Baruj Benacerraf
About
In The Last Decade
Baruj Benacerraf
516 papers receiving 22.2k citations
Hit Papers
Peers
Comparison fields: 5 of 145
- Immunology 17.4k
- Radiology, Nuclear Medicine and Imaging 6.6k
- Molecular Biology 6.4k
- Epidemiology 2.0k
- Genetics 1.8k
Countries citing papers authored by Baruj Benacerraf
This map shows the geographic impact of Baruj Benacerraf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Baruj Benacerraf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Baruj Benacerraf more than expected).
Fields of papers citing papers by Baruj Benacerraf
This network shows the impact of papers produced by Baruj Benacerraf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Baruj Benacerraf. The network helps show where Baruj Benacerraf may publish in the future.
Co-authorship network of co-authors of Baruj Benacerraf
This figure shows the co-authorship network connecting the top 25 collaborators of Baruj Benacerraf. A scholar is included among the top collaborators of Baruj Benacerraf based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Baruj Benacerraf. Baruj Benacerraf is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 173 | |
| 2 | 12 | |
| 3 | Regulation of the immune response to tumor antigen. V. Modulation of suppressor t-cell activity in vivo. | 3 |
| 4 | Idiotypic analysis of anti-GAT antibodies. VI. Identification and strain distribution of the GA-1 idiotype. | 14 |
| 5 | 109 | |
| 6 | Antigen-Specific T Cell-Mediated Suppression: III. Induction of Antigen-Specific Suppressor T Cells (TS2) in L-Glutamic Acid60-L-Alanine30-L-Tyrosine10 (GAT) Responder Mice by Nonresponder-Derived GAT-Suppressor Factor (GAT-TSF) | 38 |
| 7 | Idiotypic analysis of anti-GAT antibodies. I. Presence of common idiotypic specificities in both responder and nonresponder mice. | 54 |
| 8 | 64 | |
| 9 | Immunosuppressive Factor(s) Extracted from Lymphoid Cells of Nonresponder Mice Primed with L-Glutamic Acid60-L-Alanine30-L-Tyrosine10 (GAT) I. Activity and Antigenic Specificity | 107 |
| 10 | H-2-linked genetic control of resistance to histocompatible tumors. | 20 |
| 11 | Cell interactions between histoincompatible T and B lymphocytes. VII. Cooperative responses between lymphocytes are controlled by genes in the I region of the H-2 complex breakdown → | 219 |
| 12 | Immunological tolerance; mechanisms and potential therapeutic applications | 35 |
| 13 | Genetic Control of Specific Immune Responses breakdown → | 526 |
| 14 | 14 | |
| 15 | 3 | |
| 16 | 5 | |
| 17 | Increased phagocytic activity of the res produced by transmissible agent derived from transplantable tumors. Abstr. | 1 |
| 18 | 19 | |
| 19 | [Effect of the quantity of iodine fixed on normal and heat-modified blood proteins on the phagocytosis of these colloids by reticuloendothelial cells]. | 8 |
| 20 | 8 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.