Annette M.G. Dirac
- Molecular Biology top 2%
- Ubiquitin and proteasome pathways 10
- RNA and protein synthesis mechanisms 4
- Cancer Research top 2%
- NF-κB Signaling Pathways 3
- Oncology top 2%
- Cancer-related Molecular Pathways 2
- Virology top 5%
- HIV Research and Treatment 5
- Cell Biology top 2%
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- interferon and immune responses 2
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- Genetics and Neurodevelopmental Disorders 2
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- HIV/AIDS drug development and treatment 2
- Co-authors
- René BernardsThijn R. BrummelkampSebastian NijmanTitia K. SixmaMark P.A. Luna‐VargasArno VeldsAlan D. D’AndreaRon Kerkhoven
- Partner nations
- NetherlandsDenmarkUnited States
In The Last Decade
Annette M.G. Dirac
18 papers receiving 4.3k citations
Hit Papers
Peers
Comparison fields: 5 of 97
- Molecular Biology 3.7k
- Cancer Research 811
- Oncology 1.2k
- Virology 149
- Cell Biology 477
Countries citing papers authored by Annette M.G. Dirac
This map shows the geographic impact of Annette M.G. Dirac's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Annette M.G. Dirac with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Annette M.G. Dirac more than expected).
Fields of papers citing papers by Annette M.G. Dirac
This network shows the impact of papers produced by Annette M.G. Dirac. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Annette M.G. Dirac. The network helps show where Annette M.G. Dirac may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Annette M.G. Dirac, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2020 | 25 | |
| 2 | 2012 | 33 | |
| 3 | 2012 | 332 | |
| 4 | 2011 | 216 | |
| 5 | 2010 | 69 | |
| 6 | 2010 | 72 | |
| 7 | 2006 | 35 | |
| 8 | 2006 | 62 | |
| 9 | 2005 | 458 | |
| 10 | A Genomic and Functional Inventory of Deubiquitinating Enzymesbreakdown → | 2005 | 1514 |
| 11 | 2005 | 7 | |
| 12 | Loss of the cylindromatosis tumour suppressor inhibits apoptosis by activating NF-κBbreakdown → | 2003 | 778 |
| 13 | 2003 | 201 | |
| 14 | 2002 | 44 | |
| 15 | 2002 | 22 | |
| 16 | 2001 | 29 | |
| 17 | 2001 | 40 | |
| 18 | 1999 | 411 |
About Annette M.G. Dirac
Annette M.G. Dirac is a scholar working on Virology, Cancer Research and Molecular Biology, having authored 18 papers that have together received 4.3k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (10 papers), HIV Research and Treatment (5 papers), RNA and protein synthesis mechanisms (4 papers), NF-κB Signaling Pathways (3 papers), interferon and immune responses (2 papers), Genetics and Neurodevelopmental Disorders (2 papers), Cancer-related Molecular Pathways (2 papers) and HIV/AIDS drug development and treatment (2 papers). The work is most often cited by research in Molecular Biology (3.7k citations), Cancer Research (811 citations) and Oncology (1.2k citations). Annette M.G. Dirac has collaborated with scholars based in Netherlands, Denmark and United States. Frequent co-authors include René Bernards, Thijn R. Brummelkamp, Sebastian Nijman, Titia K. Sixma, Mark P.A. Luna‐Vargas, Arno Velds, Alan D. D’Andrea, Ron Kerkhoven, Tony T. Huang and Hediye Erdjument‐Bromage.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.