Thomas D. Gindhart

1.4k citations
30 papers · 1.2k indexed · h-index 17
Topics
Ubiquitin and proteasome pathways (5 papers)Immune Response and Inflammation (5 papers)Glycosylation and Glycoproteins Research (4 papers)

In The Last Decade

Thomas D. Gindhart

30 papers receiving 1.1k citations

Peers

Thomas D. Gindhart
Comparison fields: 5 of 92
  • Immunology 446
  • Molecular Biology 407
  • Oncology 186
  • Genetics 145
  • Cancer Research 99
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Citations per field
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Citations per year

Countries citing papers authored by Thomas D. Gindhart

Since Specialization
Citations

This map shows the geographic impact of Thomas D. Gindhart's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas D. Gindhart with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas D. Gindhart more than expected).

Fields of papers citing papers by Thomas D. Gindhart

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas D. Gindhart. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas D. Gindhart. The network helps show where Thomas D. Gindhart may publish in the future.

Co-authorship network of co-authors of Thomas D. Gindhart

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas D. Gindhart. A scholar is included among the top collaborators of Thomas D. Gindhart based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas D. Gindhart. Thomas D. Gindhart is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 68
2 4
3 9
4 107
5 14
6 2
7 6
8 20
9
Phorbol diester and epidermal growth factor receptors in 12-O-tetradecanoylphorbol-13-acetate-resistant and -sensitive mouse epidermal cells.
32
10 21
11 1
12 52
13
SUPPRESSION OF KILLING IN VITRO BY TUMOR-PROMOTING PHORBOL DIESTERS
3
14 17
15 105
16 19
17 47
18
Genetics of neoplasia--impact of ecogenetics on oncogenesis. A review.
28
19
beta-Estradiol reduces natural killer cells in mice.
181
20 131

About Thomas D. Gindhart

Thomas D. Gindhart is a scholar working on Cancer Research, Oncology and Immunology, having authored 30 papers that have together received 1.2k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (5 papers), Immune Response and Inflammation (5 papers) and Glycosylation and Glycoproteins Research (4 papers). The work is most often cited by research in Immunology (446 citations), Biochemistry (41 citations) and Oncology (186 citations). Thomas D. Gindhart has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include William E. Seaman, Marcia A. Blackman, Norman Talal, Nancy H. Colburn, Jirayr R. Roubinian, Yoshiyuki Nakamura, Bakul I. Dalal, John S. Greenspan, Zena Werb and Judith G. Hall. Their work appears in journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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