William W. Schlaepfer
- Molecular Biology top 2%
- Cell Biology top 0.2%
- Cellular and Molecular Neuroscience top 1%
- Neurology top 1%
- Physiology top 5%
- Co-authors
- Un‐Jin P. ZimmermanHong LinMartin J. CardenJinbin ZhaiRafaela Cañete-SolerRichard P. BungeJennifer BruceV M Lee
- Topics
- Skin and Cellular Biology Research (45 papers)Nerve injury and regeneration (16 papers)Cellular Mechanics and Interactions (14 papers)
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
William W. Schlaepfer
107 papers receiving 5.5k citations
Peers
Comparison fields: 5 of 123
- Molecular Biology 2.7k
- Cell Biology 2.6k
- Cellular and Molecular Neuroscience 1.9k
- Neurology 1.1k
- Physiology 626
Countries citing papers authored by William W. Schlaepfer
This map shows the geographic impact of William W. Schlaepfer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William W. Schlaepfer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William W. Schlaepfer more than expected).
Fields of papers citing papers by William W. Schlaepfer
This network shows the impact of papers produced by William W. Schlaepfer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William W. Schlaepfer. The network helps show where William W. Schlaepfer may publish in the future.
Co-authorship network of co-authors of William W. Schlaepfer
This figure shows the co-authorship network connecting the top 25 collaborators of William W. Schlaepfer. A scholar is included among the top collaborators of William W. Schlaepfer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William W. Schlaepfer. William W. Schlaepfer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 93 | |
| 3 | 38 | |
| 4 | 24 | |
| 5 | 157 | |
| 6 | 10 | |
| 7 | 5 | |
| 8 | 50 | |
| 9 | 5 | |
| 10 | 36 | |
| 11 | 27 | |
| 12 | 32 | |
| 13 | 13 | |
| 14 | 30 | |
| 15 | 18 | |
| 16 | 81 | |
| 17 | 97 | |
| 18 | 21 | |
| 19 | 15 | |
| 20 | 35 |
About William W. Schlaepfer
William W. Schlaepfer is a scholar working on Cell Biology, Neurology and Cellular and Molecular Neuroscience, having authored 107 papers that have together received 5.8k indexed citations. Recurring topics across this work include Skin and Cellular Biology Research (45 papers), Nerve injury and regeneration (16 papers) and Cellular Mechanics and Interactions (14 papers). The work is most often cited by research in Cell Biology (2.6k citations), Cellular and Molecular Neuroscience (1.9k citations) and Developmental Neuroscience (398 citations). William W. Schlaepfer has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Un‐Jin P. Zimmerman, Hong Lin, Martin J. Carden, Jinbin Zhai, Rafaela Cañete-Soler, Richard P. Bunge, Jennifer Bruce, V M Lee, Mary Hasler and Michael L. Schwartz. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.