Ana Cuenda
- Molecular Biology top 0.5%
- Oncology top 1%
- Immunology top 1%
- Cancer Research top 1%
- Cell Biology top 0.5%
- Co-authors
- Philip CohenDario R. AlessiAlan R. SaltielDavid T. DudleySimon RousseauRoger MeierYair N. DozaP P Cohen
- Topics
- Melanoma and MAPK Pathways (37 papers)Protein Kinase Regulation and GTPase Signaling (18 papers)Cellular Mechanics and Interactions (12 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryAngewandte Chemie International Edition
- Partner nations
- SpainUnited KingdomUnited States
In The Last Decade
Ana Cuenda
76 papers receiving 11.3k citations
Hit Papers
Peers
Comparison fields: 5 of 127
- Molecular Biology 7.9k
- Oncology 2.1k
- Immunology 1.9k
- Cancer Research 1.6k
- Cell Biology 1.4k
Countries citing papers authored by Ana Cuenda
This map shows the geographic impact of Ana Cuenda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ana Cuenda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ana Cuenda more than expected).
Fields of papers citing papers by Ana Cuenda
This network shows the impact of papers produced by Ana Cuenda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ana Cuenda. The network helps show where Ana Cuenda may publish in the future.
Co-authorship network of co-authors of Ana Cuenda
This figure shows the co-authorship network connecting the top 25 collaborators of Ana Cuenda. A scholar is included among the top collaborators of Ana Cuenda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ana Cuenda. Ana Cuenda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 4 | |
| 3 | 16 | |
| 4 | 23 | |
| 5 | 54 | |
| 6 | 31 | |
| 7 | 89 | |
| 8 | 70 | |
| 9 | 23 | |
| 10 | 82 | |
| 11 | p38 MAP-Kinases pathway regulation, function and role in human diseasesbreakdown → | 1115 |
| 12 | 48 | |
| 13 | 189 | |
| 14 | 89 | |
| 15 | 96 | |
| 16 | 100 | |
| 17 | 49 | |
| 18 | 193 | |
| 19 | 26 | |
| 20 | 18 |
About Ana Cuenda
Ana Cuenda is a scholar working on Cell Biology, Molecular Biology and Cancer Research, having authored 76 papers that have together received 11.5k indexed citations. Recurring topics across this work include Melanoma and MAPK Pathways (37 papers), Protein Kinase Regulation and GTPase Signaling (18 papers) and Cellular Mechanics and Interactions (12 papers). The work is most often cited by research in Molecular Biology (7.9k citations), Cancer Research (1.6k citations) and Immunology (1.9k citations). Ana Cuenda has collaborated with scholars based in Spain, United Kingdom and United States. Frequent co-authors include Philip Cohen, Dario R. Alessi, Alan R. Saltiel, David T. Dudley, Simon Rousseau, Roger Meier, Yair N. Doza, P P Cohen, T. F. Gallagher and J.B. Rouse. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Angewandte Chemie International Edition.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.