John H. Richburg

3.1k total citations
47 papers, 2.5k citations indexed

About

John H. Richburg is a scholar working on Reproductive Medicine, Molecular Biology and Surgery. According to data from OpenAlex, John H. Richburg has authored 47 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Reproductive Medicine, 26 papers in Molecular Biology and 8 papers in Surgery. Recurrent topics in John H. Richburg's work include Sperm and Testicular Function (32 papers), Cell death mechanisms and regulation (16 papers) and Effects and risks of endocrine disrupting chemicals (7 papers). John H. Richburg is often cited by papers focused on Sperm and Testicular Function (32 papers), Cell death mechanisms and regulation (16 papers) and Effects and risks of endocrine disrupting chemicals (7 papers). John H. Richburg collaborates with scholars based in United States, France and United Kingdom. John H. Richburg's co-authors include Kim Boekelheide, Jeongwu Lee, Pei‐Li Yao, Marvin L. Meistrich, Yi-Chen Lin, Adrian Nañez, Elizabeth Shipp, Caitlin J. Murphy, Frederick C. Kauffman and Hong Gao and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Free Radical Biology and Medicine.

In The Last Decade

John H. Richburg

46 papers receiving 2.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John H. Richburg United States 27 1.3k 1.1k 587 576 276 47 2.5k
Elissa W.P. Wong United States 26 1.2k 1.0× 931 0.9× 303 0.5× 614 1.1× 174 0.6× 38 2.3k
Brigitte Le Magueresse‐Battistoni France 29 778 0.6× 609 0.6× 610 1.0× 411 0.7× 267 1.0× 65 2.1k
Zuomin Zhou China 33 1.4k 1.1× 1.5k 1.4× 223 0.4× 1.1k 1.9× 322 1.2× 125 3.4k
Karin Williams United Kingdom 29 540 0.4× 967 0.9× 307 0.5× 233 0.4× 321 1.2× 44 2.5k
Yi‐Liang Miao China 32 701 0.6× 1.5k 1.4× 216 0.4× 1.3k 2.2× 228 0.8× 99 2.9k
Will M. Lee Hong Kong 40 2.7k 2.1× 1.6k 1.5× 282 0.5× 1.4k 2.5× 162 0.6× 72 4.0k
M. Parvinen Finland 38 2.2k 1.8× 1.6k 1.6× 136 0.2× 1.1k 1.8× 206 0.7× 80 3.9k
Li‐Jun Huo China 27 524 0.4× 830 0.8× 324 0.6× 831 1.4× 181 0.7× 88 2.0k
Keesook Lee South Korea 29 457 0.4× 1.3k 1.3× 143 0.2× 203 0.4× 222 0.8× 74 2.7k

Countries citing papers authored by John H. Richburg

Since Specialization
Citations

This map shows the geographic impact of John H. Richburg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John H. Richburg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John H. Richburg more than expected).

Fields of papers citing papers by John H. Richburg

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John H. Richburg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John H. Richburg. The network helps show where John H. Richburg may publish in the future.

