W. K. Dowjat

539 total citations
22 papers, 425 citations indexed

About

W. K. Dowjat is a scholar working on Molecular Biology, Physiology and Immunology. According to data from OpenAlex, W. K. Dowjat has authored 22 papers receiving a total of 425 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 6 papers in Physiology and 4 papers in Immunology. Recurrent topics in W. K. Dowjat's work include Alzheimer's disease research and treatments (6 papers), Mitochondrial Function and Pathology (3 papers) and Down syndrome and intellectual disability research (2 papers). W. K. Dowjat is often cited by papers focused on Alzheimer's disease research and treatments (6 papers), Mitochondrial Function and Pathology (3 papers) and Down syndrome and intellectual disability research (2 papers). W. K. Dowjat collaborates with scholars based in United States, Poland and Italy. W. K. Dowjat's co-authors include Jerzy Węgiel, Thomas Wısnıewskı, Izabela Kuchna, Tatyana Adayev, Krzysztof Nowicki, Henryk M. Wı́sniewski, Spiros Efthimiopoulos, Blas Frangione, J Kulczycki and Wanda Łojkowska and has published in prestigious journals such as Journal of Cell Science, Neuroscience and International Journal of Cancer.

In The Last Decade

W. K. Dowjat

22 papers receiving 413 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
W. K. Dowjat United States 11 194 181 124 98 60 22 425
Woo‐Joo Song South Korea 8 212 1.1× 390 2.2× 418 3.4× 298 3.0× 55 0.9× 11 806
Andrew Mears United Kingdom 5 143 0.7× 187 1.0× 44 0.4× 28 0.3× 57 0.9× 5 355
Min‐Su Jung South Korea 7 65 0.3× 166 0.9× 108 0.9× 82 0.8× 22 0.4× 7 369
Anna Henry Australia 10 387 2.0× 328 1.8× 12 0.1× 63 0.6× 74 1.2× 11 592
Manasi Malik United States 8 219 1.1× 196 1.1× 23 0.2× 32 0.3× 22 0.4× 10 501
Nicolas Malmanche Portugal 10 140 0.7× 246 1.4× 19 0.2× 42 0.4× 24 0.4× 13 376
A. Palkama Finland 13 125 0.6× 171 0.9× 103 0.8× 11 0.1× 21 0.3× 61 500
Jayashree Viswanathan Finland 12 263 1.4× 205 1.1× 9 0.1× 69 0.7× 30 0.5× 23 454
Ling Nga Chan Hong Kong 11 36 0.2× 153 0.8× 53 0.4× 32 0.3× 24 0.4× 15 501
D R Davis United Kingdom 6 147 0.8× 174 1.0× 8 0.1× 41 0.4× 34 0.6× 7 372

Countries citing papers authored by W. K. Dowjat

Since Specialization
Citations

This map shows the geographic impact of W. K. Dowjat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. K. Dowjat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. K. Dowjat more than expected).

Fields of papers citing papers by W. K. Dowjat

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. K. Dowjat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. K. Dowjat. The network helps show where W. K. Dowjat may publish in the future.

