant p53. Child s Nervous System. 17(3). 134–138. 6 indexed citations
14.
Park, In‐Chul, Myung‐Jin Park, Chang‐Hun Rhee, et al.. (2001). Protein kinase C activation by PMA rapidly induces apoptosis through caspase-3/CPP32 and serine protease(s) in a gastric cancer cell line. International Journal of Oncology. 18(5). 1077–83. 22 indexed citations
15.
Park, In‐Chul, Myung Jin Park, Sang‐Hyeok Woo, et al.. (2001). Tumor Necrosis Factor-Related Apoptosis Inducing Ligand (TRAIL)-Induced Apoptosis is Dependent on Activation of Cysteine and Serine Proteases. Cytokine. 15(3). 166–170. 8 indexed citations
16.
17.
Lee, Seung‐Hoon, et al.. (1998). Loss of heterozygosity on chromosome 22q and 17p correlates with aggressiveness of meningiomas. Journal of Neuro-Oncology. 40(2). 101–106. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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