Avital Swisa

2.1k total citations
19 papers, 1.3k citations indexed

About

Avital Swisa is a scholar working on Surgery, Molecular Biology and Genetics. According to data from OpenAlex, Avital Swisa has authored 19 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Surgery, 13 papers in Molecular Biology and 8 papers in Genetics. Recurrent topics in Avital Swisa's work include Pancreatic function and diabetes (15 papers), Diabetes and associated disorders (7 papers) and Metabolism, Diabetes, and Cancer (6 papers). Avital Swisa is often cited by papers focused on Pancreatic function and diabetes (15 papers), Diabetes and associated disorders (7 papers) and Metabolism, Diabetes, and Cancer (6 papers). Avital Swisa collaborates with scholars based in Israel, United States and Belgium. Avital Swisa's co-authors include Yuval Dor, Benjamin Gläser, Zvi Granot, Nabeel Bardeesy, Miri Stolovich-Rain, Oded Meyuhas, Sushma Gurumurthy, Gerald Bailey, Ronald A. DePinho and Aram F. Hezel and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Molecular and Cellular Biology.

In The Last Decade

Avital Swisa

19 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Avital Swisa Israel 16 755 586 317 255 187 19 1.3k
Brigitte Koeberlein United States 17 588 0.8× 851 1.5× 508 1.6× 404 1.6× 108 0.6× 26 1.5k
Maki Moritani Japan 22 804 1.1× 435 0.7× 440 1.4× 310 1.2× 71 0.4× 58 1.5k
Gilda Cobellis Italy 20 690 0.9× 217 0.4× 130 0.4× 86 0.3× 114 0.6× 40 1.1k
Meritxell Rovira Spain 17 554 0.7× 793 1.4× 368 1.2× 178 0.7× 59 0.3× 28 1.2k
Yafa Ariav Israel 13 658 0.9× 481 0.8× 159 0.5× 183 0.7× 130 0.7× 13 1.2k
Hong Y. Choi Canada 13 342 0.5× 353 0.6× 106 0.3× 118 0.5× 93 0.5× 25 763
Jacob Hald Denmark 11 1.1k 1.4× 1.2k 2.0× 732 2.3× 340 1.3× 61 0.3× 19 1.9k
Kenichiro Furuyama Japan 13 633 0.8× 1.1k 1.8× 632 2.0× 420 1.6× 40 0.2× 22 1.6k
Adam Denley Australia 12 1.1k 1.5× 186 0.3× 237 0.7× 732 2.9× 111 0.6× 15 1.6k
Erika Peverelli Italy 24 595 0.8× 312 0.5× 226 0.7× 736 2.9× 70 0.4× 73 1.5k

Countries citing papers authored by Avital Swisa

Since Specialization
Citations

This map shows the geographic impact of Avital Swisa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Avital Swisa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Avital Swisa more than expected).

Fields of papers citing papers by Avital Swisa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Avital Swisa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Avital Swisa. The network helps show where Avital Swisa may publish in the future.

