W. David Sedwick

3.4k citations

61 papers · 2.8k indexed · 1 hit paper · h-index 27

Impact in

Pathology and Forensic Medicine top 1%
- Genetic factors in colorectal cancer
Cancer Research top 2%
- Cancer Genomics and Diagnostics
- Carcinogens and Genotoxicity Assessment

Papers in

Pathology and Forensic Medicine 11
- Genetic factors in colorectal cancer 10
Cancer Research 8
- Cancer Genomics and Diagnostics 4

Co-authors: Martina Veigl David Korn Teresa Shu-Fong Wang Sanford D. Markowitz Lawrence C. Sowers Barbara Ramsay Shaw James Lutterbaugh Paul Modrich
Journals: Mutation research. Fundamental and molecular mechanisms of mutagenesis (5 papers)Journal of Biological Chemistry (5 papers)Molecular Pharmacology (4 papers)Experimental Biology and Medicine (4 papers)Oncogene (4 papers)
Partner nations: United States United Kingdom Canada

In The Last Decade

W. David Sedwick

61 papers receiving 2.5k citations

Hit Papers

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Biallelic inactivation of hMLH 1 by epigenetic gene silencing, a novel mechanism causing human MSI cancers 1998 · 507 citations

Peers

Countries citing papers authored by W. David Sedwick

Since Specialization

Citations

This map shows the geographic impact of W. David Sedwick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. David Sedwick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. David Sedwick more than expected).

Fields of papers citing papers by W. David Sedwick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. David Sedwick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. David Sedwick. The network helps show where W. David Sedwick may publish in the future.

