William C. Forrester

8.2k citations

33 papers · 5.2k indexed · 2 hit papers · h-index 26

Molecular Biology top 1%
Genetics top 1%
Oncology top 5%
Immunology top 5%
Genetics top 2%

Co-authors: Mark Groudine Frédérique Gay Thalia Papayannopoulou Yujiang Geno Shi Yang Shi Guangchao Sui El Bachir Affar Rudolf Grosschedl
Topics: Genomics and Chromatin Dynamics (10 papers)CRISPR and Genetic Engineering (8 papers)Animal Genetics and Reproduction (7 papers)
Cited by: Molecular Biology Genetics Aging
Journals: Nature Science Cell
Partner nations: United States Switzerland China

In The Last Decade

William C. Forrester

33 papers receiving 5.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A DNA vector-based RNAi technology to suppress gene expression in mammalian cells

2002 955 citations Guangchao Sui, El Bachir Affar et al. Proceedings of the National Academy of Sciences profile →
p53 inhibits CRISPR–Cas9 engineering in human pluripotent stem cells

2018 658 citations Robert J. Ihry, Kathleen A. Worringer et al. Nature Medicine profile →

Peers

Countries citing papers authored by William C. Forrester

Since Specialization

Citations

This map shows the geographic impact of William C. Forrester's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William C. Forrester with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William C. Forrester more than expected).

Fields of papers citing papers by William C. Forrester

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William C. Forrester. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William C. Forrester. The network helps show where William C. Forrester may publish in the future.