Co-authorship network of co-authors of Avital Swisa

This figure shows the co-authorship network connecting the top 25 collaborators of Avital Swisa. A scholar is included among the top collaborators of Avital Swisa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Avital Swisa. Avital Swisa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Swisa, Avital, Julia E. Kieckhaefer, Scott G. Daniel, et al.. (2024). The evolutionarily ancient FOXA transcription factors shape the murine gut microbiome via control of epithelial glycosylation. Developmental Cell. 59(16). 2069–2084.e8. 3 indexed citations
2.
Swisa, Avital, Elisabetta Manduchi, Yemin Lan, et al.. (2022). H3K27me3 Demethylases Maintain the Transcriptional and Epigenomic Landscape of the Intestinal Epithelium. Cellular and Molecular Gastroenterology and Hepatology. 15(4). 821–839. 2 indexed citations
3.
Borghesan, Michela, Juan Fafián‐Labora, Olga Eleftheriadou, et al.. (2019). Small Extracellular Vesicles Are Key Regulators of Non-cell Autonomous Intercellular Communication in Senescence via the Interferon Protein IFITM3. Cell Reports. 27(13). 3956–3971.e6. 205 indexed citations
4.
Tran, Stella, Ajit Shah, Gao Sun, et al.. (2019). mTORC1-to-AMPK switching underlies β cell metabolic plasticity during maturation and diabetes. Journal of Clinical Investigation. 129(10). 4124–4137. 93 indexed citations
5.
Mandelbaum, Amitai D., Sharon Kredo‐Russo, Nadav Myers, et al.. (2019). miR-17-92 and miR-106b-25 clusters regulate beta cell mitotic checkpoint and insulin secretion in mice. Diabetologia. 62(9). 1653–1666. 15 indexed citations
6.
Horwitz, E. Philip, Lars Krogvold, Avital Swisa, et al.. (2018). β-Cell DNA Damage Response Promotes Islet Inflammation in Type 1 Diabetes. Diabetes. 67(11). 2305–2318. 45 indexed citations
7.
Swisa, Avital, Benjamin Gläser, & Yuval Dor. (2017). Metabolic Stress and Compromised Identity of Pancreatic Beta Cells. Frontiers in Genetics. 8. 21–21. 116 indexed citations
8.
Ziv, Oren, E. Philip Horwitz, Hai Zemmour, et al.. (2016). Pancreatic β-Cells Express the Fetal Islet Hormone Gastrin in Rodent and Human Diabetes. Diabetes. 66(2). 426–436. 45 indexed citations
9.
Swisa, Avital, Dana Avrahami, Noa Eden, et al.. (2016). PAX6 maintains β cell identity by repressing genes of alternative islet cell types. Journal of Clinical Investigation. 127(1). 230–243. 116 indexed citations
10.
Karin, Omer, Avital Swisa, Benjamin Gläser, Yuval Dor, & Uri Alon. (2016). Dynamical compensation in physiological circuits. Molecular Systems Biology. 12(11). 886–886. 57 indexed citations
11.
Swisa, Avital, Zvi Granot, Natalia A. Tamarina, et al.. (2015). Loss of Liver Kinase B1 (LKB1) in Beta Cells Enhances Glucose-stimulated Insulin Secretion Despite Profound Mitochondrial Defects. Journal of Biological Chemistry. 290(34). 20934–20946. 31 indexed citations
12.
Khalaileh, Abed, Avigail Dreazen, Avital Swisa, et al.. (2013). Phosphorylation of Ribosomal Protein S6 Attenuates DNA Damage and Tumor Suppression during Development of Pancreatic Cancer. Cancer Research. 73(6). 1811–1820. 67 indexed citations
13.
Leu, Nico De, Yves Heremans, Luc Baeyens, et al.. (2013). Conditional Hypovascularization and Hypoxia in Islets Do Not Overtly Influence Adult β-Cell Mass or Function. Diabetes. 62(12). 4165–4173. 19 indexed citations
14.
Wikström, Jakob D., Tal Israeli, Etty Bachar-Wikström, et al.. (2013). AMPK Regulates ER Morphology and Function in Stressed Pancreatic β-Cells via Phosphorylation of DRP1. Molecular Endocrinology. 27(10). 1706–1723. 100 indexed citations
15.
Leu, Nico De, Yves Heremans, Violette Coppens, et al.. (2013). Short-term overexpression of VEGF-A in mouse beta cells indirectly stimulates their proliferation and protects against diabetes. Diabetologia. 57(1). 140–147. 20 indexed citations
16.
Klochendler, Agnes, Noa Weinberg-Corem, Avital Swisa, et al.. (2012). A Transgenic Mouse Marking Live Replicating Cells Reveals In Vivo Transcriptional Program of Proliferation. Developmental Cell. 23(4). 681–690. 45 indexed citations
17.
Granot, Zvi, Avital Swisa, Judith Magenheim, et al.. (2009). LKB1 Regulates Pancreatic β Cell Size, Polarity, and Function. Cell Metabolism. 10(4). 296–308. 138 indexed citations
18.
Khalaileh, Abed, Tsufit Gonen‐Gross, Judith Magenheim, et al.. (2008). Determinants of pancreatic β‐cell regeneration. Diabetes Obesity and Metabolism. 10(s4). 128–135. 10 indexed citations
19.
Hezel, Aram F., Sushma Gurumurthy, Zvi Granot, et al.. (2008). Pancreatic Lkb1 Deletion Leads to Acinar Polarity Defects and Cystic Neoplasms. Molecular and Cellular Biology. 28(7). 2414–2425. 128 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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