D. Luke Fischer
- Neurology top 5%
- Cellular and Molecular Neuroscience top 10%
- Neurology top 10%
- Molecular Biology
- Physiology
- Co-authors
- Caryl E. SortwellChristopher J. KempTimothy J. CollierMegan F. DuffyNicole K. PolinskiKatrina L. PaumierJoseph R. PattersonFredric P. Manfredsson
- Topics
- Parkinson's Disease Mechanisms and Treatments (12 papers)Neurological disorders and treatments (7 papers)Nerve injury and regeneration (7 papers)
- Partner nations
- United StatesQatarSouth Africa
In The Last Decade
D. Luke Fischer
18 papers receiving 440 citations
Peers
Comparison fields: 5 of 54
- Neurology 312
- Cellular and Molecular Neuroscience 231
- Neurology 111
- Molecular Biology 67
- Physiology 63
Countries citing papers authored by D. Luke Fischer
This map shows the geographic impact of D. Luke Fischer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Luke Fischer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Luke Fischer more than expected).
Fields of papers citing papers by D. Luke Fischer
This network shows the impact of papers produced by D. Luke Fischer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Luke Fischer. The network helps show where D. Luke Fischer may publish in the future.
Co-authorship network of co-authors of D. Luke Fischer
This figure shows the co-authorship network connecting the top 25 collaborators of D. Luke Fischer. A scholar is included among the top collaborators of D. Luke Fischer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with D. Luke Fischer. D. Luke Fischer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 0 | |
| 3 | 1 | |
| 4 | 22 | |
| 5 | A Precision Medicine Approach to Dementia Care: Syndrome, Etiology, and Copathology. | 2 |
| 6 | 1 | |
| 7 | 8 | |
| 8 | 15 | |
| 9 | 16 | |
| 10 | 35 | |
| 11 | 15 | |
| 12 | 140 | |
| 13 | 26 | |
| 14 | 18 | |
| 15 | 42 | |
| 16 | 27 | |
| 17 | 20 | |
| 18 | 17 | |
| 19 | 15 | |
| 20 | 25 |
About D. Luke Fischer
D. Luke Fischer is a scholar working on Neurology, Cellular and Molecular Neuroscience and Neurology, having authored 20 papers that have together received 445 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (12 papers), Neurological disorders and treatments (7 papers) and Nerve injury and regeneration (7 papers). The work is most often cited by research in Neurology (312 citations), Neurology (111 citations) and Cellular and Molecular Neuroscience (231 citations). D. Luke Fischer has collaborated with scholars based in United States, Qatar and South Africa. Frequent co-authors include Caryl E. Sortwell, Christopher J. Kemp, Timothy J. Collier, Megan F. Duffy, Nicole K. Polinski, Katrina L. Paumier, Joseph R. Patterson, Fredric P. Manfredsson, Allyson Cole-Strauss and Kathy Steece‐Collier. Their work appears in journals such as Journal of Neuroscience, PLoS ONE and Annals of Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.