Co-authorship network of co-authors of William C. Forrester

This figure shows the co-authorship network connecting the top 25 collaborators of William C. Forrester. A scholar is included among the top collaborators of William C. Forrester based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with William C. Forrester. William C. Forrester is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Synergy between BRD9- and IKZF3-Targeting as a Therapeutic Strategy for Multiple Myeloma 2024 ·Cancers ·Basudev Chowdhury,Swati Garg,Wei Ni,Martin Sattler,(unknown),Chengcheng Meng,(unknown),Richard M. Stone,William C. Forrester,Edmund Harrington,Sara J. Buhrlage,James D. Griffin,Ellen Weisberg	2
2	A strategy to assess the cellular activity of E3 ligase components against neo-substrates using electrophilic probes 2021 ·Cell chemical biology ·Benika J. Pinch,Dennis L. Buckley,Scott Gleim,Scott M. Brittain,Laura Tandeske,(unknown),(unknown),(unknown),Lei Xu,(unknown),Markus Schirle,Elizabeth R. Sprague,William C. Forrester,Dustin Dovala,Lynn M. McGregor,Claudio R. Thoma	14
3	p53 dynamics vary between tissues and are linked with radiation sensitivity 2021 ·Nature Communications ·Jacob Stewart-Ornstein,Yoshiko Iwamoto,Miles A. Miller,Mark Prytyskach,Stéphane Ferretti,Philipp Holzer,Joerg Kallen,Pascal Furet,Ashwini Jambhekar,William C. Forrester,Ralph Weissleder,Galit Lahav	54
4	Manumycin polyketides act as molecular glues between UBR7 and P53 2020 ·Nature Chemical Biology ·Yosuke Isobe,Mikiko Okumura,Lynn M. McGregor,Scott M. Brittain,Michael D. Jones,Xiaoyou Liang,R. Larry White,William C. Forrester,Jeffrey M. McKenna,John A. Tallarico,Markus Schirle,Thomas J. Maimone,Daniel K. Nomura	91
5	p53 inhibits CRISPR–Cas9 engineering in human pluripotent stem cellsbreakdown → 2018 ·Nature Medicine ·Robert J. Ihry,Kathleen A. Worringer,Max R. Salick,Elizabeth Frias,Daniel Ho,Kraig M. Theriault,Sravya Kommineni,Julie Chen,Marie Sondey,Chaoyang Ye,Ranjit Randhawa,(unknown),Zinger Yang,Gregory McAllister,Carsten Russ,John Reece-Hoyes,William C. Forrester,Gregory R. Hoffman,Ricardo Dolmetsch,Ajamete Kaykas	658
6	Recurrent ubiquitin B silencing in gynecological cancers establishes dependence on ubiquitin C 2017 ·Journal of Clinical Investigation ·Alexia T. Kedves,Scott Gleim,Xiaoyou Liang,Dennis M. Bonal,Frederic Sigoillot,Fred Harbinski,(unknown),(unknown),Elizabeth George,Prafulla C. Gokhale,Quang‐Dé Nguyen,Paul T. Kirschmeier,R Distel,Jeremy L. Jenkins,Michael S. Goldberg,William C. Forrester	23
7	p53 pulses lead to distinct patterns of gene expression albeit similar DNA-binding dynamics 2017 ·Nature Structural & Molecular Biology ·Antonina Hafner,Jacob Stewart-Ornstein,Jeremy E. Purvis,William C. Forrester,Martha L. Bulyk,Galit Lahav	77
8	Cell-to-Cell Variation in p53 Dynamics Leads to Fractional Killing 2016 ·Cell ·Andrew L. Paek,Julia Liu,Alexander Loewer,William C. Forrester,Galit Lahav	197
9	A DNA vector-based RNAi technology to suppress gene expression in mammalian cellsbreakdown → 2002 ·Proceedings of the National Academy of Sciences ·Guangchao Sui,(unknown),El Bachir Affar,Frédérique Gay,Yujiang Geno Shi,William C. Forrester,Yang Shi	955
10	Pushing the Envelope 2002 ·Molecular Cell ·Nobuhiko Shinkura,William C. Forrester	69
11	The Retinoblastoma Protein Acts as a Transcriptional Coactivator Required for Osteogenic Differentiation 2001 ·Molecular Cell ·David M. Thomas,Shannon A. Carty,Denise M. Piscopo,Jong-Seo Lee,(unknown),William C. Forrester,Philip W. Hinds	303
12	Nuclear matrix attachment regions antagonize methylation-dependent repression of long-range enhancer-promoter interactions 1999 ·Genes & Development ·William C. Forrester,Luis Ángel Fernández,Rudolf Grosschedl	70
13	Nuclear Matrix Attachment Regions Confer Long-range Function upon the Immunoglobulin Enhancer 1998 ·Cold Spring Harbor Symposia on Quantitative Biology ·Luis Ángel Fernández,Michael Winkler,William C. Forrester,Thomas Jenuwein,Rudolf Grosschedl	12
14	Extension of chromatin accessibility by nuclear matrix attachment regions 1997 ·Nature ·Thomas Jenuwein,William C. Forrester,Luis Ángel Fernández,Götz Laible,(unknown),Rudolf Grosschedl	207
15	Inhibition of an Erythroid Differentiation Switch by the Helix-Loop-Helix Protein Id1 1995 ·Journal of Biological Chemistry ·James Lister,William C. Forrester,Margaret H. Baron	87
16	Translocation of an Erythroid-Specific Hypersensitive Site in Deletion-Type Hereditary Persistence of Fetal Hemoglobin 1990 ·Molecular and Cellular Biology ·James T. Elder,William C. Forrester,Craig B. Thompson,Dixie L. Mager,Paula S. Henthorn,Mary Peretz,Thalia Papayannopoulou,Mark Groudine	9
17	High-level beta-globin expression after retroviral transfer of locus activation region-containing human beta-globin gene derivatives into murine erythroleukemia cells. 1990 ·Proceedings of the National Academy of Sciences ·Ulrike Novak,Elizabeth Harris,William C. Forrester,Mark Groudine,Richard Gelinas	110
18	A deletion of the human beta-globin locus activation region causes a major alteration in chromatin structure and replication across the entire beta-globin locus. 1990 ·Genes & Development ·William C. Forrester,Elliot Epner,Monica Driscoll,Tariq Enver,M Brice,Thalia Papayannopoulou,Mark Groudine	393
19	Evidence for a locus activation region: the formation of developmentally stable hypersensitive sites in globin-expressing hybrids 1987 ·Nucleic Acids Research ·William C. Forrester,S. Takegawa,Thalia Papayannopoulou,(unknown),Mark Groudine	393
20	DNA sequence studies of simian virus 40 chromosomal excision and integration in rat cells 1984 ·Journal of Molecular Biology ·Peter A. Bullock,William C. Forrester,Michael R. Botchan	83

About William C. Forrester

William C. Forrester is a scholar working on Molecular Biology, Oncology and Genetics, having authored 33 papers that have together received 5.2k indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (10 papers), CRISPR and Genetic Engineering (8 papers) and Animal Genetics and Reproduction (7 papers). The work is most often cited by research in Molecular Biology (4.5k citations), Genetics (1.4k citations) and Aging (75 citations). William C. Forrester has collaborated with scholars based in United States, Switzerland and China. Frequent co-authors include Mark Groudine, Frédérique Gay, Thalia Papayannopoulou, Yujiang Geno Shi, Yang Shi, Guangchao Sui, El Bachir Affar, Rudolf Grosschedl, Elliot Epner and Thomas Jenuwein. Their work appears in journals such as Nature, Science and Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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