Co-authorship network of co-authors of W. K. Dowjat

This figure shows the co-authorship network connecting the top 25 collaborators of W. K. Dowjat. A scholar is included among the top collaborators of W. K. Dowjat based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. K. Dowjat. W. K. Dowjat is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kaczmarski, Wojciech, Madhabi Barua, Bozena Mazur‐Kolecka, et al.. (2013). Intracellular distribution of differentially phosphorylated dual‐specificity tyrosine phosphorylation‐regulated kinase 1A (DYRK1A). Journal of Neuroscience Research. 92(2). 162–173. 22 indexed citations
2.
Dowjat, W. K., et al.. (2006). Trisomy-driven overexpression of DYRK1A kinase in the brain of subjects with Down syndrome. Neuroscience Letters. 413(1). 77–81. 145 indexed citations
3.
Kulczycki, J, et al.. (2002). [Familial Alzheimer's disease connected with mutation in presenilin gene 1 (P117L)].. PubMed. 35(2). 213–24. 5 indexed citations
4.
Dowjat, W. K., H Wiśniewski, & Thomas Wısnıewskı. (2001). Alzheimer’s disease presenilin-1 expression modulates the assembly of neurofilaments. Neuroscience. 103(1). 1–8. 14 indexed citations
5.
Dowjat, W. K., Thomas Wısnıewskı, Spiros Efthimiopoulos, & Henryk M. Wı́sniewski. (1999). Inhibition of neurite outgrowth by familial Alzheimer's disease-linked presenilin-1 mutations. Neuroscience Letters. 267(2). 141–144. 27 indexed citations
6.
Węgiel, Jerzy, Henryk M. Wı́sniewski, Izabela Kuchna, et al.. (1998). Cell-Type-Specific Enhancement of Amyloid-β Deposition in a Novel Presenilin-1 Mutation (P117L). Journal of Neuropathology & Experimental Neurology. 57(9). 831–838. 19 indexed citations
7.
Wısnıewskı, Thomas, W. K. Dowjat, Joseph D. Buxbaum, et al.. (1998). A novel Polish presenilin-1 mutation (P117L) is associated with familial Alzheimerʼs disease and leads to death as early as the age of 28 years. Neuroreport. 9(2). 217–221. 80 indexed citations
8.
Wısnıewskı, Thomas, W. K. Dowjat, Bruno Permanne, et al.. (1997). Presenilin-1 is associated with Alzheimer's disease amyloid.. PubMed. 151(2). 601–10. 21 indexed citations
9.
Dowjat, W. K., et al.. (1996). DNA and RNA strand scission by copper, zinc and manganese superoxide dismutases. BioMetals. 9(4). 327–35. 13 indexed citations
10.
Dowjat, W. K., Xi Huang, Sofia Cosentino, & Max Costa. (1996). Peroxidase deficiency of nickel-transformed hamster cells correlates with their increased resistance to cytotoxicity of peroxides. BioMetals. 9(2). 151–6. 1 indexed citations
11.
Lin, Xinhua, W. K. Dowjat, & Maria R. Castro Costa. (1994). Nickel-induced transformation of human cells causes loss of the phosphorylation of the retinoblastoma protein.. PubMed. 54(10). 2751–4. 17 indexed citations
12.
13.
Dowjat, W. K., et al.. (1989). In Vitro Proliferation of Murine Spleen Cells: I. Strain Variation of Response to Medium from Cultures of EL-4 Cells. Immunological Investigations. 18(8). 1007–1017. 2 indexed citations
14.
Dowjat, W. K., Ya Cao, Kunio Nagashima, Ayako Sakai, & Nancy H. Colburn. (1988). Comparison of P+ ‐active and ‐inactive pro‐1 homologues from human nasopharyngeal carcinoma cells. Molecular Carcinogenesis. 1(1). 33–40. 2 indexed citations
15.
Colburn, Nancy H., et al.. (1988). Transfer by pro gene transfection of tumor promoter-sensitive phenotype to promotion-insensitive JB6 cells.. PubMed. 48(5). 1195–200. 12 indexed citations
16.
Shimada, Tomiko, W. K. Dowjat, Thomas D. Gindhart, Michael I. Lerman, & Nancy H. Colburn. (1987). Lifespan extension of basal cell nevus syndrome fibroblasts by transfection with mouse pro or v‐myc genes. International Journal of Cancer. 39(5). 649–655. 4 indexed citations
17.
Quackenbush, Lindi J., W. K. Dowjat, & Marek Zaleski. (1985). IA mutation affects anti-Thy-1 response in mice. Immunogenetics. 21(6). 617–621. 2 indexed citations
18.
Quackenbush, Lindi J., W. K. Dowjat, & Marek Zaleski. (1985). Search for I-A trans-complementation affecting the anti-THY-1 response in mice: a final report. Immunological Investigations. 14(4). 379–385. 1 indexed citations
19.
Dowjat, W. K., Lindi J. Quackenbush, & Marek Zaleski. (1985). Strain differences in the response of normal murine spleen cells to interleukin-2 (IL-2). Immunological Investigations. 14(6). 493–501. 4 indexed citations
20.
Dowjat, W. K., et al.. (1982). Adhesion and locomotion of l5222 cells on endothelium, collagen and glass. Journal of Cell Science. 55(1). 317–325. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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