Martha L. Bulyk
- Molecular Biology top 0.2%
- Cancer Research top 1%
- Genetics top 1%
- Plant Science top 2%
- Immunology top 2%
- Co-authors
- Michael F. BergerSavina JaegerDaniel E. NewburgerAnthony PhilippakisAntonina HafnerGalit LahavAshwini JambhekarAndrew R. Gehrke
- Topics
- Genomics and Chromatin Dynamics (71 papers)RNA Research and Splicing (30 papers)RNA and protein synthesis mechanisms (27 papers)
- Partner nations
- United StatesCanadaChina
In The Last Decade
Martha L. Bulyk
113 papers receiving 12.2k citations
Hit Papers
Peers
Comparison fields: 5 of 155
- Molecular Biology 10.2k
- Cancer Research 1.7k
- Genetics 1.5k
- Plant Science 1.2k
- Immunology 1.0k
Countries citing papers authored by Martha L. Bulyk
This map shows the geographic impact of Martha L. Bulyk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martha L. Bulyk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martha L. Bulyk more than expected).
Fields of papers citing papers by Martha L. Bulyk
This network shows the impact of papers produced by Martha L. Bulyk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martha L. Bulyk. The network helps show where Martha L. Bulyk may publish in the future.
Co-authorship network of co-authors of Martha L. Bulyk
This figure shows the co-authorship network connecting the top 25 collaborators of Martha L. Bulyk. A scholar is included among the top collaborators of Martha L. Bulyk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martha L. Bulyk. Martha L. Bulyk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 19 | |
| 3 | 24 | |
| 4 | 3 | |
| 5 | 32 | |
| 6 | 38 | |
| 7 | 29 | |
| 8 | 66 | |
| 9 | 2 | |
| 10 | 433 | |
| 11 | STAT3 Activation of miR-21 and miR-181b-1 via PTEN and CYLD Are Part of the Epigenetic Switch Linking Inflammation to Cancerbreakdown → | 709 |
| 12 | 134 | |
| 13 | 38 | |
| 14 | Diversity and Complexity in DNA Recognition by Transcription Factorsbreakdown → | 761 |
| 15 | 163 | |
| 16 | 41 | |
| 17 | 186 | |
| 18 | 284 | |
| 19 | 59 | |
| 20 | 46 |
About Martha L. Bulyk
Martha L. Bulyk is a scholar working on Molecular Biology, Aging and Health Informatics, having authored 114 papers that have together received 12.3k indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (71 papers), RNA Research and Splicing (30 papers) and RNA and protein synthesis mechanisms (27 papers). The work is most often cited by research in Aging (265 citations), Molecular Biology (10.2k citations) and Cancer Research (1.7k citations). Martha L. Bulyk has collaborated with scholars based in United States, Canada and China. Frequent co-authors include Michael F. Berger, Savina Jaeger, Daniel E. Newburger, Anthony Philippakis, Antonina Hafner, Galit Lahav, Ashwini Jambhekar, Andrew R. Gehrke, Heather A. Hirsch and Dimitrios Iliopoulos. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.