Martina Veigl

2.5k total citations · 1 hit paper
38 papers, 1.9k citations indexed

About

Martina Veigl is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Oncology. According to data from OpenAlex, Martina Veigl has authored 38 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 14 papers in Pathology and Forensic Medicine and 11 papers in Oncology. Recurrent topics in Martina Veigl's work include Genetic factors in colorectal cancer (14 papers), DNA Repair Mechanisms (10 papers) and Cancer Genomics and Diagnostics (5 papers). Martina Veigl is often cited by papers focused on Genetic factors in colorectal cancer (14 papers), DNA Repair Mechanisms (10 papers) and Cancer Genomics and Diagnostics (5 papers). Martina Veigl collaborates with scholars based in United States, China and Sweden. Martina Veigl's co-authors include W. David Sedwick, Sanford D. Markowitz, James Lutterbaugh, Paul Modrich, Guo‐Min Li, Thomas Vanaman, Lakshmi Kasturi, James T. Drummond, Sumudra Periyasamy and James K. V. Willson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Blood.

In The Last Decade

Martina Veigl

38 papers receiving 1.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Biallelic inactivation of hMLH 1 by epigenetic gene silencing, a novel mechanism causing human MSI cancers

1998 507 citations Martina Veigl, Lakshmi Kasturi et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Martina Veigl United States	20	1.1k	853	677	619	198	38	1.9k
M Meuth United Kingdom	23	1.4k 1.3×	497 0.6×	392 0.6×	522 0.8×	272 1.4×	43	1.9k
Turid Knutsen United States	23	969 0.9×	359 0.4×	752 1.1×	382 0.6×	253 1.3×	37	2.1k
Pietro Taverna United States	27	1.8k 1.6×	455 0.5×	788 1.2×	359 0.6×	128 0.6×	90	2.5k
John Sgouros United Kingdom	11	1.4k 1.3×	233 0.3×	346 0.5×	370 0.6×	251 1.3×	18	1.8k
William E. Pierceall United States	25	1.4k 1.3×	390 0.5×	1.1k 1.7×	351 0.6×	114 0.6×	55	2.5k
James Watters United States	21	1.2k 1.2×	187 0.2×	815 1.2×	400 0.6×	237 1.2×	40	1.9k
Silje Nord Norway	21	1.1k 1.0×	202 0.2×	566 0.8×	917 1.5×	416 2.1×	40	1.9k
Gregory J. Riggins United States	21	1.0k 1.0×	193 0.2×	432 0.6×	326 0.5×	169 0.9×	35	1.9k
I Taylor United States	19	1.6k 1.5×	449 0.5×	553 0.8×	395 0.6×	190 1.0×	40	2.9k
Giuseppe Roscilli Italy	25	1.1k 1.0×	177 0.2×	573 0.8×	367 0.6×	272 1.4×	48	1.8k

Countries citing papers authored by Martina Veigl

Since Specialization

Citations

This map shows the geographic impact of Martina Veigl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Martina Veigl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Martina Veigl more than expected).

Fields of papers citing papers by Martina Veigl

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Martina Veigl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Martina Veigl. The network helps show where Martina Veigl may publish in the future.

Co-authorship network of co-authors of Martina Veigl

This figure shows the co-authorship network connecting the top 25 collaborators of Martina Veigl. A scholar is included among the top collaborators of Martina Veigl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Martina Veigl. Martina Veigl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Hao, Yujun, Baoyu He, Liping Wu, et al.. (2022). Nuclear translocation of p85β promotes tumorigenesis of PIK3CA helical domain mutant cancer. Nature Communications. 13(1). 1974–1974. 24 indexed citations

Cummings, Linda C., Prashanthi N. Thota, Joseph Willis, et al.. (2017). A nonrandomized trial of vitamin D supplementation for Barrett’s esophagus. PLoS ONE. 12(9). e0184928–e0184928. 9 indexed citations

Gittleman, Haley, Alona Merkulova, Omar Alhalabi, et al.. (2016). A Cross-sectional Study of KLKB1 and PRCP Polymorphisms in Patient Samples with Cardiovascular Disease. Frontiers in Medicine. 3. 17–17. 16 indexed citations

