Adrian Nañez

1.2k total citations · 1 hit paper
16 papers, 942 citations indexed

About

Adrian Nañez is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Reproductive Medicine. According to data from OpenAlex, Adrian Nañez has authored 16 papers receiving a total of 942 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 3 papers in Pediatrics, Perinatology and Child Health and 3 papers in Reproductive Medicine. Recurrent topics in Adrian Nañez's work include Renal and related cancers (6 papers), Birth, Development, and Health (3 papers) and Sperm and Testicular Function (3 papers). Adrian Nañez is often cited by papers focused on Renal and related cancers (6 papers), Birth, Development, and Health (3 papers) and Sperm and Testicular Function (3 papers). Adrian Nañez collaborates with scholars based in United States. Adrian Nañez's co-authors include John H. Richburg, Donna F. Kusewitt, Jinhee Kim, Ken Inoki, Mustafa Şahin, Maria D. Person, Mark J. Walker, Kirsteen H. Maclean, Jianjun Shen and Gordon B. Mills and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Cancer Research and Environmental Health Perspectives.

In The Last Decade

Adrian Nañez

16 papers receiving 927 citations

Hit Papers

ATM signals to TSC2 in the cytoplasm to regulate mTORC1 i... 2010 2026 2015 2020 2010 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Adrian Nañez United States 10 602 253 146 111 110 16 942
Pengli Bu United States 17 500 0.8× 97 0.4× 136 0.9× 66 0.6× 27 0.2× 29 946
Charles E. Foulds United States 19 1.1k 1.8× 112 0.4× 320 2.2× 170 1.5× 47 0.4× 28 1.7k
Étienne Audet‐Walsh Canada 23 682 1.1× 95 0.4× 399 2.7× 82 0.7× 55 0.5× 58 1.4k
Gabriele De Luca Italy 21 880 1.5× 91 0.4× 355 2.4× 52 0.5× 50 0.5× 37 1.3k
S. Nilsson Sweden 13 377 0.6× 103 0.4× 103 0.7× 36 0.3× 51 0.5× 17 1.1k
Yonghai Li China 18 292 0.5× 63 0.2× 88 0.6× 36 0.3× 61 0.6× 41 888
H. Zankl Germany 20 610 1.0× 189 0.7× 282 1.9× 68 0.6× 20 0.2× 78 1.5k
Delphine Courilleau France 15 788 1.3× 115 0.5× 97 0.7× 39 0.4× 19 0.2× 19 1.1k
In-geun Ryoo South Korea 15 660 1.1× 87 0.3× 181 1.2× 52 0.5× 16 0.1× 19 1.0k

Countries citing papers authored by Adrian Nañez

Since Specialization
Citations

This map shows the geographic impact of Adrian Nañez's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Adrian Nañez with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Adrian Nañez more than expected).

Fields of papers citing papers by Adrian Nañez

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Adrian Nañez. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Adrian Nañez. The network helps show where Adrian Nañez may publish in the future.

Co-authorship network of co-authors of Adrian Nañez

This figure shows the co-authorship network connecting the top 25 collaborators of Adrian Nañez. A scholar is included among the top collaborators of Adrian Nañez based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Adrian Nañez. Adrian Nañez is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Pettus, Liping H., Matthew P. Bourbeau, Michael D. Bartberger, et al.. (2019). Discovery of AM-6494: A Potent and Orally Efficacious β-Site Amyloid Precursor Protein Cleaving Enzyme 1 (BACE1) Inhibitor with in Vivo Selectivity over BACE2. Journal of Medicinal Chemistry. 63(5). 2263–2281. 28 indexed citations
2.
Ramos, Kenneth S., et al.. (2013). Albumin-Like Proteins Are Critical Regulators of Vascular Redox Signaling. Oxidative Medicine and Cellular Longevity. 2013. 1–10. 2 indexed citations
3.
Nañez, Adrian, et al.. (2011). AHR Regulates WT1 Genetic Programming during Murine Nephrogenesis. Molecular Medicine. 17(11-12). 1275–1284. 7 indexed citations
4.
Nañez, Adrian, Irma N. Ramos, & Kenneth S. Ramos. (2011). A Mutant Ahr Allele Protects the Embryonic Kidney from Hydrocarbon-Induced Deficits in Fetal Programming. Environmental Health Perspectives. 119(12). 1745–1753. 11 indexed citations
5.
Ramos, Kenneth S., Diego E. Montoya–Durango, Ivo Teneng, Adrian Nañez, & Vilius Stribinskis. (2011). Epigenetic control of embryonic renal cell differentiation by L1 retrotransposon. Birth Defects Research Part A Clinical and Molecular Teratology. 91(8). 693–702. 17 indexed citations
6.
Alexander, Angela, Jinhee Kim, Adrian Nañez, et al.. (2010). ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS. Proceedings of the National Academy of Sciences. 107(9). 4153–4158. 576 indexed citations breakdown →
7.
Alexander, Angela, Shengli Cai, Jinhee Kim, et al.. (2010). Abstract 4831: ATM signals to TSC2 in the cytoplasm to regulate mTORC1 and autophagy in response to ROS. Cancer Research. 70(8_Supplement). 4831–4831. 3 indexed citations
8.
Ramos, Kenneth S. & Adrian Nañez. (2009). Genetic regulatory networks of nephrogenesis: Deregulation of WT1 splicing by benzo(a)pyrene. Birth Defects Research Part C Embryo Today Reviews. 87(2). 192–197. 6 indexed citations
9.
Ramos, Kenneth S., et al.. (2007). From genomics to mechanistic insight: A global perspective on molecular deficits induced by environmental agents. Environmental and Molecular Mutagenesis. 48(5). 395–399. 5 indexed citations
10.
Falahatpisheh, Hadi, Adrian Nañez, Diego E. Montoya–Durango, et al.. (2007). Activation profiles of HSPA5 during the glomerular mesangial cell stress response to chemical injury. Cell Stress and Chaperones. 12(3). 209–209. 17 indexed citations
11.
Bozulic, Larry D., Mohammad Tariq Malik, David Powell, et al.. (2007). Plasma membrane Ca2+-ATPase associates with CLP36, α-actinin and actin in human platelets. Thrombosis and Haemostasis. 97(4). 587–597. 31 indexed citations
12.
Ramos, Kenneth S., et al.. (2006). Modulation of Biological Regulatory Networks During Nephrogenesis. Drug Metabolism Reviews. 38(4). 677–683. 2 indexed citations
13.
Nañez, Adrian, et al.. (2005). Disruption of glomerular cell-cell and cell-matrix interactions in hydrocarbon nephropathy. American Journal of Physiology-Renal Physiology. 289(6). F1291–F1303. 15 indexed citations
14.
Richburg, John H. & Adrian Nañez. (2003). Fas- or FasL-deficient mice display an increased sensitivity to nitrobenzene-induced testicular germ cell apoptosis. Toxicology Letters. 139(1). 1–10. 30 indexed citations
15.
Richburg, John H., et al.. (2000). Sensitivity of Testicular Germ Cells to Toxicant-Induced Apoptosis in gld Mice That Express a Nonfunctional Form of Fas Ligand1. Endocrinology. 141(2). 787–793. 115 indexed citations
16.
Richburg, John H., Adrian Nañez, & Hong Gao. (1999). Participation of the Fas-Signaling System in the Initiation of Germ Cell Apoptosis in Young Rat Testes after Exposure to Mono-(2-Ethylhexyl) Phthalate. Toxicology and Applied Pharmacology. 160(3). 271–278. 77 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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