Co-authors

The 25 scholars most cited alongside W. David Sedwick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with W. David Sedwick Line = papers co-authored together W. David Sedwick links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	On systems and control approaches to therapeutic gain BMC Cancer ·Tomas Radivoyevitch, Kenneth A. Loparo, Robert Jackson, W. David Sedwick	2006	7
2	Somatic mutation of hPMS2 as a possible cause of sporadic human colon cancer with microsatellite instability Oncogene ·Ai‐Hong Ma, Liang Xia, Susan J. Littman, Sandra E. Swinler, Gabriel Lader, Alexander Polinkovsky, Joseph Olechnowicz, Lakshmi Kasturi, James Lutterbaugh, Paul Modrich, Martina Veigl, Sanford D. Markowitz, W. David Sedwick	2000	23
3	Cytotoxicity and Mutagenicity of Frameshift-Inducing Agent ICR191 in Mismatch Repair-Deficient Colon Cancer Cells JNCI Journal of the National Cancer Institute ·Wei-Dong Chen, James R. Eshleman, M. Reza Aminoshariae, Ai‐Hong Ma, Neil Veloso, Sanford D. Markowitz, W. David Sedwick, Martina Veigl	2000	15
4	Mismatch repair and drug responses in cancer Drug Resistance Updates ·W. David Sedwick, Sanford D. Markowitz, Martina Veigl	1999	6
5	Chromosome number and structure both are markedly stable in RER colorectal cancers and are not destabilized by mutation of p53 Oncogene ·James R. Eshleman, Graham Casey, M Kochera, W. David Sedwick, Sandra E. Swinler, Martina Veigl, James K. V. Willson, Stuart Schwartz, Sanford D. Markowitz	1998	106
6	Increased transversions in a novel mutator colon cancer cell line Oncogene ·James R. Eshleman, Preston S. Donover, Susan J. Littman, Sandra E. Swinler, Guo‐Min Li, James Lutterbaugh, James K. V. Willson, Paul Modrich, W. David Sedwick, Sanford D. Markowitz, Martina Veigl	1998	13
7	Effect of lsopropyl‐β‐D‐thiogalactopyranosid induction of the lac operon on the specificity of spontaneous and doxorubicin‐induced mutations in Escherichia coli Environmental and Molecular Mutagenesis ·Martina Veigl, S. Donover, R. David Anderson, Lee M. Akst, Caitlin Sedwick, W. David Sedwick	1995	2
8	Correlation of doxorubicin footprints with deletion endpoints in lacO of E. coli Mutation research. Fundamental and molecular mechanisms of mutagenesis ·W. David Sedwick, R. David Anderson, Jeff Baxter, S. Donover, Steven Schneiter, Martina Veigl	1995	2
9	Molecular analysis of hypoxanthine phosphoribosyltransferase gene deletions induced by α- and X-radiation in human lymphoblastoid cells Mutation research. Fundamental and molecular mechanisms of mutagenesis ·C.-Y. Bao, A.-H. Ma, Helen H. Evans, Min‐Fen Horng, J. Mencl, T. E. Hui, W. David Sedwick	1995	51
10	Sequencing of double-stranded polymerase chain reaction products for mutation analysis Mutation research. Fundamental and molecular mechanisms of mutagenesis ·R. David Anderson, C.-Y. Bao, Dana T. Minnick, Jeff Baxter, Martina Veigl, W. David Sedwick	1993	8
11	Specificities mediated by neighboring nucleotides appear to underlie mutation induced by antifolates in E. coli Mutation research. Fundamental and molecular mechanisms of mutagenesis ·Martina Veigl, Steven Schneiter, Stacey Mollis, W. David Sedwick	1991	13
12	2,4-Dichloro-5-(1-o-carboranylmethyl)-6-methylpyrimidine: a potential synthon for 5-(1-o-carboranylmethyl)pyrimidines The Journal of Organic Chemistry ·Robert C. Reynolds, Todd W. Trask, W. David Sedwick	1991	23
13	Rapid mapping of deletion and duplication mutations by the polymerase chain reaction Biochemical and Biophysical Research Communications ·R. David Anderson, Martina Veigl, W. David Sedwick	1990	3
14	The uses and limitations of calmodulin antagonists Pharmacology & Therapeutics ·Martina Veigl, Rachel E. Klevit, W. David Sedwick	1989	42
15	Hydrolysis of N3-methyl-2′-deoxycytidine: Model compound for reactivity of protonated cytosine residues in DNA Mutation research. Fundamental and molecular mechanisms of mutagenesis ·Lawrence C. Sowers, W. David Sedwick, Barbara Ramsay Shaw	1989	21
16	Base stacking and molecular polarizability: Effect of a methyl group in the 5-position of pyrimidines Biochemical and Biophysical Research Communications ·Lawrence C. Sowers, Barbara Ramsay Shaw, W. David Sedwick	1987	92
17	DNA base modification: Ionized base pairs and mutagenesis Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis ·Lawrence C. Sowers, Barbara Ramsay Shaw, Martina Veigl, W. David Sedwick	1987	90
18	Drug concentration-dependent DNA lesions are induced by the lipid-soluble antifolate, piritrexim (BW301U). Molecular Pharmacology ·Randall G. Richards, Oliver E. Brown, Maura L. Gillison, W. David Sedwick	1986	4
19	Deoxyuridine misincorporation causes site-specific mutational lesions in the lacI gene of Escherichia coli Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis ·W. David Sedwick, Oliver E. Brown, Barry W. Glickman	1986	33
20	The Specificity and Interaction with Poliovirus of an Inhibitory Bovine Serum The Journal of Immunology ·Joseph S. Pagano, Raymond V. Gilden, W. David Sedwick	1965	11

About W. David Sedwick

W. David Sedwick is a scholar working on Pathology and Forensic Medicine, Cancer Research, Molecular Biology, Oncology and Microbiology, having authored 61 papers that have together received 2.8k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (17 papers), DNA and Nucleic Acid Chemistry (15 papers), Genetic factors in colorectal cancer (10 papers), Virology and Viral Diseases (6 papers), Virus-based gene therapy research (5 papers), Synthesis and Characterization of Heterocyclic Compounds (5 papers), Cancer therapeutics and mechanisms (5 papers) and Cancer Genomics and Diagnostics (4 papers). The work is most often cited by research in Pathology and Forensic Medicine (821 citations), Cancer Research (664 citations), Oncology (718 citations), Virology (120 citations) and Molecular Biology (1.6k citations). W. David Sedwick has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Martina Veigl, David Korn, Teresa Shu-Fong Wang, Sanford D. Markowitz, Lawrence C. Sowers, Barbara Ramsay Shaw, James Lutterbaugh, Paul Modrich, Guo‐Min Li and T. J. Wiktor. Their work appears in journals such as Mutation research. Fundamental and molecular mechanisms of mutagenesis, Journal of Biological Chemistry, Molecular Pharmacology, Experimental Biology and Medicine and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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