Co-authorship network of co-authors of John H. Richburg

This figure shows the co-authorship network connecting the top 25 collaborators of John H. Richburg. A scholar is included among the top collaborators of John H. Richburg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John H. Richburg. John H. Richburg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tiwary, Richa & John H. Richburg. (2023). Mono-(2-ethylhexyl) phthalate reversibly disrupts the blood-testis barrier in pubertal rats. Toxicological Sciences. 197(2). 147–154. 10 indexed citations
2.
3.
Bona, Kristin R. Di, et al.. (2019). Copper transporter 1 (CTR1) expression by mouse testicular germ cells, but not Sertoli cells, is essential for functional spermatogenesis. PLoS ONE. 14(4). e0215522–e0215522. 14 indexed citations
4.
Voss, Jorine, et al.. (2018). MEHP-induced rat testicular inflammation does not exacerbate germ cell apoptosis. Reproduction. 156(1). 35–46. 13 indexed citations
5.
Murphy, Caitlin J., et al.. (2017). Mono-(2-ethylhexyl) phthalate-induced Sertoli cell injury stimulates the production of pro-inflammatory cytokines in Fischer 344 rats. Reproductive Toxicology. 69. 150–158. 18 indexed citations
6.
Murphy, Caitlin J. & John H. Richburg. (2014). Implications of Sertoli cell induced germ cell apoptosis to testicular pathology. PubMed. 4(2). e979110–e979110. 49 indexed citations
7.
Murphy, Caitlin J., et al.. (2014). Age- and Species-Dependent Infiltration of Macrophages into the Testis of Rats and Mice Exposed to Mono-(2-Ethylhexyl) Phthalate (MEHP)1. Biology of Reproduction. 91(1). 18–18. 30 indexed citations
8.
Richburg, John H. & Patricia B. Hoyer. (2010). Reproductive and endocrine toxicology. Elsevier eBooks. 5 indexed citations
9.
Lin, Yi-Chen, Pei‐Li Yao, & John H. Richburg. (2010). FasL Gene–Deficient Mice Display a Limited Disruption in Spermatogenesis and Inhibition of Mono-(2-ethylhexyl) Phthalate–Induced Germ Cell Apoptosis. Toxicological Sciences. 114(2). 335–345. 34 indexed citations
10.
Yao, Pei‐Li, et al.. (2006). Transcriptional Regulation of FasL Expression and Participation of sTNF-α in Response to Sertoli Cell Injury. Journal of Biological Chemistry. 282(8). 5420–5431. 55 indexed citations
11.
12.
Seaman, Fred C., et al.. (2003). Cisplatin-induced pulse of germ cell apoptosis precedes long-term elevated apoptotic rates in C57/BL/6 mouse testis. APOPTOSIS. 8(1). 101–108. 44 indexed citations
13.
Richburg, John H., Kamin J. Johnson, Heidi A. Schoenfeld, Marvin L. Meistrich, & David J. Dix. (2002). Defining the cellular and molecular mechanisms of toxicant action in the testis. Toxicology Letters. 135(3). 167–183. 43 indexed citations
14.
Richburg, John H.. (2000). The relevance of spontaneous- and chemically-induced alterations in testicular germ cell apoptosis to toxicology. Toxicology Letters. 112-113. 79–86. 149 indexed citations
15.
Lee, Jeongwu, John H. Richburg, Elizabeth Shipp, Marvin L. Meistrich, & Kim Boekelheide. (1999). The Fas System, a Regulator of Testicular Germ Cell Apoptosis, Is Differentially Up-Regulated in Sertoli Cell Versus Germ Cell Injury of the Testis*. Endocrinology. 140(2). 852–858. 241 indexed citations
16.
Richburg, John H., Adrian Nañez, & Hong Gao. (1999). Participation of the Fas-Signaling System in the Initiation of Germ Cell Apoptosis in Young Rat Testes after Exposure to Mono-(2-Ethylhexyl) Phthalate. Toxicology and Applied Pharmacology. 160(3). 271–278. 77 indexed citations
17.
Boekelheide, Kim, Jeongwu Lee, Elizabeth Shipp, John H. Richburg, & Gang Li. (1998). Expression of Fas system-related genes in the testis during development and after toxicant exposure. Toxicology Letters. 102-103. 503–508. 42 indexed citations
18.
Lee, Jeongwu, et al.. (1997). The Fas System Is a Key Regulator of Germ Cell Apoptosis in the Testis*. Endocrinology. 138(5). 2081–2088. 443 indexed citations
19.
Zaleski, Jan, John H. Richburg, & Frederick C. Kauffman. (1993). Preservation of the rate and profile of xenobiotic metabolism in rat hepatocytes stored in liquid nitrogen. Biochemical Pharmacology. 46(1). 111–116. 44 indexed citations
20.
Halleck, Margaret S., John H. Richburg, & Frederick C. Kauffman. (1992). Reversible and irreversible oxidant injury to PC12 cells by hydrogen peroxide. Free Radical Biology and Medicine. 12(2). 137–144. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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