Michael B. Kastan
Impact in
- Oncology top 0.02%
- Cancer-related Molecular Pathways
- Cancer Research top 0.05%
- Carcinogens and Genotoxicity Assessment
Papers in
-
- DNA Repair Mechanisms 78
- RNA modifications and cancer 11
- Cell death mechanisms and regulation 11
- RNA Research and Splicing 10
- Oncology 90
- Cancer-related Molecular Pathways 78
- Co-authors
- Christopher J. Bakkenist (10 shared papers)Jiří Bártek (4 shared papers)Leland H. Hartwell (1 shared paper)Dae‐Sik Lim (11 shared papers)Christine E. Canman (19 shared papers)Seong‐Tae Kim (8 shared papers)Bert Vogelstein (2 shared papers)Onyinye Onyekwere (2 shared papers)
- Journals
- Genes & Development (11 papers)Molecular and Cellular Biology (9 papers)Proceedings of the National Academy of Sciences (8 papers)Cancer Research (7 papers)Cell (6 papers)
- Partner nations
- United StatesUnited KingdomIsrael
In The Last Decade
Michael B. Kastan
155 papers receiving 38.3k citations
Michael B. Kastan's Hit Papers
Peers
Comparison fields: 5 of 166
- Oncology 18.4k
- Cancer Research 8.3k
- Molecular Biology 30.5k
- Biotechnology 3.0k
- Cell Biology 4.0k
Countries citing papers authored by Michael B. Kastan
This map shows the geographic impact of Michael B. Kastan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael B. Kastan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael B. Kastan more than expected).
Fields of papers citing papers by Michael B. Kastan
This network shows the impact of papers produced by Michael B. Kastan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael B. Kastan. The network helps show where Michael B. Kastan may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael B. Kastan, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 156 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Participation of p53 protein in the cellular response to DNA damage. Hit paper breakdown → | 1991 | 3276 |
| 2 | DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociation Hit paper breakdown → | 2003 | 2680 |
| 3 | Cell Cycle Control and Cancer Hit paper breakdown → | 1994 | 2151 |
| 4 | Cell-cycle checkpoints and cancer Hit paper breakdown → | 2004 | 2144 |
| 5 | Activation of the ATM Kinase by Ionizing Radiation and Phosphorylation of p53 Hit paper breakdown → | 1998 | 1676 |
| 6 | Wild-type p53 is a cell cycle checkpoint determinant following irradiation. Hit paper breakdown → | 1992 | 1597 |
| 7 | The complexity of p53 modulation: emerging patterns from divergent signals Hit paper breakdown → | 1998 | 1115 |
| 8 | Conversion of Bcl-2 to a Bax-like Death Effector by Caspases Hit paper breakdown → | 1997 | 943 |
| 9 | The NLRP3 Inflammasome Protects against Loss of Epithelial Integrity and Mortality during Experimental Colitis Hit paper breakdown → | 2010 | 826 |
| 10 | Interaction of the p53-Regulated Protein Gadd45 with Proliferating Cell Nuclear Antigen Hit paper breakdown → | 1994 | 805 |
| 11 | DNA strand breaks: the DNA template alterations that trigger p53-dependent DNA damage response pathways. Hit paper breakdown → | 1994 | 722 |
| 12 | DNA damage induces phosphorylation of the amino terminus of p53 Hit paper breakdown → | 1997 | 677 |
| 13 | Substrate Specificities and Identification of Putative Substrates of ATM Kinase Family Members Hit paper breakdown → | 1999 | 665 |
| 14 | ATM phosphorylates p95/nbs1 in an S-phase checkpoint pathway Hit paper breakdown → | 2000 | 643 |
| 15 | The many substrates and functions of ATM Hit paper breakdown → | 2000 | 633 |
| 16 | ATM signals to TSC2 in the cytoplasm to regulate mTORC1 in response to ROS Hit paper breakdown → | 2010 | 576 |
| 17 | Regulation of p53 Translation and Induction after DNA Damage by Ribosomal Protein L26 and Nucleolin Hit paper breakdown → | 2005 | 535 |
| 18 | ATM-dependent phosphorylation of Mdm2 on serine 395: role in p53 activation by DNA damage Hit paper breakdown → | 2001 | 514 |
| 19 | 2006 | 495 | |
| 20 | Autophagy Links Inflammasomes to Atherosclerotic Progression Hit paper breakdown → | 2012 | 479 |
About Michael B. Kastan
Michael B. Kastan is a scholar working on Molecular Biology, Oncology, Cancer Research, Biotechnology and Cell Biology, having authored 156 papers that have together received 39.1k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (78 papers), DNA Repair Mechanisms (78 papers), Carcinogens and Genotoxicity Assessment (28 papers), Cancer Research and Treatments (19 papers), Microtubule and mitosis dynamics (14 papers), RNA modifications and cancer (11 papers), Cell death mechanisms and regulation (11 papers) and RNA Research and Splicing (10 papers). The work is most often cited by research in Oncology (18.4k citations), Cancer Research (8.3k citations), Molecular Biology (30.5k citations), Biotechnology (3.0k citations) and Cell Biology (4.0k citations). Michael B. Kastan has collaborated with scholars based in United States, United Kingdom and Israel. Frequent co-authors include Christopher J. Bakkenist, Jiří Bártek, Leland H. Hartwell, Dae‐Sik Lim, Christine E. Canman, Seong‐Tae Kim, Bert Vogelstein, Onyinye Onyekwere, R W Craig and Bo Xu. Their work appears in journals such as Genes & Development, Molecular and Cellular Biology, Proceedings of the National Academy of Sciences, Cancer Research and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.