Guda, Kishore, Martina Veigl, Vinay Varadan, et al.. (2015). Novel recurrently mutated genes in African American colon cancers. Proceedings of the National Academy of Sciences. 112(4). 1149–1154. 92 indexed citations

Adams, Mark D., Martina Veigl, Zhenghe Wang, et al.. (2012). Global mutational profiling of formalin-fixed human colon cancers from a pathology archive. Modern Pathology. 25(12). 1599–1608. 17 indexed citations

Li, Long Shan, Julio C. Morales, Martina Veigl, et al.. (2009). DNA mismatch repair (MMR)‐dependent 5‐fluorouracil cytotoxicity and the potential for new therapeutic targets. British Journal of Pharmacology. 158(3). 679–692. 60 indexed citations

Kaminski, Beth A., Suzanne Kadereit, R Eric Miller, et al.. (2003). Reduced expression of NFAT-associated genes in UCB versus adult CD4+ T lymphocytes during primary stimulation. Blood. 102(13). 4608–4617. 75 indexed citations

Ma, Ai‐Hong, Liang Xia, Susan J. Littman, et al.. (2000). Somatic mutation of hPMS2 as a possible cause of sporadic human colon cancer with microsatellite instability. Oncogene. 19(18). 2249–2256. 23 indexed citations

Allay, Esther R., Martina Veigl, & Stanton L. Gerson. (1999). Mice over-expressing human O6alkylguanine-DNA alkyltransferase selectively reduce O6methylguanine mediated carcinogenic mutations to threshold levels after N-methyl-N-nitrosourea. Oncogene. 18(25). 3783–3787. 38 indexed citations

10.

Sedwick, W. David, Sanford D. Markowitz, & Martina Veigl. (1999). Mismatch repair and drug responses in cancer. Drug Resistance Updates. 2(5). 295–306. 6 indexed citations

11.

Eshleman, James R., Graham Casey, W. David Sedwick, et al.. (1998). Chromosome number and structure both are markedly stable in RER colorectal cancers and are not destabilized by mutation of p53. Oncogene. 17(6). 719–725. 106 indexed citations

12.

Eshleman, James R., Preston S. Donover, Susan J. Littman, et al.. (1998). Increased transversions in a novel mutator colon cancer cell line. Oncogene. 16(9). 1125–1130. 13 indexed citations

13.

Veigl, Martina, et al.. (1995). Effect of lsopropyl‐β‐D‐thiogalactopyranosid induction of the lac operon on the specificity of spontaneous and doxorubicin‐induced mutations in Escherichia coli. Environmental and Molecular Mutagenesis. 26(1). 16–25. 2 indexed citations

14.

Sedwick, W. David, et al.. (1995). Correlation of doxorubicin footprints with deletion endpoints in lacO of E. coli. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 326(1). 17–27. 2 indexed citations

15.

Anderson, R. David, et al.. (1993). Sequencing of double-stranded polymerase chain reaction products for mutation analysis. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 288(1). 181–185. 8 indexed citations

16.

Minnick, Dana T., Stanton L. Gerson, Luba Dumenco, Martina Veigl, & W. David Sedwick. (1993). Specificity of bischloroethylnitrosourea-induced mutation in a Chinese hamster ovary cell line transformed to express human O6-alkylguanine-DNA alkyltransferase.. PubMed. 53(5). 997–1003. 28 indexed citations

17.

Veigl, Martina, et al.. (1991). Specificities mediated by neighboring nucleotides appear to underlie mutation induced by antifolates in E. coli. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 246(1). 75–91. 13 indexed citations

18.

Anderson, R. David, Martina Veigl, & W. David Sedwick. (1990). Rapid mapping of deletion and duplication mutations by the polymerase chain reaction. Biochemical and Biophysical Research Communications. 170(2). 416–420. 3 indexed citations

19.

Veigl, Martina, Rachel E. Klevit, & W. David Sedwick. (1989). The uses and limitations of calmodulin antagonists. Pharmacology & Therapeutics. 44(2). 181–239. 42 indexed citations

20.

Sowers, Lawrence C., Barbara Ramsay Shaw, Martina Veigl, & W. David Sedwick. (1987). DNA base modification: Ionized base pairs and mutagenesis. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 177(2). 201–218. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact