Nathan Levine

962 total citations
15 papers, 700 citations indexed

About

Nathan Levine is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, Nathan Levine has authored 15 papers receiving a total of 700 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Cellular and Molecular Neuroscience, 9 papers in Neurology and 4 papers in Molecular Biology. Recurrent topics in Nathan Levine's work include Parkinson's Disease Mechanisms and Treatments (8 papers), Neurological disorders and treatments (4 papers) and Nerve injury and regeneration (4 papers). Nathan Levine is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (8 papers), Neurological disorders and treatments (4 papers) and Nerve injury and regeneration (4 papers). Nathan Levine collaborates with scholars based in United States, Switzerland and France. Nathan Levine's co-authors include Holly Kelly, Darren J. Moore, Timothy J. Collier, Jiyan Ma, Caryl E. Sortwell, Kathy Steece‐Collier, Xinhe Wang, Katelyn Becker, Michelle Zhang and Andrew P. Lieberman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, European Journal of Neuroscience and Experimental Neurology.

In The Last Decade

Nathan Levine

15 papers receiving 692 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nathan Levine United States 10 432 292 182 149 94 15 700
Armando Arroyo United States 9 428 1.0× 271 0.9× 266 1.5× 215 1.4× 107 1.1× 9 825
Carla Brancia Italy 15 120 0.3× 158 0.5× 183 1.0× 128 0.9× 34 0.4× 22 531
James Dell’Orco United States 9 107 0.2× 295 1.0× 287 1.6× 58 0.4× 32 0.3× 9 533
Brigitte Delespierre France 11 66 0.2× 209 0.7× 143 0.8× 74 0.5× 98 1.0× 14 643
Ané Korff United States 12 216 0.5× 65 0.2× 235 1.3× 223 1.5× 147 1.6× 13 645
Amanda Singleton United States 14 666 1.5× 490 1.7× 290 1.6× 202 1.4× 170 1.8× 17 985
John D. Schwankhaus United States 13 269 0.6× 192 0.7× 257 1.4× 107 0.7× 72 0.8× 25 674
Jens Ebentheuer Germany 13 832 1.9× 182 0.6× 128 0.7× 148 1.0× 127 1.4× 15 983
Kazuya Ando Japan 10 265 0.6× 290 1.0× 125 0.7× 51 0.3× 98 1.0× 28 507

Countries citing papers authored by Nathan Levine

Since Specialization
Citations

This map shows the geographic impact of Nathan Levine's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan Levine with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan Levine more than expected).

Fields of papers citing papers by Nathan Levine

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan Levine. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan Levine. The network helps show where Nathan Levine may publish in the future.

Co-authorship network of co-authors of Nathan Levine

This figure shows the co-authorship network connecting the top 25 collaborators of Nathan Levine. A scholar is included among the top collaborators of Nathan Levine based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathan Levine. Nathan Levine is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Levine, Nathan, et al.. (2024). Adult-onset deletion of ATP13A2 in mice induces progressive nigrostriatal pathway dopaminergic degeneration and lysosomal abnormalities. npj Parkinson s Disease. 10(1). 133–133. 5 indexed citations
2.
Chen, Xi, Elpida Tsika, Nathan Levine, & Darren J. Moore. (2023). VPS35 and α-Synuclein fail to interact to modulate neurodegeneration in rodent models of Parkinson’s disease. Molecular Neurodegeneration. 18(1). 51–51. 6 indexed citations
3.
Rauova, Lubica, Catherine J. Field, Nathan Levine, et al.. (2023). OC 76.1 Chadox1 Exposure-Induced Neutrophil-Activating Peptide 2 (NAP2) Antibodies: Murine-Based Insights into the Pathogenesis of Thrombosis with Thrombocytopenia Syndrome (TTS). Research and Practice in Thrombosis and Haemostasis. 7. 100348–100348. 1 indexed citations
4.
Tsika, Elpida, Kaela Kelly, Nathan Levine, et al.. (2020). Dopaminergic neurodegeneration induced by Parkinson's disease-linked G2019S LRRK2 is dependent on kinase and GTPase activity. Proceedings of the National Academy of Sciences. 117(29). 17296–17307. 43 indexed citations
5.
Chen, Xi, Erin T. Williams, Nathan Levine, et al.. (2019). Parkinson’s disease-linked D620N VPS35 knockin mice manifest tau neuropathology and dopaminergic neurodegeneration. Proceedings of the National Academy of Sciences. 116(12). 5765–5774. 75 indexed citations
6.
Wang, Xinhe, Katelyn Becker, Nathan Levine, et al.. (2019). Pathogenic alpha-synuclein aggregates preferentially bind to mitochondria and affect cellular respiration. Acta Neuropathologica Communications. 7(1). 41–41. 144 indexed citations
7.
Patterson, Joseph R., Megan F. Duffy, Christopher J. Kemp, et al.. (2019). Time course and magnitude of alpha-synuclein inclusion formation and nigrostriatal degeneration in the rat model of synucleinopathy triggered by intrastriatal α-synuclein preformed fibrils. Neurobiology of Disease. 130. 104525–104525. 73 indexed citations
8.
Levine, Nathan, David J. Rademacher, Timothy J. Collier, et al.. (2013). Advances in thin tissue Golgi-Cox impregnation: Fast, reliable methods for multi-assay analyses in rodent and non-human primate brain. Journal of Neuroscience Methods. 213(2). 214–227. 27 indexed citations
9.
Spieles-Engemann, Anne L., Kathy Steece‐Collier, Michael M. Behbehani, et al.. (2011). Subthalamic Nucleus Stimulation Increases Brain Derived Neurotrophic Factor in the Nigrostriatal System and Primary Motor Cortex. Journal of Parkinson s Disease. 1(1). 123–136. 76 indexed citations
10.
Spieles-Engemann, Anne L., Kathy Steece‐Collier, Michael M. Behbehani, et al.. (2011). Subthalamic nucleus stimulation increases brain derived neurotrophic factor in the nigrostriatal system and primary motor cortex.. PubMed. 1(1). 123–36. 71 indexed citations
11.
Soderstrom, Katherine E., et al.. (2010). Impact of dendritic spine preservation in medium spiny neurons on dopamine graft efficacy and the expression of dyskinesias in parkinsonian rats. European Journal of Neuroscience. 31(3). 478–490. 50 indexed citations
12.
Terpstra, Brian T., Timothy J. Collier, Deanna M. Marchionini, et al.. (2007). Increased cell suspension concentration augments the survival rate of grafted tyrosine hydroxylase immunoreactive neurons. Journal of Neuroscience Methods. 166(1). 13–19. 17 indexed citations
13.
Lipton, Jack W., Lin Pei, Kim B. Seroogy, et al.. (2006). Genetics and dyskinesias: Role of DARPP32 modulation in parkinsonian rats. Experimental Neurology. 198(2). 583–584. 1 indexed citations
14.
Haas, Felix, et al.. (1988). Changes in Serum K + in Healthy and in Asthmatic Subjects during Exercise. American Review of Respiratory Disease. 137(4). 833–836. 7 indexed citations
15.
Levine, Nathan & Holly Kelly. (1978). Measurement of pH in the rat epididymis in vivo. Reproduction. 52(2). 333–335. 104 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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